J W Bauer

Summary

Country: Austria

Publications

  1. ncbi Large melanocytic nevi in hereditary epidermolysis bullosa
    J W Bauer
    Department of Dermatology, General Hospital Salzburg, Austria
    J Am Acad Dermatol 44:577-84. 2001
  2. ncbi Galanin family of peptides in skin function
    J W Bauer
    Department of Dermatology, SALK and Paracelsus Medical University Salzburg, Müller Hauptstrasse 48, 5020 Salzburg, Austria
    EXS 102:51-9. 2010
  3. ncbi Epidermolysis bullosa naevi reveal a distinctive dermoscopic pattern
    C M Lanschuetzer
    Department of Dermatology, Paracelsus Private Medical University, Muellner Hauptstrasse 48, A-5020 Salzburg, Austria
    Br J Dermatol 153:97-102. 2005
  4. ncbi A localized variant of paraneoplastic pemphigus: acantholysis associated with malignant melanoma
    H Schaeppi
    Department of Dermatology, General Hospital Salzburg, Mullner Hauptstrasse 48, A 5020 Salzburg, Austria
    Br J Dermatol 144:1249-54. 2001
  5. ncbi Nonsense-associated altered splicing of the Patched gene fails to suppress carcinogenesis in Gorlin syndrome
    M Laimer
    Division of Molecular Dermatology, Department of Dermatology, General Hospital Salzburg, Paracelsus Private Medical University Salzburg, Muellner Hauptstrasse 48, 5020 Salzburg, Austria
    Br J Dermatol 159:222-7. 2008
  6. ncbi Cytokeratin 8 interacts with clumping factor B: a new possible virulence factor target
    M Haim
    Division of Molecular Dermatology, Department of Dermatology, University Hospital, Paracelsus Medical University, 5020 Salzburg, Austria
    Microbiology 156:3710-21. 2010
  7. ncbi Introducing a fast and simple PCR-RFLP analysis for the detection of mutant thiopurine S-methyltransferase alleles TPMT*3A and TPMT*3C
    K Oender
    Department of Dermatology, Paracelsus Private Medical University, Salzburg, Austria
    J Eur Acad Dermatol Venereol 20:396-400. 2006
  8. ncbi Aberrant heterodimerization of keratin 16 with keratin 6A in HaCaT keratinocytes results in diminished cellular migration
    A Trost
    Division of Molecular Dermatology, Department of Dermatology, Paracelsus Medical University Salzburg, Mullner Hauptstrasse 48, A 5020 Salzburg, Austria
    Mech Ageing Dev 131:346-53. 2010
  9. ncbi Relative quantitation of protein-protein interaction strength within the yeast two-hybrid system via fluorescence beta-galactosidase activity detection in a high-throughput and low-cost manner
    K Oender
    Division of Molecular Dermatology, Department of Dermatology, Paracelsus Private Medical University Salzburg, Salzburg, Austria
    Assay Drug Dev Technol 4:709-19. 2006
  10. ncbi Telepathology using immunofluorescence/immunoperoxidase microscopy
    C M Lanschuetzer
    Department of Dermatology, Paracelsus Private Medical University, Salzburg, Austria
    J Telemed Telecare 10:39-43. 2004

Collaborators

  • B Kofler
  • W Salmhofer
  • H Schaeppi
  • T Hashimoto
  • D Metze
  • H P Soyer
  • H Hintner
  • C M Lanschuetzer
  • A Trost
  • M Laimer
  • K Oender
  • M Haim
  • K Onder
  • M Emberger
  • A Klausegger
  • G Pohla-Gubo
  • C J Maier
  • P Desch
  • H A Reitsamer
  • V Wally
  • S Feichtner
  • G Achatz
  • R H Maier
  • P Schlager
  • S Selhofer
  • L Koller
  • K Richter
  • P Niedermayr
  • B Paulweber
  • H Hundsberger
  • A Diem
  • R Hametner
  • B Schafleitner
  • W H Muss

Detail Information

Publications12

  1. ncbi Large melanocytic nevi in hereditary epidermolysis bullosa
    J W Bauer
    Department of Dermatology, General Hospital Salzburg, Austria
    J Am Acad Dermatol 44:577-84. 2001
    ..Because nevi with these criteria do not fit in any of the known categories, we suggest the term EB nevi...
  2. ncbi Galanin family of peptides in skin function
    J W Bauer
    Department of Dermatology, SALK and Paracelsus Medical University Salzburg, Müller Hauptstrasse 48, 5020 Salzburg, Austria
    EXS 102:51-9. 2010
    ..We discuss these data in light of the role of the galanin peptide family in inflammation and cell proliferation...
  3. ncbi Epidermolysis bullosa naevi reveal a distinctive dermoscopic pattern
    C M Lanschuetzer
    Department of Dermatology, Paracelsus Private Medical University, Muellner Hauptstrasse 48, A-5020 Salzburg, Austria
    Br J Dermatol 153:97-102. 2005
    ..Nevertheless, as an unequivocal discrimination from malignant melanoma in vivo is sometimes not possible, regular clinical follow up of EB naevi with histopathological evaluation of highly suspicious lesions is mandatory...
  4. ncbi A localized variant of paraneoplastic pemphigus: acantholysis associated with malignant melanoma
    H Schaeppi
    Department of Dermatology, General Hospital Salzburg, Mullner Hauptstrasse 48, A 5020 Salzburg, Austria
    Br J Dermatol 144:1249-54. 2001
    ..We conclude that these autoantibodies, probably in conjunction with cofactors produced by the tumour, could play a part in the pathogenesis of this variant of FSA, for which we propose the term 'localized paraneoplastic pemphigus.'..
  5. ncbi Nonsense-associated altered splicing of the Patched gene fails to suppress carcinogenesis in Gorlin syndrome
    M Laimer
    Division of Molecular Dermatology, Department of Dermatology, General Hospital Salzburg, Paracelsus Private Medical University Salzburg, Muellner Hauptstrasse 48, 5020 Salzburg, Austria
    Br J Dermatol 159:222-7. 2008
    ..The pathogenetic alterations described give insights into the sequence of events leading to cellular transformation and underscore the importance of the PTCH protein in skin homeostasis...
  6. ncbi Cytokeratin 8 interacts with clumping factor B: a new possible virulence factor target
    M Haim
    Division of Molecular Dermatology, Department of Dermatology, University Hospital, Paracelsus Medical University, 5020 Salzburg, Austria
    Microbiology 156:3710-21. 2010
    ..Analysis with S. aureus strain Newman deficient in clumping factor B showed the clumping factor B-dependence of the interaction with CK8. We postulate that the clumping factor B-CK8 interaction is a novel factor in S. aureus infections...
  7. ncbi Introducing a fast and simple PCR-RFLP analysis for the detection of mutant thiopurine S-methyltransferase alleles TPMT*3A and TPMT*3C
    K Oender
    Department of Dermatology, Paracelsus Private Medical University, Salzburg, Austria
    J Eur Acad Dermatol Venereol 20:396-400. 2006
    ..To avoid laborious biochemical and sequencing assays, we evaluated a new restriction fragment length polymorphism (RFLP) analysis...
  8. ncbi Aberrant heterodimerization of keratin 16 with keratin 6A in HaCaT keratinocytes results in diminished cellular migration
    A Trost
    Division of Molecular Dermatology, Department of Dermatology, Paracelsus Medical University Salzburg, Mullner Hauptstrasse 48, A 5020 Salzburg, Austria
    Mech Ageing Dev 131:346-53. 2010
    ..Taken together, these data highlight the possibility of a physiological role for K6/K16 heterodimers in keratinocyte cell migration, in addition to the heterodimer's known functions in cell differentiation and mechanical resilience...
  9. ncbi Relative quantitation of protein-protein interaction strength within the yeast two-hybrid system via fluorescence beta-galactosidase activity detection in a high-throughput and low-cost manner
    K Oender
    Division of Molecular Dermatology, Department of Dermatology, Paracelsus Private Medical University Salzburg, Salzburg, Austria
    Assay Drug Dev Technol 4:709-19. 2006
    ....
  10. ncbi Telepathology using immunofluorescence/immunoperoxidase microscopy
    C M Lanschuetzer
    Department of Dermatology, Paracelsus Private Medical University, Salzburg, Austria
    J Telemed Telecare 10:39-43. 2004
    ....
  11. ncbi Galanin family of peptides in skin function
    J W Bauer
    Department of Dermatology, SALK and Paracelsus Medical University Salzburg, Muellner Hauptstrasse 48, 5020 Salzburg, Austria
    Cell Mol Life Sci 65:1820-5. 2008
    ..We discuss these data in light of the role of the galanin peptide family in inflammation and cell proliferation...
  12. ncbi Characteristic immunohistochemical and ultrastructural findings indicate that Kindler's syndrome is an apoptotic skin disorder
    C M Lanschuetzer
    Department of Dermatology, and Institute of Pathological Anatomy, General Hospital Salzburg, Salzburg, Austria
    J Cutan Pathol 30:553-60. 2003
    ..and provide evidence that Kindler's syndrome might primarily be an apoptotic disorder of basal keratinocytes...