Research Topics
Species | J M FordSummaryAffiliation: Stanford University Country: USA Publications
| Collaborators
|
Detail Information
Publications
Failures of automatic and strategic processing in schizophrenia: comparisons of event-related brain potential and startle blink modificationJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, CA 94305 5550, USA
Schizophr Res 37:149-63. 1999..These results suggest that different neural circuits are involved in blink and cortical reflections of startle modification in schizophrenics and controls, with both automatic and strategic processes being impaired in schizophrenia...- Schizophrenia: the broken P300 and beyondJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, CA 94305 5548, USA
Psychophysiology 36:667-82. 1999..P300 amplitude reduction is related to enduring negative symptoms, waning of attention, and gray matter volume deficits. ERP components other than P300 can also manifest the biological reality of various symptoms of the disease... - Cortical responsiveness during inner speech in schizophrenia: an event-related potential studyJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, CA 94305 5550, USA
Am J Psychiatry 158:1914-6. 2001..The study assessed the effects of inner speech on auditory cortical responsiveness in schizophrenia... - Neurophysiological evidence of corollary discharge dysfunction in schizophreniaJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, CA 943054 5550, USA
Am J Psychiatry 158:2069-71. 2001..Event-related potentials were used to test whether failures of corollary discharge during speech contribute to the pathophysiology of schizophrenia... - Left temporal deficit of P300 in patients with schizophrenia: effects of taskJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, CA 94305 5550, USA
Int J Psychophysiol 38:71-9. 2000..Failure to find P300 asymmetry is not related to the presence or absence of a button pressing task, or the hand used for button pressing. Rather, P300 asymmetry may be related to structural neuroanatomical asymmetries... - N1 and P300 abnormalities in patients with schizophrenia, epilepsy, and epilepsy with schizophrenialike featuresJ M Ford
Department of Psychiatry and Behavioral Science, Stanford University School of Medicine, Stanford, California 94304, USA
Biol Psychiatry 49:848-60. 2001..N1 amplitude reduction appears to be specific to schizophrenia, suggesting its sensitivity to the distinct etiology of schizophrenia... - Event-related brain potential evidence of spared knowledge in Alzheimer's diseaseJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine and Palo Alto Veterans Affairs Health Care System, California 94305-5550, USA
Psychol Aging 16:161-76. 2001..Thus, although the name of an item may be inaccessible in confrontation naming, N400 shows that knowledge is intact enough to prime cortical responses... - Cortical responsiveness during talking and listening in schizophrenia: an event-related brain potential studyJ M Ford
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, California 94305-5550, USA
Biol Psychiatry 50:540-9. 2001..Listening reduced N1 equivalently in both groups. CONCLUSIONS: Although the failure of N1 to be reduced during talking was not related to current hallucinations in patients, it may be related to the potential to hallucinate... - Proapoptotic p53-interacting protein 53BP2 is induced by UV irradiation but suppressed by p53C D Lopez
Divisions of Medical Oncology, Department of Pediatrics, Stanford University School of Medicine, Stanford, California 94305, USA
Mol Cell Biol 20:8018-25. 2000..Our data provide new insight into 53BP2 function and open new avenues for investigation into the cellular response to genotoxic stress... - Decreased UV sensitivity, mismatch repair activity and abnormal cell cycle checkpoints in skin cancer cell lines derived from UVB-irradiated XPA-deficient miceM Ichikawa
Division of Cellular Genetics, Osaka University, Suita, Japan
Mutat Res 459:285-98. 2000.... - The p53-regulated cyclin-dependent kinase inhibitor, p21 (cip1, waf1, sdi1), is not required for global genomic and transcription-coupled nucleotide excision repair of UV-induced DNA photoproductsS Adimoolam
Department of Medicine, Division of Oncology, Stanford University School of Medicine, Stanford, California 94305, USA
J Biol Chem 276:25813-22. 2001..Therefore, our results indicate that p53-dependent nucleotide excision repair does not require the function of the p21 gene product and is independent of p53-regulated cell cycle checkpoints... - p53-mediated DNA repair responses to UV radiation: studies of mouse cells lacking p53, p21, and/or gadd45 genesM L Smith
Division of Basic Science, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Mol Cell Biol 20:3705-14. 2000..We provide evidence that Gadd45 affects chromatin remodelling of templates concurrent with DNA repair, thus indicating that Gadd45 may participate in the coupling between chromatin assembly and DNA repair...