L Iannazzo

Summary

Affiliation: RMIT University
Country: Australia

Publications

  1. pmc The structural requirements for phorbol esters to enhance serotonin and acetylcholine release from rat brain cortex
    L Iannazzo
    Prince Henry s Institute of Medical Research, Victoria, Australia
    Br J Pharmacol 127:1177-89. 1999
  2. ncbi request reprint M(2)/M(4)-muscarinic receptors mediate automodulation of acetylcholine outflow from mouse cortex
    L Iannazzo
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Neurosci Lett 287:129-32. 2000
  3. ncbi request reprint Modulation of acetylcholine release from mouse cortex by protein kinase C: dependence on stimulation intensity
    L Iannazzo
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Life Sci 67:31-8. 2000
  4. pmc No involvement of nicotinic receptors in the facilitation of acetylcholine outflow in mouse cortex in the presence of neostigmine and atropine
    L Iannazzo
    Prince Henry s Institute of Medical Research, P O Box 5152, Clayton, Victoria, Australia 3168
    Br J Pharmacol 130:2008-14. 2000
  5. ncbi request reprint Involvement of B-50 (GAP-43) phosphorylation in the modulation of transmitter release by protein kinase C
    L Iannazzo
    RMIT University, School of Medical Sciences, Bundoora, Victoria, Australia
    Clin Exp Pharmacol Physiol 28:901-4. 2001
  6. ncbi request reprint Protein kinase C and transmitter release
    H Majewski
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Clin Exp Pharmacol Physiol 24:619-23. 1997
  7. ncbi request reprint Protein kinase C: a physiological mediator of enhanced transmitter output
    H Majewski
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Prog Neurobiol 55:463-75. 1998

Collaborators

Detail Information

Publications7

  1. pmc The structural requirements for phorbol esters to enhance serotonin and acetylcholine release from rat brain cortex
    L Iannazzo
    Prince Henry s Institute of Medical Research, Victoria, Australia
    Br J Pharmacol 127:1177-89. 1999
    ..Some compounds notably PDA, PMBA, dPPhen, dPPhenA had different potencies across noradrenaline, dopamine and serotonin...
  2. ncbi request reprint M(2)/M(4)-muscarinic receptors mediate automodulation of acetylcholine outflow from mouse cortex
    L Iannazzo
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Neurosci Lett 287:129-32. 2000
    ..Linear regression analysis revealed that the data fitted the M(2) (r(2)>0.97) and M(4) (r(2)>0.85) subtypes best, with no correlation for the M(1) and M(3) subtypes...
  3. ncbi request reprint Modulation of acetylcholine release from mouse cortex by protein kinase C: dependence on stimulation intensity
    L Iannazzo
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Life Sci 67:31-8. 2000
    ..It may be that these inhibitors do not effectively block PKC in this functional system. Indeed, polymyxin B completely blocked the facilitatory effect of PDB but Ro 318220 was without effect...
  4. pmc No involvement of nicotinic receptors in the facilitation of acetylcholine outflow in mouse cortex in the presence of neostigmine and atropine
    L Iannazzo
    Prince Henry s Institute of Medical Research, P O Box 5152, Clayton, Victoria, Australia 3168
    Br J Pharmacol 130:2008-14. 2000
    ....
  5. ncbi request reprint Involvement of B-50 (GAP-43) phosphorylation in the modulation of transmitter release by protein kinase C
    L Iannazzo
    RMIT University, School of Medical Sciences, Bundoora, Victoria, Australia
    Clin Exp Pharmacol Physiol 28:901-4. 2001
    ..Therefore, it is possible that the phosphorylation status of B-50 directly influences the organization of actin filaments, thereby allowing transmitter output to be sustained under high levels of stimulation...
  6. ncbi request reprint Protein kinase C and transmitter release
    H Majewski
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Clin Exp Pharmacol Physiol 24:619-23. 1997
    ..Activation of PKC represents one of the most powerful ways to enhance transmitter release and may have therapeutic potential...
  7. ncbi request reprint Protein kinase C: a physiological mediator of enhanced transmitter output
    H Majewski
    Prince Henry s Institute of Medical Research, Clayton, Victoria, Australia
    Prog Neurobiol 55:463-75. 1998
    ....