Steven K Lundy

Summary

Affiliation: University of Michigan
Country: USA

Publications

  1. ncbi Reduced Fas ligand-expressing splenic CD5+ B lymphocytes in severe collagen-induced arthritis
    Steven K Lundy
    Department of Internal Medicine, Division of Rheumatology, University of Michigan Medical School, Ann Arbor, MI 48109 2200, USA
    Arthritis Res Ther 11:R128. 2009
  2. ncbi Molecular interactions between T cells and fibroblast-like synoviocytes: role of membrane tumor necrosis factor-alpha on cytokine-activated T cells
    Chinh N Tran
    Department of Internal Medicine, Division of Rheumatology, Rheumatic Disease Core Center, University of Michigan Medical School, Ann Arbor, Michigan, USA
    Am J Pathol 171:1588-98. 2007
  3. ncbi Cell-cell interactions in rheumatoid arthritis synovium
    David A Fox
    Division of Rheumatology and Rheumatic Diseases Research Core Center, 3918 Taubman Center, 1500 East Medical Center Drive, The University of Michigan, Ann Arbor, MI 48109, USA
    Rheum Dis Clin North Am 36:311-23. 2010
  4. ncbi CD8+ T cell contributions to allergen induced pulmonary inflammation and airway hyperreactivity
    Matthew A Schaller
    Department of Pathology, University of Michigan Medical School, Ann Arbor, MI 48109-0602, USA
    Eur J Immunol 35:2061-70. 2005
  5. ncbi Th17 cells in human disease
    Laura A Tesmer
    Department of Internal Medicine, Division of Rheumatology, Rheumatic Disease Core Center, University of Michigan, Ann Arbor, MI 48109 5358, USA
    Immunol Rev 223:87-113. 2008
  6. ncbi Presentation of arthritogenic peptide to antigen-specific T cells by fibroblast-like synoviocytes
    Chinh N Tran
    University of Michigan Rheumatic Disease Core Center, Ann Arbor, USA
    Arthritis Rheum 56:1497-506. 2007
  7. ncbi Cells of the synovium in rheumatoid arthritis. T lymphocytes
    Steven K Lundy
    Department of Internal Medicine, Division of Rheumatology and Rheumatic Diseases Core Center, University of Michigan Medical School, Ann Arbor, MI 48109 2200, USA
    Arthritis Res Ther 9:202. 2007
  8. ncbi High-throughput profiling of ion channel activity in primary human lymphocytes
    Daniel J Estes
    Department of Biomedical Engineering, University of Michigan, Ann Arbor, Michigan 48109, USA
    Anal Chem 80:3728-35. 2008
  9. ncbi Attenuation of allergen-induced responses in CCR6-/- mice is dependent upon altered pulmonary T lymphocyte activation
    Steven K Lundy
    Department of Pathology, University of Michigan Medical School, Ann Arbor, MI 48109, USA
    J Immunol 174:2054-60. 2005

Collaborators

Detail Information

Publications9

  1. ncbi Reduced Fas ligand-expressing splenic CD5+ B lymphocytes in severe collagen-induced arthritis
    Steven K Lundy
    Department of Internal Medicine, Division of Rheumatology, University of Michigan Medical School, Ann Arbor, MI 48109 2200, USA
    Arthritis Res Ther 11:R128. 2009
    ..The objective was to study immune regulation in a mouse model of rheumatoid arthritis that exhibits considerable heterogeneity of disease activity...
  2. ncbi Molecular interactions between T cells and fibroblast-like synoviocytes: role of membrane tumor necrosis factor-alpha on cytokine-activated T cells
    Chinh N Tran
    Department of Internal Medicine, Division of Rheumatology, Rheumatic Disease Core Center, University of Michigan Medical School, Ann Arbor, Michigan, USA
    Am J Pathol 171:1588-98. 2007
    ....
  3. ncbi Cell-cell interactions in rheumatoid arthritis synovium
    David A Fox
    Division of Rheumatology and Rheumatic Diseases Research Core Center, 3918 Taubman Center, 1500 East Medical Center Drive, The University of Michigan, Ann Arbor, MI 48109, USA
    Rheum Dis Clin North Am 36:311-23. 2010
    ..This article highlights selected cell-cell interactions in rheumatoid arthritis synovium that may be worthy of evaluation as future therapeutic targets...
  4. ncbi CD8+ T cell contributions to allergen induced pulmonary inflammation and airway hyperreactivity
    Matthew A Schaller
    Department of Pathology, University of Michigan Medical School, Ann Arbor, MI 48109-0602, USA
    Eur J Immunol 35:2061-70. 2005
    ..Altogether, these studies highlight a role for CD8(+) T lymphocytes in the development of allergen-induced airway responses...
  5. ncbi Th17 cells in human disease
    Laura A Tesmer
    Department of Internal Medicine, Division of Rheumatology, Rheumatic Disease Core Center, University of Michigan, Ann Arbor, MI 48109 5358, USA
    Immunol Rev 223:87-113. 2008
    ....
  6. ncbi Presentation of arthritogenic peptide to antigen-specific T cells by fibroblast-like synoviocytes
    Chinh N Tran
    University of Michigan Rheumatic Disease Core Center, Ann Arbor, USA
    Arthritis Rheum 56:1497-506. 2007
    ..To assess the ability of rheumatoid arthritis (RA) fibroblast-like synoviocytes (FLS) to function as antigen-presenting cells (APCs) for arthritogenic autoantigens found within inflamed joint tissues...
  7. ncbi Cells of the synovium in rheumatoid arthritis. T lymphocytes
    Steven K Lundy
    Department of Internal Medicine, Division of Rheumatology and Rheumatic Diseases Core Center, University of Michigan Medical School, Ann Arbor, MI 48109 2200, USA
    Arthritis Res Ther 9:202. 2007
    ..In light of the overwhelming evidence that disrupted T-cell homeostasis greatly contributes to joint pathology in RA, the therapeutic potential of targeting activators of pro-inflammatory T cells or their products is compelling...
  8. ncbi High-throughput profiling of ion channel activity in primary human lymphocytes
    Daniel J Estes
    Department of Biomedical Engineering, University of Michigan, Ann Arbor, Michigan 48109, USA
    Anal Chem 80:3728-35. 2008
    ..Moreover, this work presents an enabling methodology that can be applied widely, allowing high-throughput screening of specific voltage-gated ion channels in a variety of primary cells...
  9. ncbi Attenuation of allergen-induced responses in CCR6-/- mice is dependent upon altered pulmonary T lymphocyte activation
    Steven K Lundy
    Department of Pathology, University of Michigan Medical School, Ann Arbor, MI 48109, USA
    J Immunol 174:2054-60. 2005
    ..Altogether, the defect in CCR6-/- mice appears to be primarily due to an alteration in T cell activation, but also appears to include local pulmonary APC defects...