Research Topics
Species | Christopher V DayasSummaryAffiliation: University of Newcastle Country: Australia Publications
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Detail Information
Publications
Cocaine potentiates excitatory drive in the perifornical/lateral hypothalamusJiann Wei Yeoh
1School of Biomedical Sciences and Pharmacy, and the Centre for Translational Neuroscience and Mental Health Research, University of Newcastle, Callaghan, NSW 2308, Australia
J Physiol 590:3677-89. 2012....
Stimuli linked to ethanol availability activate hypothalamic CART and orexin neurons in a reinstatement model of relapseChristopher V Dayas
Molecular and Integrative Neurosciences Department, The Scripps Research Institute, La Jolla, CA, USA
Biol Psychiatry 63:152-7. 2008..However, the potential role of other hypothalamic feeding peptides, such as cocaine- and amphetamine-regulated transcript (CART), in conditioned reinstatement has yet to be explored...
Distinct patterns of neural activation associated with ethanol seeking: effects of naltrexoneChristopher V Dayas
Molecular and Integrative Neurosciences Department, The Scripps Research Institute, La Jolla, California 92037, USA
Biol Psychiatry 61:979-89. 2007....
Activation of group II metabotropic glutamate receptors attenuates both stress and cue-induced ethanol-seeking and modulates c-fos expression in the hippocampus and amygdalaYu Zhao
Molecular and Integrative Neurosciences Department, The Scripps Research Institute, La Jolla, California 92037, USA
J Neurosci 26:9967-74. 2006..The results implicate group II mGluRs as a shared neuropharmacological substrate for ethanol-seeking elicited by both drug cues and stress and identify group II mGluRs as promising treatment targets for relapse prevention...
Hypothalamic paraventricular nucleus neurons regulate medullary catecholamine cell responses to restraint stressChristopher V Dayas
Department of Physiology and Pharmacology, School of Biomedical Sciences, University of Queensland, Brisbane, Queensland 4072, Australia
J Comp Neurol 478:22-34. 2004..Considered in concert with previous results, these data suggest that the activation of medullary CA cells in response to psychological stress may involve a critical input from the PVN...
