Research Topics
Species | David HorwitzSummaryAffiliation: University of Southern California Country: USA Publications
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Publications
Critical role of IL-2 and TGF-beta in generation, function and stabilization of Foxp3+CD4+ TregDavid A Horwitz
Division of Rheumatology and Immunology, Department of Medicine, University of Southern California Keck School of Medicine, Los Angeles, CA 90033, USA
Eur J Immunol 38:912-5. 2008..Unlike mouse CD4+CD25(-) cells, which rapidly become polyclonal Foxp3+CD25+ Treg when activated appropriately with IL-2 and TGF-beta, human T cells require multiple stimulations to become similar suppressor cells...
Natural and TGF-beta-induced Foxp3(+)CD4(+) CD25(+) regulatory T cells are not mirror images of each otherDavid A Horwitz
Division of Rheumatology and Immunology, Department of Medicine, University of Southern California Keck School of Medicine, Los Angeles, CA 90033, USA
Trends Immunol 29:429-35. 2008..Thus, nTregs and iTregs may have different roles in the adaptive immune response...
Regulatory T cells in systemic lupus erythematosus: past, present and futureDavid A Horwitz
Department of Medicine, Division of Rheumatology and Immunology, University of Southern California, Keck School of Medicine, Los Angeles, CA 90033, USA
Arthritis Res Ther 10:227. 2008..The role of immunogenic and tolerogenic dendritic cells in controlling Tregs is discussed, along with new strategies to normalize Treg function in systemic lupus erythematosus...
Regulatory T cells generated ex vivo as an approach for the therapy of autoimmune diseaseDavid A Horwitz
The Division of Rheumatology and Immunology, Department of Medicine, Keck School of Medicine of the University of Southern California, 2011 Zonal Avenue, HMR 711, Los Angeles, CA 90033, USA
Semin Immunol 16:135-43. 2004..These studies suggest that the adoptive transfer of CD4+ T regulatory cells generated ex vivo with IL-2 and TGF-beta as a treatment for autoimmune diseases may have sustained, long-term beneficial effects...
Natural and induced CD4+CD25+ cells educate CD4+CD25- cells to develop suppressive activity: the role of IL-2, TGF-beta, and IL-10Song Guo Zheng
Division of Rheumatology and Immunology, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, USA
J Immunol 172:5213-21. 2004..We suggest that the long-term effects of adoptively transferred natural-like CD4(+)CD25(+) regulatory cells induced ex vivo are due to their ability to generate new cytokine-producing CD4(+) regulatory T cells in vivo...
Generation ex vivo of TGF-beta-producing regulatory T cells from CD4+CD25- precursorsSong Guo Zheng
Division of Rheumatology and Immunology, Department of Medicine, Keck School of Medicine, University of Southern California, 2211 Zonal Avenue, Los Angeles, CA 90033, USA
J Immunol 169:4183-9. 2002..The ability to induce CD4(+) and CD8(+) cells to become regulatory cells ex vivo has the potential to be useful in the treatment of autoimmune diseases and to prevent transplant rejection...
The role of the combination of IL-2 and TGF-beta or IL-10 in the generation and function of CD4+ CD25+ and CD8+ regulatory T cell subsetsDavid A Horwitz
Division of Rheumatology and Immunology, Department of Medicine, Keck School of Medicine of the University of Southern California, Los Angeles 90033 1034, USA
J Leukoc Biol 74:471-8. 2003..Finally, the potential use of regulatory T cells generated ex vivo as an adoptive immunotherapy for certain autoimmune diseases, to prevent organ graft rejection, or to prevent pathologic host responses to infectious agents is discussed...
TGF-beta requires CTLA-4 early after T cell activation to induce FoxP3 and generate adaptive CD4+CD25+ regulatory cellsSong Guo Zheng
Division of Rheumatology and Immunology, Department of Medicine, University of Southern California, Keck School of Medicine, Los Angeles, CA 90033, USA
J Immunol 176:3321-9. 2006..Although natural CD4+CD25+ cells develop normally in CTLA-4(-/-) mice, the lack of TGF-beta-induced, peripheral CD4+CD25+ suppressor cells in these mice may contribute to their rapid demise...
Cutting edge: Foxp3+CD4+CD25+ regulatory T cells induced by IL-2 and TGF-beta are resistant to Th17 conversion by IL-6Song Guo Zheng
Division of Rheumatology and Immunology, Department of Medicine, Keck School of Medicine at University of Southern California, Los Angeles, CA 90033, USA
J Immunol 180:7112-6. 2008..The resistance of iTregs to Th17 conversion suggests that they can function more effectively than nTregs in an inflammatory milieu and emphasizes the central role of IL-2 in combination with TGF-beta to maintain immunologic homeostasis...
The potential of human regulatory T cells generated ex vivo as a treatment for lupus and other chronic inflammatory diseasesDavid A Horwitz
The Division of Rheumatology and Immunology, Department of Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA
Arthritis Res 4:241-6. 2002..This novel adoptive immunotherapy also has the potential to prevent the rejection of allogeneic transplants...
Cutting edge: all-trans retinoic acid sustains the stability and function of natural regulatory T cells in an inflammatory milieuXiaohui Zhou
Division of Rheumatology, University of South California, Los Angeles, CA 90033, USA
J Immunol 185:2675-9. 2010..We suggest that nTregs treated with atRA may represent a novel treatment strategy to control established chronic immune-mediated inflammatory diseases...
IL-2 is essential for TGF-beta to convert naive CD4+CD25- cells to CD25+Foxp3+ regulatory T cells and for expansion of these cellsSong Guo Zheng
Division of Rheumatology and Immunology, Department of Medicine, University of Southern California, Keck School of Medicine, Los Angeles, CA 90033, USA
J Immunol 178:2018-27. 2007....
Transfer of regulatory T cells generated ex vivo modifies graft rejection through induction of tolerogenic CD4+CD25+ cells in the recipientSong Guo Zheng
Division of Rheumatology and Immunology, Department of Medicine, 2011 Zonal Avenue, HMR 711
Int Immunol 18:279-89. 2006..Thus, Tregs generated ex vivo can act like a vaccine that generates host suppressor cells with the potential to protect MHC-mismatched organ grafts from rejection...
Transforming growth factor-beta: taking control of T cells' life and deathDavid A Horwitz
Division of Rheumatology and Immunology, Keck School of Medicine, University of Southern California, Los Angeles, California 90033, USA
Immunity 25:399-401. 2006..2006) and Li et al. (2006a) created mice with T cells lacking TGF-beta signaling receptors. Both report that TGF-beta signaling by T cells is absolutely essential for tolerance and homeostasis...
Role of SMAD and non-SMAD signals in the development of Th17 and regulatory T cellsLing Lu
Division of Rheumatology, Department of Medicine, Saban Research Institute, Children s Hospital Los Angeles, Keck School of Medicine, University of Southern California, Los Angeles, CA 90033, USA
J Immunol 184:4295-306. 2010..Therefore, selective targeting of these intracellular TGF-beta signaling pathways during iTreg and Th17 cell development might lead to the development of therapies in treating autoimmune and other chronic inflammatory diseases...
Synergistic effect of TGF-beta superfamily members on the induction of Foxp3+ TregLing Lu
Division of Rheumatology, Department of Medicine, Keck School of Medicine, University of Southern California, and Developmental Biology Program, Department of Surgery, Children s Hospital Los Angeles, Los Angeles, CA 90033, USA
Eur J Immunol 40:142-52. 2010..In addition, the synergistic effect of BMP-2/4 and TGF-beta indicates that the simultaneous manipulation of TGF-beta and BMP signaling might have considerable effects in the clinical setting for the enhancement of Treg purity and yield...
Increase of CD4+ CD25+ regulatory T-cells in the liver of patients with hepatocellular carcinomaXiu Hua Yang
Division of Gastroenterology and Hepatology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan
J Hepatol 45:254-62. 2006..CONCLUSIONS: Our results suggest that CD4+ CD25+ T-cells in the marginal region of HCC may play a critical role in controlling CD8+ cytotoxic T-cell activity and, thereby, contribute to the progression of HCC...
Research Grants
- IMMUNOREGULATORY MECHANISMS IN THE RHEUMATIC DISEASESDavid Horwitz; Fiscal Year: 1999....
- FLOW CYTOMETERDavid Horwitz; Fiscal Year: 2002..This new instrument will allow us the flexibility to develop new uses for the flow cytometer as dictated by our current and future users. ..
