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Genomes and Genes | CG8188SummaryGene Symbol: CG8188 Description: CG8188 gene product from transcript CG8188-RB Alias: Dmel\CG8188, Ubc, CG8188-PA, CG8188-PB Species: fruit fly Top Publications
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Publications
DmPAR-6 directs epithelial polarity and asymmetric cell division of neuroblasts in DrosophilaM Petronczki
Research Institute of Molecular Pathology, Dr Bohr Gasse 7, A 1030 Vienna, Austria
Nat Cell Biol 3:43-9. 2001....
Photic signaling by cryptochrome in the Drosophila circadian systemF J Lin
Department of Neuroscience, Howard Hughes Medical Institute, University of Pennsylvania Medical School, Philadelphia, Pennsylvania 19104, USA
Mol Cell Biol 21:7287-94. 2001..High levels of CRY block TIM ubiquitination, suggesting a mechanism by which light-driven changes in CRY could control TIM ubiquitination...
Tumor suppressor CYLD regulates JNK-induced cell death in DrosophilaLei Xue
Howard Hughes Medical Institute, Department of Genetics, Yale University School of Medicine, New Haven, CT 06536, USA
Dev Cell 13:446-54. 2007..These data provide a molecular mechanism for the tumor suppressor function of this evolutionary conserved molecule by indicating that dCYLD plays a critical role in modulating TNF-JNK-mediated cell death...
Downregulation of vertebrate Tel (ETV6) and Drosophila Yan is facilitated by an evolutionarily conserved mechanism of F-box-mediated ubiquitinationM Guy Roukens
Leiden University Medical Center LUMC, Department of Molecular Cell Biology, 2300 RC Leiden, The Netherlands
Mol Cell Biol 28:4394-406. 2008..Here, we found that Fbl6 regulates Tel/Yan protein stability and allows appropriate spatiotemporal control of gene expression by these repressors...
Regulation of glia number in Drosophila by Rap/Fzr, an activator of the anaphase-promoting complex, and Loco, an RGS proteinMargarita E Kaplow
Department of Biology, City College and The Graduate Center, City University of New York, New York 10031, USA
Genetics 178:2003-16. 2008..We propose that Rap/Fzr targets Loco for ubiquitination, thereby regulating glial differentiation in the developing nervous system...
Genome-wide analysis reveals MOF as a key regulator of dosage compensation and gene expression in DrosophilaJop Kind
Gene Expression Unit, European Molecular Biology Laboratory, Meyerhofstrasse 1, 69117 Heidelberg, Germany
Cell 133:813-28. 2008..Therefore, MOF is not only involved in the onset of dosage compensation, but also acts as a regulator of gene expression in the Drosophila genome...
Identification of domains responsible for ubiquitin-dependent degradation of dMyc by glycogen synthase kinase 3beta and casein kinase 1 kinasesMargherita Galletti
Department of Biology, City College of the City University of New York, 138th at Convent Ave, New York, NY 10031, USA
Mol Cell Biol 29:3424-34. 2009..Expression of the dMyc mutants in the compound eye of the adult fly results in a visible defect that is attributed to the effect of dMyc on growth, cell death, and inhibition of ommatidial differentiation...
NMNAT suppresses tau-induced neurodegeneration by promoting clearance of hyperphosphorylated tau oligomers in a Drosophila model of tauopathyYousuf O Ali
Department of Molecular and Cellular Pharmacology, Miller School of Medicine, University of Miami, Miami, FL 33136, USA
Hum Mol Genet 21:237-50. 2012..Our report on the molecular basis of NMNAT-mediated neuroprotection in tauopathies opens future investigation of this factor in other protein foldopathies...
