David Long

Summary

Affiliation: Wake Forest University School of Medicine
Country: USA

Publications

  1. ncbi Human articular chondrocytes produce IL-7 and respond to IL-7 with increased production of matrix metalloproteinase-13
    David Long
    Section of Molecular Medicine, Department of Internal Medicine, Wake Forest University School of Medicine, Medical Center Blvd, Winston Salem, North Carolina 27157, USA
    Arthritis Res Ther 10:R23. 2008
  2. ncbi Interleukin-7 stimulates secretion of S100A4 by activating the JAK/STAT signaling pathway in human articular chondrocytes
    Raghunatha R Yammani
    Wake Forest University School of Medicine, Winston Salem, North Carolina 27157, USA
    Arthritis Rheum 60:792-800. 2009
  3. ncbi Increased expression of the Akt/PKB inhibitor TRB3 in osteoarthritic chondrocytes inhibits insulin-like growth factor 1-mediated cell survival and proteoglycan synthesis
    John D Cravero
    Wake Forest University School of Medicine, Winston Salem, North Carolina, USA
    Arthritis Rheum 60:492-500. 2009

Detail Information

Publications3

  1. ncbi Human articular chondrocytes produce IL-7 and respond to IL-7 with increased production of matrix metalloproteinase-13
    David Long
    Section of Molecular Medicine, Department of Internal Medicine, Wake Forest University School of Medicine, Medical Center Blvd, Winston Salem, North Carolina 27157, USA
    Arthritis Res Ther 10:R23. 2008
    ..The purpose of this study was to discover novel mediators of cartilage destruction using fibronectin fragments as a stimulus...
  2. ncbi Interleukin-7 stimulates secretion of S100A4 by activating the JAK/STAT signaling pathway in human articular chondrocytes
    Raghunatha R Yammani
    Wake Forest University School of Medicine, Winston Salem, North Carolina 27157, USA
    Arthritis Rheum 60:792-800. 2009
    ..The aim of this study was to examine the mechanism of S100A4 secretion by chondrocytes...
  3. ncbi Increased expression of the Akt/PKB inhibitor TRB3 in osteoarthritic chondrocytes inhibits insulin-like growth factor 1-mediated cell survival and proteoglycan synthesis
    John D Cravero
    Wake Forest University School of Medicine, Winston Salem, North Carolina, USA
    Arthritis Rheum 60:492-500. 2009
    ..This study was undertaken to determine if TRB3 is expressed in chondrocytes, and whether the chondrocyte response to IGF-1 is reduced by TRB3...