Werner Schmid

Summary

Affiliation: Medical University of Vienna
Country: Austria

Publications

  1. ncbi Hypochlorite-oxidized low density lipoproteins reduce production and bioavailability of nitric oxide in RAW 264.7 macrophages by distinct mechanisms
    Werner Schmid
    Center for Physiology and Pathophysiology, Institute of Physiology, Medical University of Vienna, and Department of Urology, Rudolfstiftung Hospital, Vienna, Austria
    Life Sci 83:50-8. 2008
  2. ncbi Estimation of platelet function under high shear conditions to assist a rapid diagnosis of Heyde Syndrome
    Werner Schmid
    Center for Physiology and Pathophysiology, Institute of Physiology, Department of Internal Medicine III, Medical University of Vienna, Vienna 1090, Austria
    Platelets 19:636-40. 2008
  3. ncbi Hypochlorite-oxidized LDL induces intraplatelet ROS formation and surface exposure of CD40L--a prominent role of CD36
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 213:129-34. 2010
  4. ncbi Specific binding of hypochlorite-oxidized HDL to platelet CD36 triggers proinflammatory and procoagulant effects
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 212:153-60. 2010
  5. ncbi Platelet-stimulating effects of oxidized LDL are not attributable to toxic properties of the lipoproteins
    Werner Schmid
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstr 17 A 1090 Vienna, Austria
    Thromb Res 122:630-9. 2008
  6. ncbi Decreased VASP phosphorylation in platelets of male and female smokers of young age
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17 A 1090 Vienna, Austria
    Platelets 21:596-603. 2010
  7. ncbi Oxidation by hypochlorite converts protective HDL into a potent platelet agonist
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstrasse 17, Vienna, Austria
    FEBS Lett 582:778-84. 2008
  8. ncbi Redox-sensitive impairment of porcine coronary artery vasodilation by hypochlorite-modified LDL
    Christian A Plass
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 190:330-7. 2007
  9. ncbi CD32-mediated platelet aggregation in vitro by anti-thymocyte globulin: implication of therapy-induced in vivo thrombocytopenia
    Hendrik Jan Ankersmit
    Department of Cardiothoracic Surgery, University of Vienna, Vienna, Austria
    Am J Transplant 3:754-9. 2003

Collaborators

Detail Information

Publications9

  1. ncbi Hypochlorite-oxidized low density lipoproteins reduce production and bioavailability of nitric oxide in RAW 264.7 macrophages by distinct mechanisms
    Werner Schmid
    Center for Physiology and Pathophysiology, Institute of Physiology, Medical University of Vienna, and Department of Urology, Rudolfstiftung Hospital, Vienna, Austria
    Life Sci 83:50-8. 2008
    ..By mediating these effects on production and bioavailability of NO, hyp-OxLDL might also contribute to atherogenesis by reducing the antiatherogenic effects of nitric oxide...
  2. ncbi Estimation of platelet function under high shear conditions to assist a rapid diagnosis of Heyde Syndrome
    Werner Schmid
    Center for Physiology and Pathophysiology, Institute of Physiology, Department of Internal Medicine III, Medical University of Vienna, Vienna 1090, Austria
    Platelets 19:636-40. 2008
    ..This test offers an easy to use diagnostic tool to evaluate platelet function in suspected Heyde Syndrome thus leading to immediate tailored patient's management...
  3. ncbi Hypochlorite-oxidized LDL induces intraplatelet ROS formation and surface exposure of CD40L--a prominent role of CD36
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 213:129-34. 2010
    ....
  4. ncbi Specific binding of hypochlorite-oxidized HDL to platelet CD36 triggers proinflammatory and procoagulant effects
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 212:153-60. 2010
    ..Therefore it was the aim of this work to clarify if these platelet-activating effects result from specific binding of hypochlorite-oxidized HDL (hyp-OxHDL) to the platelet surface and to identify responsible receptors...
  5. ncbi Platelet-stimulating effects of oxidized LDL are not attributable to toxic properties of the lipoproteins
    Werner Schmid
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstr 17 A 1090 Vienna, Austria
    Thromb Res 122:630-9. 2008
    ..As a conclusion, platelet-activating effects of oxidatively modified LDL are not attributable to toxic properties of the lipoproteins and this finding might expand possibilities for therapeutical intervention...
  6. ncbi Decreased VASP phosphorylation in platelets of male and female smokers of young age
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Schwarzspanierstr 17 A 1090 Vienna, Austria
    Platelets 21:596-603. 2010
    ..Therefore, our findings add another piece of evidence to the harmful effects of smoking in both male and female smokers...
  7. ncbi Oxidation by hypochlorite converts protective HDL into a potent platelet agonist
    Alice Assinger
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstrasse 17, Vienna, Austria
    FEBS Lett 582:778-84. 2008
    ..The observed effects are dose dependent and can be blocked by an antibody directed against the lipoprotein-binding domain of platelet thrombospondin- and scavenger receptor CD36...
  8. ncbi Redox-sensitive impairment of porcine coronary artery vasodilation by hypochlorite-modified LDL
    Christian A Plass
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstr 17, A 1090 Vienna, Austria
    Atherosclerosis 190:330-7. 2007
    ....
  9. ncbi CD32-mediated platelet aggregation in vitro by anti-thymocyte globulin: implication of therapy-induced in vivo thrombocytopenia
    Hendrik Jan Ankersmit
    Department of Cardiothoracic Surgery, University of Vienna, Vienna, Austria
    Am J Transplant 3:754-9. 2003
    ..Since blocking of CD32 abrogates platelet aggregation, we suggest that CD32 plays a crucial role in ATG-induced thrombocytopenia...