Caitlin E Hill

Summary

Affiliation: University of Miami
Country: USA

Publications

  1. ncbi Acute transplantation of glial-restricted precursor cells into spinal cord contusion injuries: survival, differentiation, and effects on lesion environment and axonal regeneration
    Caitlin E Hill
    STAR Laboratories, The Laboratory for Neural Repair, Department of Neuroscience, The Ohio State University, Columbus, OH 43210, USA
    Exp Neurol 190:289-310. 2004
  2. ncbi Early necrosis and apoptosis of Schwann cells transplanted into the injured rat spinal cord
    Caitlin E Hill
    The Miami Project to Cure Paralysis, University of Miami Miller School of Medicine, Miami, FL 33136, USA
    Eur J Neurosci 26:1433-45. 2007
  3. ncbi A calpain inhibitor enhances the survival of Schwann cells in vitro and after transplantation into the injured spinal cord
    Caitlin E Hill
    The Miami Project to Cure Paralysis, University of Miami Miller School of Medicine, Miami, Florida, USA
    J Neurotrauma 27:1685-95. 2010
  4. ncbi Labeled Schwann cell transplantation: cell loss, host Schwann cell replacement, and strategies to enhance survival
    Caitlin E Hill
    Miami Project to Cure Paralysis, Department of Neurological Surgery, University of Miami Miller School of Medicine, Miami, Florida 33136, USA
    Glia 53:338-43. 2006
  5. ncbi Skin incision induces expression of axonal regeneration-related genes in adult rat spinal sensory neurons
    Caitlin E Hill
    University of Miami, The Miami Project to Cure Paralysis, Miami, Florida, USA
    J Pain 11:1066-73. 2010

Detail Information

Publications5

  1. ncbi Acute transplantation of glial-restricted precursor cells into spinal cord contusion injuries: survival, differentiation, and effects on lesion environment and axonal regeneration
    Caitlin E Hill
    STAR Laboratories, The Laboratory for Neural Repair, Department of Neuroscience, The Ohio State University, Columbus, OH 43210, USA
    Exp Neurol 190:289-310. 2004
    ..However, GRP cell transplants did alter the morphology of CST axons toward that of growth cones, and CST fibers were found within GRP cell transplants, suggesting that GRP cells may be able to support axonal growth in vivo after injury...
  2. ncbi Early necrosis and apoptosis of Schwann cells transplanted into the injured rat spinal cord
    Caitlin E Hill
    The Miami Project to Cure Paralysis, University of Miami Miller School of Medicine, Miami, FL 33136, USA
    Eur J Neurosci 26:1433-45. 2007
    ..Intervention by strategies that limit necrosis and/or apoptosis should be considered for enhancing acute survival of transplanted cells...
  3. ncbi A calpain inhibitor enhances the survival of Schwann cells in vitro and after transplantation into the injured spinal cord
    Caitlin E Hill
    The Miami Project to Cure Paralysis, University of Miami Miller School of Medicine, Miami, Florida, USA
    J Neurotrauma 27:1685-95. 2010
    ..They also suggest that in vitro assays for cell survival may be useful for establishing new compounds that can then be tested in vivo for their ability to promote transplanted SC survival...
  4. ncbi Labeled Schwann cell transplantation: cell loss, host Schwann cell replacement, and strategies to enhance survival
    Caitlin E Hill
    Miami Project to Cure Paralysis, Department of Neurological Surgery, University of Miami Miller School of Medicine, Miami, Florida 33136, USA
    Glia 53:338-43. 2006
    ..In all cases, transplantation of SCs resulted in extensive infiltration of endogenous p75+ cells into the injury site, suggesting that endogenous SCs may play an important role in the repair observed after SC transplantation...
  5. ncbi Skin incision induces expression of axonal regeneration-related genes in adult rat spinal sensory neurons
    Caitlin E Hill
    University of Miami, The Miami Project to Cure Paralysis, Miami, Florida, USA
    J Pain 11:1066-73. 2010
    ..Axonal regeneration-associated processes should be considered alongside nerve signal conduction and inflammatory/sensitization processes as possible mechanisms contributing to pain, particularly the transition from acute to chronic pain...