anak dharmapatni

Summary

Affiliation: The Universiity of Adelaide
Location: adelaide, australia
Summary:
Post Doc Researcher
Teaching in Health Sciences and Dentistry
Supervision of Post Grad Research Students

Publications

  1. ncbi TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis
    Anak A S S K Dharmapatni
    Discipline of Anatomy and Pathology, School of Medical Sciences, University of Adelaide, Frome Road, Adelaide, SA 5005, Australia
    Arthritis Res Ther 13:R51. 2011
  2. ncbi The expression of RANKL and OPG in the various grades of osteoarthritic cartilage
    Angus R Upton
    Discipline of Pathology, School of Medical Sciences, Faculty of Health Sciences, University of Adelaide, North Terrace, Level 3 Medical School North Building, Frome Road, Adelaide, SA 5005, Australia
    Rheumatol Int 32:535-40. 2012
  3. ncbi C-reactive protein in gingival crevicular fluid may be indicative of systemic inflammation
    Emma Megson
    School of Dentistry, Faculty of Health Sciences, University of Adelaide, Adelaide, South Australia, Australia
    J Clin Periodontol 37:797-804. 2010
  4. ncbi Expression of tumor necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor, fibroblast growth factor-inducible 14 protein (Fn14), in healthy tissues and in tissues affected by periodontitis
    N G Kataria
    Colgate Australian Clinical Dental Research Centre, School of Dentistry, University of Adelaide, Adelaide, SA, Australia
    J Periodontal Res 45:564-73. 2010
  5. ncbi Inhibition of apoptosis in periodontitis
    H Lucas
    Discipline of Pathology, School of Medical Sciences, University of Adelaide, Adelaide, SA 5005, Australia
    J Dent Res 89:29-33. 2010
  6. ncbi The correlation of RANK, RANKL and TNFalpha expression with bone loss volume and polyethylene wear debris around hip implants
    Christopher A Holding
    Department of Pathology, The University of Adelaide, North Terrace, Adelaide, South Australia 5000, Australia
    Biomaterials 27:5212-9. 2006
  7. ncbi Histopathological and ultrastructural features of dermal telangiectasias in systemic sclerosis
    Jennifer G Walker
    Department of Immunology, Allergy and Arthritis, Flinders Medical Centre, Adelaide, South Australia, Australia
    Pathology 37:220-5. 2005
  8. ncbi The TGF beta receptor endoglin in systemic sclerosis
    A A Dharmapatni
    Department of Immunology, Allergy, and Arthritis, Flinders Medical Centre, Bedford Park, South Australia
    Asian Pac J Allergy Immunol 19:275-82. 2001
  9. ncbi Radiochemical assay of glutathione S-epoxide transferase and its enhancement by phenobarbital in rat liver in vivo
    J Marniemi
    Biochem Pharmacol 24:1569-72. 1975
  10. ncbi Comparison between procaine and isocarboxazid metabolism in vitro by a liver microsomal amidase-esterase
    K Moroi
    Biochem Pharmacol 24:1517-21. 1975

Collaborators

Detail Information

Publications18

  1. ncbi TWEAK and Fn14 expression in the pathogenesis of joint inflammation and bone erosion in rheumatoid arthritis
    Anak A S S K Dharmapatni
    Discipline of Anatomy and Pathology, School of Medical Sciences, University of Adelaide, Frome Road, Adelaide, SA 5005, Australia
    Arthritis Res Ther 13:R51. 2011
    ..Effects of sTWEAK on osteoclasts and osteoblasts were investigated in vitro...
  2. ncbi The expression of RANKL and OPG in the various grades of osteoarthritic cartilage
    Angus R Upton
    Discipline of Pathology, School of Medical Sciences, Faculty of Health Sciences, University of Adelaide, North Terrace, Level 3 Medical School North Building, Frome Road, Adelaide, SA 5005, Australia
    Rheumatol Int 32:535-40. 2012
    ..The changes seen in the different grades of tissue may also indicate that this effect occurs during the early stages of OA development...
  3. ncbi C-reactive protein in gingival crevicular fluid may be indicative of systemic inflammation
    Emma Megson
    School of Dentistry, Faculty of Health Sciences, University of Adelaide, Adelaide, South Australia, Australia
    J Clin Periodontol 37:797-804. 2010
    ..Although the liver is the primary source of CRP, extra-hepatic production of CRP has been reported. This study aimed to determine whether CRP in GCF is produced locally in the gingivae...
  4. ncbi Expression of tumor necrosis factor-like weak inducer of apoptosis (TWEAK) and its receptor, fibroblast growth factor-inducible 14 protein (Fn14), in healthy tissues and in tissues affected by periodontitis
    N G Kataria
    Colgate Australian Clinical Dental Research Centre, School of Dentistry, University of Adelaide, Adelaide, SA, Australia
    J Periodontal Res 45:564-73. 2010
    ..The aim of the present study was to investigate the expression of TWEAK and Fn14 at the protein and mRNA levels in gingival biopsies from periodontitis patients and from clinically healthy patients...
  5. ncbi Inhibition of apoptosis in periodontitis
    H Lucas
    Discipline of Pathology, School of Medical Sciences, University of Adelaide, Adelaide, SA 5005, Australia
    J Dent Res 89:29-33. 2010
    ..Similar changes were seen at the level of mRNA. The results indicate that apoptosis in periodontitis may be inhibited by elevated expression of TRAIL decoy receptors and cleaved caspase-3 inhibitors...
  6. ncbi The correlation of RANK, RANKL and TNFalpha expression with bone loss volume and polyethylene wear debris around hip implants
    Christopher A Holding
    Department of Pathology, The University of Adelaide, North Terrace, Adelaide, South Australia 5000, Australia
    Biomaterials 27:5212-9. 2006
    ..This suggests that the interaction of TNFalpha and RANKL promotes osteoclast activity associated with polyethylene wear and therapies targeting TNF activity may be useful to treat peri-implant osteolysis...
  7. ncbi Histopathological and ultrastructural features of dermal telangiectasias in systemic sclerosis
    Jennifer G Walker
    Department of Immunology, Allergy and Arthritis, Flinders Medical Centre, Adelaide, South Australia, Australia
    Pathology 37:220-5. 2005
    ..It would be of interest to examine telangiectasias in the early phase of their formation. Alternatively, other explanations need to be explored in understanding the aetiopathogenesis of telangiectasia in scleroderma...
  8. ncbi The TGF beta receptor endoglin in systemic sclerosis
    A A Dharmapatni
    Department of Immunology, Allergy, and Arthritis, Flinders Medical Centre, Bedford Park, South Australia
    Asian Pac J Allergy Immunol 19:275-82. 2001
    ..The functional significance of this upregulation is uncertain but may reflect endothelial activation in scleroderma...
  9. ncbi Radiochemical assay of glutathione S-epoxide transferase and its enhancement by phenobarbital in rat liver in vivo
    J Marniemi
    Biochem Pharmacol 24:1569-72. 1975
  10. ncbi Comparison between procaine and isocarboxazid metabolism in vitro by a liver microsomal amidase-esterase
    K Moroi
    Biochem Pharmacol 24:1517-21. 1975
  11. ncbi Maturation of the adrenal medulla--IV. Effects of morphine
    T R Anderson
    Biochem Pharmacol 24:1469-74. 1975
  12. ncbi Studies of oxygen binding energy to hemoglobin molecule
    Y W Chow
    Biochem Biophys Res Commun 66:1424-31. 1975
  13. ncbi Atomic models for the polypeptide backbones of myohemerythrin and hemerythrin
    W A Hendrickson
    Biochem Biophys Res Commun 66:1349-56. 1975
  14. ncbi Effect of chloroquine on cultured fibroblasts: release of lysosomal hydrolases and inhibition of their uptake
    U N Wiesmann
    Biochem Biophys Res Commun 66:1338-43. 1975
  15. ncbi Metal substitutions incarbonic anhydrase: a halide ion probe study
    R J Smith
    Biochem Biophys Res Commun 66:1281-6. 1975
  16. ncbi Delineation of the intimate details of the backbone conformation of pyridine nucleotide coenzymes in aqueous solution
    K S Bose
    Biochem Biophys Res Commun 66:1173-9. 1975
  17. ncbi Formate assay in body fluids: application in methanol poisoning
    A B Makar
    Biochem Med 13:117-26. 1975
  18. ncbi Elevated expression of caspase-3 inhibitors, survivin and xIAP correlates with low levels of apoptosis in active rheumatoid synovium
    Anak A S S K Dharmapatni
    Discipline of Pathology, School of Medical Sciences, Faculty of Health Sciences, University of Adelaide, North Terrace, Adelaide, 5005 South Australia, Australia
    Arthritis Res Ther 11:R13. 2009
    ..Tumour necrosis factor-related apoptosis-inducing ligand (TRAIL) is a tumour necrosis factor (TNF) family member capable of inducing apoptosis in many cell types...