Research Topics
| Ronald DalmanSummaryAffiliation: Stanford University Country: USA Publications
Research Grants
| Collaborators
|
Detail Information
Publications
Transition to all-autogenous hemodialysis access: the role of preoperative vein mappingRonald L Dalman
Department of Surgery, Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA, USA
Ann Vasc Surg 16:624-30. 2002..The functional superiority of ADA vs. prosthetic dialysis access (PDA) in this series may be due to optimal autogenous conduit selection facilitated by preoperative vein mapping...- Oxidative stress and abdominal aneurysms: how aortic hemodynamic conditions may influence AAA diseaseRonald L Dalman
Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
Cardiovasc Surg 11:417-9. 2003 - AAA disease: mechanism, stratification, and treatmentRonald L Dalman
Department of Surgery, Stanford University Medical Center, Stanford, CA 94305 5642, USA
Ann N Y Acad Sci 1085:92-109. 2006.... - Treatment of abdominal aortic anastomotic pseudoaneurysm with percutaneous coil embolizationJames I Fann
Department of Cardiothoracic Surgery, Division of Vascular Surgery Stanford University Medical Center, VA Palo Alto HCS, 94305, USA
J Vasc Surg 35:811-4. 2002..The patient remains without recurrence of pseudoaneurysm 3.5 years later... - In vivo quantification of murine aortic cyclic strain, motion, and curvature: implications for abdominal aortic aneurysm growthCraig J Goergen
Department of Bioengineering, Stanford University, Stanford, California 94305 5431, USA
J Magn Reson Imaging 32:847-58. 2010..To develop methods to quantify cyclic strain, motion, and curvature of the murine abdominal aorta in vivo... - The utility of endovascular simulation to improve technical performance and stimulate continued interest of preclinical medical students in vascular surgeryJason T Lee
Division of Vascular Surgery, Stanford University School of Medicine, Stanford, California 94305, USA
J Surg Educ 66:367-73. 2009..The purpose of this study is to assess the ability of a simulation-based curriculum to improve the technical performance and interest level of medical students in vascular surgery... - A survey of demographics, motivations, and backgrounds among applicants to the integrated 0 + 5 vascular surgery residencyJason T Lee
Vascular Surgery Residency Fellowship, Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
J Vasc Surg 51:496-502; discussion 502-3. 2010..We compared demographics and surveyed recent applicants to our integrated program to gain more insight into their background and motivation for accelerated vascular training... - Simulation-based endovascular skills assessment: the future of credentialing?Maureen M Tedesco
Division of Vascular Surgery and the Department of Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
J Vasc Surg 47:1008-1; discussion 1014. 2008..The purpose of this study was to determine whether structured global performance assessment during endovascular simulation correlated well with trainee-reported procedural skill and prior experience level... - Influences of aortic motion and curvature on vessel expansion in murine experimental aneurysmsCraig J Goergen
Department of Bioengineering, Stanford University, 318 Campus Dr, Stanford, CA 94305 5431, USA
Arterioscler Thromb Vasc Biol 31:270-9. 2011..To quantitatively compare aortic curvature and motion with resulting aneurysm location, direction of expansion, and pathophysiological features in experimental abdominal aortic aneurysms (AAAs)... - Open versus endovascular AAA repair in patients who are morphological candidates for endovascular treatmentBradley B Hill
Division of Vascular Surgery, Stanford University Medical Center, 300 Pasteur Drive H 3638, Stanford, CA 94305 5642, USA
J Endovasc Ther 9:255-61. 2002..To compare the outcomes of open versus endovascular repair of abdominal aortic aneurysm (AAA) in a cohort of patients who fulfill morphological criteria for endovascular repair... - Preoperative thrombus volume predicts sac regression after endovascular aneurysm repairJanice J Yeung
Division of Vascular Surgery, Stanford University Medical Center, Stanford, California 94305, USA
J Endovasc Ther 16:380-8. 2009..To examine whether preoperative aneurysm thrombus volume correlated with abdominal aortic aneurysm (AAA) sac regression following endovascular aneurysm repair (EVAR)... - Aortoiliac hemodynamic and morphologic adaptation to chronic spinal cord injuryJanice J Yeung
Department of Surgery, University of Rochester, Rochester, NY, USA
J Vasc Surg 44:1254-1265. 2006..Slower aortic pressure decay and reduced wall shear stress after SCI may contribute to mural degeneration, enlargement, and an increased prevalence of AAA disease... - Aneurysm-related death: primary endpoint analysis for comparison of open and endovascular repairFrank R Arko
Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
J Vasc Surg 36:297-304. 2002..Secondary procedures are required after both open and endovascular repair. However, the magnitude, morbidity, and mortality of secondary procedures are reduced significantly with endovascular repair... - Hemodynamic regulation of CD34+ cell localization and differentiation in experimental aneurysmsEiketsu Sho
Division of Vascular Surgery, Stanford University, and Veterans Affairs Palo Alto Health Care System, Palo Alto, Calif 94304, USA
Arterioscler Thromb Vasc Biol 24:1916-21. 2004..Modulating cell lineage differentiation of mature progenitor cells may represent a novel therapeutic strategy to maintain medial cellularity and extracellular matrix integrity in AAA disease... - Apelin prevents aortic aneurysm formation by inhibiting macrophage inflammationNicholas J Leeper
Division of Cardiovascular Medicine, Department of Medicine, Stanford Univ, 300 Pasteur Dr, Stanford, California 94305, USA
Am J Physiol Heart Circ Physiol 296:H1329-35. 2009..The mechanism appears to be decreased macrophage burden, perhaps related to an apelin-mediated decrease in proinflammatory cytokine and chemokine activation... - Quantification of hemodynamics in abdominal aortic aneurysms during rest and exercise using magnetic resonance imaging and computational fluid dynamicsAndrea S Les
Department of Bioengineering, Stanford University, Stanford, CA, USA
Ann Biomed Eng 38:1288-313. 2010..We postulate that the increased MWSS, decreased OSI, and moderate turbulence present during exercise may attenuate AAA growth... - Hemodynamic influences on abdominal aortic aneurysm disease: Application of biomechanics to aneurysm pathophysiologyMonica M Dua
Division of Vascular Surgery, Stanford University School of Medicine, CA 94305, United States
Vascul Pharmacol 53:11-21. 2010..Improved understanding of aortic hemodynamic risk profiles provides an opportunity to modify patient activity patterns to minimize the risk of aneurysmal degeneration... - Hyperglycemia modulates plasminogen activator inhibitor-1 expression and aortic diameter in experimental aortic aneurysm diseaseMonica M Dua
Division of Vascular Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA
Surgery 148:429-35. 2010..We hypothesize that hyperglycemia limits AAA progression through effects on endogenous plasminogen activator inhibitor-1 (PAI-1) levels and subsequent reductions in plasmin generation... - Hyperglycemia limits experimental aortic aneurysm progressionNoriyuki Miyama
Division of Vascular Surgery, Stanford University School of Medicine, Stanford, Calif, USA
J Vasc Surg 52:975-83. 2010..Mechanisms responsible for this negative association remain unknown. We created AAAs in hyperglycemic mice to examine the influence of serum glucose concentration on experimental aneurysm progression... - Reduction of postprocedure microemboli following retrospective quality assessment and practice improvement measures for carotid angioplasty and stentingMaureen M Tedesco
Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
J Vasc Surg 49:607-12; discussion 612-3. 2009..The purpose of this study is to compare the incidence of microemboli in two distinct time periods when procedural modifications were implemented into a CAS program... - The three-dimensional micro- and nanostructure of the aortic medial lamellar unit measured using 3D confocal and electron microscopy imagingMary K O'Connell
Department of Mechanical Engineering, Stanford University, Stanford, CA, USA
Matrix Biol 27:171-81. 2008..This high resolution three-dimensional view of the aortic media reveals MLU microstructure details that suggest a highly complex and integrated mural organization that correlates with aortic mechanical properties... - Wall shear stress and strain modulate experimental aneurysm cellularityKatsuyuki Hoshina
Division of Vascular Surgery, Stanford University, Palo Alto, Calif, USA
J Vasc Surg 37:1067-74. 2003..Increased lower extremity activity may prevent or retard AAA disease through salutary effects on aortic remodeling mediated by endothelial cells and smooth muscle cells... - Continuous periaortic infusion improves doxycycline efficacy in experimental aortic aneurysmsEiketsu Sho
Division of Vascular Surgery, Stanford University, Palo Alto, CA 94304, USA
J Vasc Surg 39:1312-21. 2004..Given the recognized limitations of current technologies, adjuvant biologic therapies have the potential to improve long-term patient outcome significantly after endovascular exclusion... - Risk factors for developing postprocedural microemboli following carotid interventionsMaureen M Tedesco
Division of Vascular Surgery, Stanford University Medical Center, CA 94305, USA
J Endovasc Ther 14:561-7. 2007..To determine risk factors predictive of microemboli found on diffusion-weighted magnetic resonance imaging (DW-MRI) following carotid angioplasty and stenting (CAS) with distal protection and carotid endarterectomy (CEA)... - Comparison of cell-type-specific vs transmural aortic gene expression in experimental aneurysmsEiketsu Sho
Division of Vascular Surgery, Stanford University, USA
J Vasc Surg 41:844-52. 2005..We used laser capture microdissection (LCM) to isolate SMC and macrophage-predominant mural cell populations for gene expression analysis in variable-flow AAA... - Enhanced abdominal aortic aneurysm formation in thrombin-activatable procarboxypeptidase B-deficient miceGeoffrey Schultz
Department of Vascular Surgery, Stanford University School of Medicine, Veterans Affairs Palo Alto Health Care System, 3801 Miranda Ave, Palo Alto, CA 94304, USA
Arterioscler Thromb Vasc Biol 30:1363-70. 2010..To determine whether procarboxypeptidase B (pCPB)(-/-) mice are susceptible to accelerated abdominal aortic aneurysm (AAA) development secondary to unregulated OPN-mediated mural inflammation in the absence of CPB inhibition... - Postprocedural microembolic events following carotid surgery and carotid angioplasty and stentingMaureen M Tedesco
Division of Vascular Surgery, Stanford University Medical Center, Stanford, CA 94305, USA
J Vasc Surg 46:244-50. 2007..We compared the incidence and distribution of cerebral microembolic events after carotid angioplasty and stenting (CAS) with distal protection to standard open carotid endarterectomy (CEA) using DW-MRI... - Quantifying in vivo hemodynamic response to exercise in patients with intermittent claudication and abdominal aortic aneurysms using cine phase-contrast MRIAdam S Tenforde
Department of Bioengineering, Stanford University, Stanford, CA, USA
J Magn Reson Imaging 31:425-9. 2010..To evaluate rest and exercise hemodynamics in patients with abdominal aortic aneurysms (AAA) and peripheral occlusive disease (claudicants) using phase-contrast MRI... - Hemodynamic forces regulate mural macrophage infiltration in experimental aortic aneurysmsEiketsu Sho
Division of Vascular Surgery, Stanford University, Palo Alto, CA 94304, USA
Exp Mol Pathol 76:108-16. 2004..Hemodynamic forces may modulate AAA inflammation and diameter enlargement via direct regulation of intimal macrophage adhesion, transmural migration or survival... - Analysis of in situ and ex vivo vascular endothelial growth factor receptor expression during experimental aortic aneurysm progressionMaureen M Tedesco
Division of Vascular Surgery, Stanford University Medical Center, 300 Pasteur Drive, Stanford, CA 94305 5642, USA
Arterioscler Thromb Vasc Biol 29:1452-7. 2009..We examined VEGFR expression as a function of AAA disease progression in the Apolipoprotein E-deficient (Apo E(-/-)) murine AAA model... - Flow loading induces macrophage antioxidative gene expression in experimental aneurysmsTakeshi K Nakahashi
Veterans Affairs Palo Alto Health Care System, Palo Alto, USA
Arterioscler Thromb Vasc Biol 22:2017-22. 2002..Flow loading and alpha-tocopherol therapy reduced AAA reactive oxygen species production. CONCLUSIONS: Flow loading may attenuate AAA enlargement via wall shear or strain-related reductions in oxidative stress... - Vitamin E limits AAARonald L Dalman
Arterioscler Thromb Vasc Biol 26:e21. 2006 - Aortic wall cell proliferation via basic fibroblast growth factor gene transfer limits progression of experimental abdominal aortic aneurysmKatsuyuki Hoshina
Division of Vascular Regeneration, Graduate School of Medicine, The University of Tokyo, Japan
J Vasc Surg 40:512-8. 2004..These findings suggest that increased medial cellularity inhibits aneurysm formation, which possibly offers a clue for developing a new strategy for treatment of AAAs... - Medical management of small abdominal aortic aneurysmsB Timothy Baxter
University of Nebraska Medical Center, Omaha, NE, USA
Circulation 117:1883-9. 2008..Additional studies are needed to clarify the potential role of doxycycline, roxithromycin, and statin therapy in the progression of aneurysmal disease...
Research Grants
- Mechano-biologic Determinants of AAA DiseaseRonald Dalman; Fiscal Year: 2007..Circulating progenitor cell localization and differentiation will be examined via dual staining strategies in chimeric mice. ..
- AAA Disease: Mechanism, Stratification and TreatmentRonald Dalman; Fiscal Year: 2007....