Research Topics
| M ShiotaSummaryAffiliation: Vanderbilt University Country: USA Publications
| Collaborators
|
Detail Information
Publications
Glucokinase gene locus transgenic mice are resistant to the development of obesity-induced type 2 diabetesM Shiota
Department of Molecular Physiology and Biophysics, School of Medicine, Vanderbilt University, Nashville, Tennessee 37232 0615, USA
Diabetes 50:622-9. 2001..Together, these data indicate that increased GK levels dramatically lessen the development of both hyperglycemia and hyperinsulinemia associated with the feeding of an HF diet...
Small amounts of fructose markedly augment net hepatic glucose uptake in the conscious dogM Shiota
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee 37232 0615, USA
Diabetes 47:867-73. 1998..These data suggest that glucokinase translocation within the hepatocyte is a major determinant of hepatic glucose uptake by the dog in vivo...
Combined intraportal infusion of acetylcholine and adrenergic blockers augments net hepatic glucose uptakeM Shiota
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, TN 37232 0615, USA
Am J Physiol Endocrinol Metab 278:E544-52. 2000..4 +/- 0.6 and 4.6 +/- 0.6 mg. kg(-1). min(-1) by 220 and 300 min, respectively. These data are consistent with the hypothesis that alterations in nerve activity contribute to the increase in NHGU seen after portal glucose delivery...
Inclusion of low amounts of fructose with an intraduodenal glucose load markedly reduces postprandial hyperglycemia and hyperinsulinemia in the conscious dogMasakazu Shiota
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee 37232 0615, USA
Diabetes 51:469-78. 2002..As a result, postprandial hyperglycemia and insulin release by the pancreatic beta-cell were reduced. In conclusion, catalytic amounts of fructose have the ability to improve glucose tolerance...
Cell-specific roles of glucokinase in glucose homeostasisC Postic
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA
Recent Prog Horm Res 56:195-217. 2001..Moreover, whether the pathophysiology of PHHI also involves multitissue dysfunction remains to be explored...
Phosphoenolpyruvate carboxykinase is necessary for the integration of hepatic energy metabolismP She
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA
Mol Cell Biol 20:6508-17. 2000..Thus, hepatic PEPCK may function more as an integrator of hepatic energy metabolism than as a determinant of gluconeogenesis...
Dual roles for glucokinase in glucose homeostasis as determined by liver and pancreatic beta cell-specific gene knock-outs using Cre recombinaseC Postic
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA
J Biol Chem 274:305-15. 1999..Interestingly, hepatic GK knock-out mice also have impaired insulin secretion in response to glucose. These studies indicate that deficiencies in both beta cell and hepatic GK contribute to the hyperglycemia of MODY-2...
Pancreatic response to mild non-insulin-induced hypoglycemia does not involve extrinsic neural inputS M Sherck
Department of Molecular Physiology and Biophysics, Vanderbilt University, Nashville, Tennessee 37232 0615, USA
Diabetes 50:2487-96. 2001..It thus can be concluded that the responses of alpha- and beta-cells to mild non-insulin-induced hypoglycemia can occur in the absence of extrinsic neural input...
Substrate-induced nuclear export and peripheral compartmentalization of hepatic glucokinase correlates with glycogen depositionT L Jetton
Department of Molecular Physiology and Biophysics, Vanderbilt University School of Medicine, Nashville, TN 37232 0615, USA
Int J Exp Diabetes Res 2:173-86. 2001..These data suggest that a compartmentalization exists of both active GK and the initial sites of glycogen deposition at the hepatocyte surface...
Mice lacking alpha-calcitonin gene-related peptide exhibit normal cardiovascular regulation and neuromuscular developmentJ T Lu
Department of Pharmacology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA
Mol Cell Neurosci 14:99-120. 1999..These results suggest that alphaCGRP is not required for the systemic regulation of cardiovascular hemodynamics or development of the neuromuscular junction...
Engineering physiologically regulated insulin secretion in non-beta cells by expressing glucagon-like peptide 1 receptorL Wu
Department of Medicine, Vanderbilt University Medical Center, Nashville, TN 37232, USA
Gene Ther 10:1712-20. 2003..These results demonstrate a new approach to engineer physiologically regulated insulin secretion by non-beta cells...
Identification of the gene variations in human CD22Y Hatta
Department of Human Genetics, Graduate School of Medicine, University of Tokyo, 7 3 1 Hongo, Bunkyo ku, Tokyo 113 0033, Japan
Immunogenetics 49:280-6. 1999..These findings indicate that a number of genetic variants are present in CD22, and suggest that CD22 could be considered a candidate for the susceptibility genes to autoimmune diseases...
Endotoxin modulates arachidonic acid-induced glycogenolysis in the perfused rat liverK Kimura
Department of Biomedical Sciences, Graduate School of Veterinary Medicine, Hokkaido University, Sapporo, Japan
Horm Metab Res 30:178-81. 1998..The release of Tx B2 by AA increased after LPS-injection. These results suggest that priming of hepatic macrophage by endotoxin in vivo enhances Tx synthesis, resulting in modulating hepatic glycogenolysis...
Characterization of the transforming activity of p80, a hyperphosphorylated protein in a Ki-1 lymphoma cell line with chromosomal translocation t(2;5)J Fujimoto
Department of Oncology, University of Tokyo, Japan
Proc Natl Acad Sci U S A 93:4181-6. 1996..Association of these mutants with GRB2 was still observed, suggesting that interaction of p80 with GRB2 but not with Shc or IRS-1 was relevant for cell transformation...
