Felicia Y T Yap

Summary

Country: Australia

Publications

  1. ncbi request reprint Advanced glycation end products as environmental risk factors for the development of type 1 diabetes
    Felicia Y T Yap
    BakerIDI Diabetes and Heart Institute, Melbourne, Victoria, Australia
    Curr Drug Targets 13:526-40. 2012
  2. pmc Advanced glycation end products are direct modulators of β-cell function
    Melinda T Coughlan
    Division of Diabetes Complications, Diabetes and Metabolism Division, Baker IDI Heart and Diabetes Institute, Melbourne, Australia
    Diabetes 60:2523-32. 2011
  3. doi request reprint Deletion of bone-marrow-derived receptor for AGEs (RAGE) improves renal function in an experimental mouse model of diabetes
    Greg Tesch
    Department of Nephrology, Monash Medical Centre, Monash Health, Clayton, Melbourne, Vic, Australia
    Diabetologia 57:1977-85. 2014
  4. doi request reprint Effects of high-density lipoprotein elevation with cholesteryl ester transfer protein inhibition on insulin secretion
    Andrew L Siebel
    Baker IDI Heart and Diabetes Institute, Melbourne, Australia
    Circ Res 113:167-75. 2013
  5. ncbi request reprint Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-alpha-dependent pathway
    Vicki Thallas-Bonke
    JDRF Albert Einstein Centre for Diabetes Complications, Diabetes and Metabolism Division, Baker Medical Research Institute, P O Box 6492, St Kilda Rd, Central, Melbourne, Victoria, Australia
    Diabetes 57:460-9. 2008

Collaborators

Detail Information

Publications5

  1. ncbi request reprint Advanced glycation end products as environmental risk factors for the development of type 1 diabetes
    Felicia Y T Yap
    BakerIDI Diabetes and Heart Institute, Melbourne, Victoria, Australia
    Curr Drug Targets 13:526-40. 2012
    ....
  2. pmc Advanced glycation end products are direct modulators of β-cell function
    Melinda T Coughlan
    Division of Diabetes Complications, Diabetes and Metabolism Division, Baker IDI Heart and Diabetes Institute, Melbourne, Australia
    Diabetes 60:2523-32. 2011
    ..Excess accumulation of advanced glycation end products (AGEs) contributes to aging and chronic diseases. We aimed to obtain evidence that exposure to AGEs plays a role in the development of type 1 diabetes (T1D)...
  3. doi request reprint Deletion of bone-marrow-derived receptor for AGEs (RAGE) improves renal function in an experimental mouse model of diabetes
    Greg Tesch
    Department of Nephrology, Monash Medical Centre, Monash Health, Clayton, Melbourne, Vic, Australia
    Diabetologia 57:1977-85. 2014
    ..The aim of this study was to determine whether deletion of RAGE from bone-marrow-derived cells influences the pathogenesis of experimental diabetic nephropathy...
  4. doi request reprint Effects of high-density lipoprotein elevation with cholesteryl ester transfer protein inhibition on insulin secretion
    Andrew L Siebel
    Baker IDI Heart and Diabetes Institute, Melbourne, Australia
    Circ Res 113:167-75. 2013
    ..High-density lipoprotein cholesterol elevation via cholesteryl ester transfer protein (CETP) inhibition represents a novel therapy for atherosclerosis, which also may have relevance for type 2 diabetes mellitus...
  5. ncbi request reprint Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-alpha-dependent pathway
    Vicki Thallas-Bonke
    JDRF Albert Einstein Centre for Diabetes Complications, Diabetes and Metabolism Division, Baker Medical Research Institute, P O Box 6492, St Kilda Rd, Central, Melbourne, Victoria, Australia
    Diabetes 57:460-9. 2008
    ..Since NADPH oxidase activation is closely linked to other putative pathways, its interaction with changes in protein kinase C (PKC) and increased advanced glycation was examined...