Research Topics
Species | Vidya GopalakrishnanSummaryAffiliation: The University of Texas Country: USA Publications
| Collaborators
|
Detail Information
Publications
Myoblast-derived neuronal cells form glutamatergic neurons in the mouse cerebellumVidya Gopalakrishnan
Department of Pediatrics, The University of Texas M D Anderson Cancer Center, Houston, Texas 77030, USA
Stem Cells 28:1839-47. 2010..Our findings suggest that although activation of REST-target genes can reprogram myoblasts to assume a general neuronal phenotype, the subtype specificity may then be directed by the brain microenvironment...
Histone acetylation resulting in resistance to methotrexate in choroid plexus cellsPreethi Prasad
Department of Pediatrics, The University of Texas M D Anderson Cancer Center, Houston, TX 77030, USA
J Neurooncol 91:279-86. 2009..We also confirmed this finding in human choroid plexus carcinoma cells. Methotrexate should not be combined with HDACI in the treatment of choroid plexus carcinoma...
Reactivation of death receptor 4 (DR4) expression sensitizes medulloblastoma cell lines to TRAILDolly G Aguilera
Department of Pediatrics, University of Texas, M D Anderson Cancer Center, Houston, 77030, USA
J Neurooncol 93:303-18. 2009....
Chromatin remodelling at the topoisomerase II-beta promoter is associated with enhanced sensitivity to etoposide in human neuroblastoma cell linesChandra M Das
Division of Pediatrics, Children s Cancer Hospital, The University of Texas M D Anderson Cancer Center, Houston, TX, USA
Eur J Cancer 46:2771-80. 2010..These results suggest a potential epigenetic mechanism of regulation of the topoisomerase II-beta gene and a possible role for its increased expression in the sensitivity of SK-N-AS neuroblastoma cells to etoposide...
Valproic acid induces p21 and topoisomerase-II (alpha/beta) expression and synergistically enhances etoposide cytotoxicity in human glioblastoma cell linesChandra M Das
Department of Pediatrics, Unit 853, University of Texas, M D Anderson Cancer Center, 1515 Holcombe Blvd, Houston, TX 77030, USA
J Neurooncol 85:159-70. 2007..In this study, we have examined the ability of the histone deacetylase inhibitor, valproic acid (VPA) to modulate gene expression and sensitize glioblastoma cell lines to the cytotoxic effects of etoposide in vitro...
Conversion of myoblasts to physiologically active neuronal phenotypeYumi Watanabe
Department of Molecular Genetics, The University of Texas M.D. Anderson Cancer Center, Houston, TX 77030, USA
Genes Dev 18:889-900. 2004....
Abnormal expression of REST/NRSF and Myc in neural stem/progenitor cells causes cerebellar tumors by blocking neuronal differentiationXiaohua Su
Department of Molecular Genetics, University of Texas M D Anderson Cancer Center, 1515 Holcombe Boulevard, Mail 1006, Room S13 8136C, Houston, TX 77030, USA
Mol Cell Biol 26:1666-78. 2006..Furthermore, these results suggest that such a mechanism plays a role in the formation of human medulloblastoma...
Influence of valproic acid on outcome of high-grade gliomas in childrenAmirhadi Masoudi
Division of Pediatrics, The University of Texas M D Anderson Cancer Center, 1515 Holcombe Blvd, Houston, TX 77030, USA
Anticancer Res 28:2437-42. 2008..As the drug has been given frequently as an antiepileptic drug, a retrospective analysis was conducted to ensure relevant information was not missed before a prospective study was launched...
Retinoic acid induces REST degradation and neuronal differentiation by modulating the expression of SCF(?-TRCP) in neuroblastoma cellsAkanksha Singh
Department of Pediatrics, The University of Texas MD Anderson Cancer Center, Houston, Texas
Cancer 117:5189-202. 2011..Because retinoic acid promotes neuronal differentiation, the authors postulated that it involves modulation of REST expression...
Measuring cytotoxicity: a new perspective on LC50Ming Zhang
Department of Bioinformatics and Computational Biology, The University of Texas M D Anderson Cancer Center, Houston, TX 77030, USA
Anticancer Res 27:35-8. 2007..A dose-response relationship was observed between the parameter k2 in this formula and the concentration of the cytotoxic agent etoposide, in high-grade glioma cells was independent of incubation time...
