F Asmus

Summary

Publications

  1. doi request reprint Dystonia-plus syndromes
    F Asmus
    Department of Neurodegenerative Disease, Hertie Institute for Clinical Brain Research, University of Tuebingen, Tuebingen, Germany
    Eur J Neurol 17:37-45. 2010
  2. doi request reprint Reverse sensory geste in cervical dystonia
    Friedrich Asmus
    Department of Neurodegenerative Diseases, Center of Neurology, Hertie Institute for Clinical Brain Research, Tuebingen, Germany
    Mov Disord 24:297-300. 2009
  3. doi request reprint Kick and rush: paradoxical kinesia in Parkinson disease
    Friedrich Asmus
    Dystonia Genetics Unit, Hertie Institute for Clinical Brain Research, Center of Neurology, University of Tuebingen, Otfried Müller Str 27, 4th Floor, D 72076 Tuebingen, Germany
    Neurology 71:695. 2008
  4. ncbi request reprint Strong genetic evidence for association of TOR1A/TOR1B with idiopathic dystonia
    C Kamm
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tuebingen, Hoppe Seyler Str 3, 72086 Tuebingen, Germany
    Neurology 67:1857-9. 2006
  5. ncbi request reprint A novel TITF-1 mutation causes benign hereditary chorea with response to levodopa
    F Asmus
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, Tuebingen, Germany
    Neurology 64:1952-4. 2005
  6. doi request reprint A novel clinical syndrome revealing a deficiency of the muscarinic M3 receptor
    J K Pomper
    Department of Cognitive Neurology, Hertie Institute for Clinical Brain Research, University of Tubingen, 72076 Tubingen, Germany
    Neurology 76:451-5. 2011
  7. ncbi request reprint The role of alpha-synuclein gene multiplications in early-onset Parkinson's disease and dementia with Lewy bodies
    A Hofer
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tubingen, Germany
    J Neural Transm 112:1249-54. 2005
  8. ncbi request reprint Excellent response to apomorphine in Parkinsonism with optic atrophy unresponsive to oral antiparkinsonian medication
    Angela Deutschlander
    Department of Neurology, Klinikum Grosshadern, Ludwig Maximilians University, Munich, Germany
    Mov Disord 21:1523-5. 2006
  9. doi request reprint Ultrasound-guided injection of the iliopsoas muscle with botulinum toxin in camptocormia
    Rainer von Coelln
    Department for Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tuebingen, Tuebingen, Germany
    Mov Disord 23:889-92. 2008
  10. ncbi request reprint PARK11 is not linked with Parkinson's disease in European families
    Jürgen Prestel
    Hertie Institute for Clinical Brain Research, Department of Neurodegenerative Diseases, University of Tuebingen, Hoppe Seyler Str 3, 72076 Tuebingen, Germany
    Eur J Hum Genet 13:193-7. 2005

Collaborators

Detail Information

Publications12

  1. doi request reprint Dystonia-plus syndromes
    F Asmus
    Department of Neurodegenerative Disease, Hertie Institute for Clinical Brain Research, University of Tuebingen, Tuebingen, Germany
    Eur J Neurol 17:37-45. 2010
    ..Recently, a novel form of dystonia-parkinsonism (DYT16) has been found to be linked to mutations in the PRKRA gene, whose relation to basal ganglia disorders is yet unknown ...
  2. doi request reprint Reverse sensory geste in cervical dystonia
    Friedrich Asmus
    Department of Neurodegenerative Diseases, Center of Neurology, Hertie Institute for Clinical Brain Research, Tuebingen, Germany
    Mov Disord 24:297-300. 2009
    ..Assessment of rSG should be included in the routine examination of CD patients, since BTX treatment may have to beadjusted accordingly to be efficacious...
  3. doi request reprint Kick and rush: paradoxical kinesia in Parkinson disease
    Friedrich Asmus
    Dystonia Genetics Unit, Hertie Institute for Clinical Brain Research, Center of Neurology, University of Tuebingen, Otfried Müller Str 27, 4th Floor, D 72076 Tuebingen, Germany
    Neurology 71:695. 2008
  4. ncbi request reprint Strong genetic evidence for association of TOR1A/TOR1B with idiopathic dystonia
    C Kamm
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tuebingen, Hoppe Seyler Str 3, 72086 Tuebingen, Germany
    Neurology 67:1857-9. 2006
    ..000008, in a larger cohort of German and Austrian patients with predominantly focal sporadic dystonia...
  5. ncbi request reprint A novel TITF-1 mutation causes benign hereditary chorea with response to levodopa
    F Asmus
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, Tuebingen, Germany
    Neurology 64:1952-4. 2005
    ..Surprisingly, treatment with levodopa improved gait dramatically and reduced chorea in two patients. Dopaminergic drugs should be considered a useful therapeutic option in BHC...
  6. doi request reprint A novel clinical syndrome revealing a deficiency of the muscarinic M3 receptor
    J K Pomper
    Department of Cognitive Neurology, Hertie Institute for Clinical Brain Research, University of Tubingen, 72076 Tubingen, Germany
    Neurology 76:451-5. 2011
    ..No clinical disorders have been caused by dysfunction of any of the 5 subtypes (M1-M5) of muscarinic receptors. We present a patient with a novel clinical syndrome that we suggest results from a deficiency of the muscarinic M3 receptor...
  7. ncbi request reprint The role of alpha-synuclein gene multiplications in early-onset Parkinson's disease and dementia with Lewy bodies
    A Hofer
    Department of Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tubingen, Germany
    J Neural Transm 112:1249-54. 2005
    ..A triplication of the alpha-synuclein gene was found to cause autosomal dominant Lewy body disease in two distinct families...
  8. ncbi request reprint Excellent response to apomorphine in Parkinsonism with optic atrophy unresponsive to oral antiparkinsonian medication
    Angela Deutschlander
    Department of Neurology, Klinikum Grosshadern, Ludwig Maximilians University, Munich, Germany
    Mov Disord 21:1523-5. 2006
    ..The severe motor symptoms of both patients were conspicuously alleviated by apomorphine delivered subcutaneously. One patient underwent bilateral subthalamic nucleus deep brain stimulation, which also proved effective...
  9. doi request reprint Ultrasound-guided injection of the iliopsoas muscle with botulinum toxin in camptocormia
    Rainer von Coelln
    Department for Neurodegenerative Diseases, Hertie Institute for Clinical Brain Research, University of Tuebingen, Tuebingen, Germany
    Mov Disord 23:889-92. 2008
    ..In conclusion, injection of BTX into the iliopsoas does not appear to be a promising approach for the treatment of parkinsonism-associated camptocormia...
  10. ncbi request reprint PARK11 is not linked with Parkinson's disease in European families
    Jürgen Prestel
    Hertie Institute for Clinical Brain Research, Department of Neurodegenerative Diseases, University of Tuebingen, Hoppe Seyler Str 3, 72076 Tuebingen, Germany
    Eur J Hum Genet 13:193-7. 2005
    ..Therefore, PARK11 does not seem to play a major role for familial PD in the European population...
  11. pmc The PARK8 locus in autosomal dominant parkinsonism: confirmation of linkage and further delineation of the disease-containing interval
    Alexander Zimprich
    Hertie Institute for Clinical Brain Research, Department for Neurodegenerative Diseases, University of Tuebingen, Tuebingen, Germany
    Am J Hum Genet 74:11-9. 2004
    ..7 Mb). Our data provide evidence that the PARK8 locus is responsible for the disease in a subset of families of white ancestry with autosomal dominant parkinsonism, suggesting that it could be a more common locus...
  12. ncbi request reprint Point mutations in exon 1 of the NR4A2 gene are not a major cause of familial Parkinson's disease
    Alexander Zimprich
    Neurogenetics 4:219-20. 2003