Research Topics
Genomes and Genes | J N WeinsteinSummaryAffiliation: National Cancer Institute Country: USA Publications
| Collaborators
|
Detail Information
Publications
Comparing cDNA and oligonucleotide array data: concordance of gene expression across platforms for the NCI-60 cancer cellsJae K Lee
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892 8322, USA
Genome Biol 4:R82. 2003..Global concordance is parameterized by a 'correlation of correlations' coefficient...
GoMiner: a resource for biological interpretation of genomic and proteomic dataBarry R Zeeberg
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Genome Biol 4:R28. 2003..The first is a tree-like structure analogous to that in the AmiGO browser and the second is a compact, dynamically interactive 'directed acyclic graph'. Genes displayed in GoMiner are linked to major public bioinformatics resources...
The LeFE algorithm: embracing the complexity of gene expression in the interpretation of microarray dataGabriel S Eichler
Genomics and Bioinformatics Groups, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Genome Biol 8:R187. 2007..We also compare it with previously published algorithms, including Gene Set Enrichment Analysis. LeFE regularly identifies statistically significant functional themes consistent with known biology...
MatchMiner: a tool for batch navigation among gene and gene product identifiersKimberly J Bussey
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, NIH Building 37, Bethesda, MD 20892-4255, USA
Genome Biol 4:R27. 2003..The user inputs a list of gene identifiers and then uses the Merge function to find the overlap with a second list of identifiers of either the same or a different type or uses the LookUp function to find corresponding identifiers...
High-Throughput GoMiner, an 'industrial-strength' integrative gene ontology tool for interpretation of multiple-microarray experiments, with application to studies of Common Variable Immune Deficiency (CVID)Barry R Zeeberg
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
BMC Bioinformatics 6:168. 2005..We wanted to provide a computational resource that automates the analysis of multiple microarrays and then integrates the results across all of them in useful exportable output files and visualizations...
Quality assessment of microarrays: visualization of spatial artifacts and quantitation of regional biasesMark Reimers
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland 20892, USA
BMC Bioinformatics 6:166. 2005..Current practice in quality assessment for microarrays does not address regional biases...
AbMiner: a bioinformatic resource on available monoclonal antibodies and corresponding gene identifiers for genomic, proteomic, and immunologic studiesSylvia M Major
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, USA
BMC Bioinformatics 7:192. 2006....
VennMaster: area-proportional Euler diagrams for functional GO analysis of microarraysHans A Kestler
Neural Information Processing, University of Ulm, Germany
BMC Bioinformatics 9:67. 2008..Additional graphical methods are therefore needed to augment the GO graphical representation...
SpliceMiner: a high-throughput database implementation of the NCBI Evidence Viewer for microarray splice variant analysisAri B Kahn
Department of Bioinformatics, George Mason University, Fairfax, Virginia, USA
BMC Bioinformatics 8:75. 2007..Accurate analysis of microarray expression data and design of new arrays for alternative splicing require assessment of probes at the sequence and exon levels...
Spotlight on molecular profiling: "Integromic" analysis of the NCI-60 cancer cell linesJohn N Weinstein
Laboratory of Molecular Pharmacology, National Cancer Institute, 37 Convent Drive, Room 5056B, Bethesda, MD 20892, USA
Mol Cancer Ther 5:2601-5. 2006
An information-intensive approach to the molecular pharmacology of cancerJ N Weinstein
Laboratory of Molecular Pharmacology LMP, Division of Basic Science, National Cancer Institute NCI, National Institutes of Health, Bethesda, MD 20892, USA
Science 275:343-9. 1997..It remains to be seen how effective this information-intensive strategy will be at generating new clinically active agents...
The bioinformatics of microarray gene expression profilingJohn N Weinstein
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, Bethesda, Maryland, USA
Cytometry 47:46-9. 2002
Searching for pharmacogenomic markers: the synergy between omic and hypothesis-driven researchJ N Weinstein
Laboratory of Molecular Pharmacology, National Cancer Institute, Bethesda, MD, USA
Dis Markers 17:77-88. 2001....
A protein expression database for the molecular pharmacology of cancerT G Myers
Laboratory of Molecular Pharmacology, National Cancer Institute NCI, Bethesda, MD 20852, USA
Electrophoresis 18:647-53. 1997..Links to pertinent databases and tools of analysis will be updated progressively at http:@www.nci.nih.gov/intra/lmp/jnwbio.htm and http:@epnwsl.ncifcrf.gov:2345/dis3d/dtp.++ +html...
Characterization of the p53 tumor suppressor pathway in cell lines of the National Cancer Institute anticancer drug screen and correlations with the growth-inhibitory potency of 123 anticancer agentsP M O'Connor
Division of Basic Sciences, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 57:4285-300. 1997..This information also allows for the large-scale analysis of the more than 60,000 compounds tested against these lines for novel agents that might exploit defective p53 function as a means of preferential toxicity...
Quantitative structure-antitumor activity relationships of camptothecin analogues: cluster analysis and genetic algorithm-based studiesY Fan
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
J Med Chem 44:3254-63. 2001..The cross-validated r(2) for the final GFA model was 0.783, indicating a predictive QSAR model...
Mining and visualizing large anticancer drug discovery databasesL M Shi
Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892 4255, USA
J Chem Inf Comput Sci 40:367-79. 2000..Mining the database can provide useful information: (a) for the development of anticancer drugs; (b) for a better understanding of the molecular pharmacology of cancer; and (c) for improvement of the drug discovery process...
MedMiner: an Internet text-mining tool for biomedical information, with application to gene expression profilingL Tanabe
National Institutes of Health, Bethesda, MD, USA
Biotechniques 27:1210-4, 1216-7. 1999..More generally still, MedMiner can be used to organize the information returned from any arbitrary PubMed search...
Transcript and protein expression profiles of the NCI-60 cancer cell panel: an integromic microarray studyUma T Shankavaram
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute NIH, Bethesda, MD 20892, USA
Mol Cancer Ther 6:820-32. 2007..71). Because the transcript-based technologies are more mature and are currently able to assess larger numbers of genes at one time, they continue to be useful, even when the ultimate aim is information about proteins...
mRNA and microRNA expression profiles of the NCI-60 integrated with drug activitiesHongfang Liu
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA
Mol Cancer Ther 9:1080-91. 2010..The data sets presented here will facilitate the identification of groups of mRNAs, microRNAs, and drugs that potentially affect and interact with one another...
Predicting drug sensitivity and resistance: profiling ABC transporter genes in cancer cellsGergely Szakacs
Laboratory of Cell Biology, Center for Cancer Research, NCI, NIH, Bethesda, MD, 20892, USA
Cancer Cell 6:129-37. 2004..Unexpectedly, we also found and validated compounds whose activity is potentiated, rather than antagonized, by the MDR1 multidrug transporter. Such compounds may serve as leads for development...
Proteomic profiling of the NCI-60 cancer cell lines using new high-density reverse-phase lysate microarraysSatoshi Nishizuka
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Proc Natl Acad Sci U S A 100:14229-34. 2003....
Gadd45, a p53-responsive stress protein, modifies DNA accessibility on damaged chromatinF Carrier
Laboratory of Biological Chemistry, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892 4255, USA
Mol Cell Biol 19:1673-85. 1999..Both histone acetylation and UV radiation are thought to destabilize the nucleosomal structure. Hence, these results imply that Gadd45 can recognize an altered chromatin state and modulate DNA accessibility to cellular proteins...
Comparison of a neural net-based QSAR algorithm (PCANN) with Hologram- and multiple linear regression-based QSAR approaches: application to 1,4-dihydropyridine-based calcium channel antagonistsV N Viswanadhan
Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
J Chem Inf Comput Sci 41:505-11. 2001..The present approach (PCANN) may prove useful for rapid assessment of the potential for biological activity when dealing with large chemical libraries...
Asparagine synthetase as a causal, predictive biomarker for L-asparaginase activity in ovarian cancer cellsPhilip L Lorenzi
Genomics and Bioinformatics Group, Room 5056B, 37 Convent Drive, Bethesda, MD 20892, USA
Mol Cancer Ther 5:2613-23. 2006..Overall, this pharmacogenomic/pharmacoproteomic study suggests the use of l-ASP for treatment of a subset of ovarian cancers (and perhaps other tumor types), with ASNS as a biomarker for patient selection...
Profiling SLCO and SLC22 genes in the NCI-60 cancer cell lines to identify drug uptake transportersMitsunori Okabe
Laboratory of Cell Biology, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, MD 20892, USA
Mol Cancer Ther 7:3081-91. 2008..Our results indicate that the gene expression database can be used to identify SLCO and SLC22 family members that confer sensitivity to cancer cells...
Chromosomal instability is associated with higher expression of genes implicated in epithelial-mesenchymal transition, cancer invasiveness, and metastasis and with lower expression of genes involved in cell cycle checkpoints, DNA repair, and chromatin maiAnna V Roschke
Genetics Branch, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20889 5105, USA
Neoplasia 10:1222-30. 2008..These same karyotypic features are negatively correlated with the expression of genes involved in cell cycle checkpoints, DNA repair, and chromatin maintenance...
Multifactorial regulation of E-cadherin expression: an integrative studyWilliam C Reinhold
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA
Mol Cancer Ther 9:1-16. 2010..Finally, levels of cellular E-cad expression are associated with levels of cell-cell adhesion and response to drug treatment...
Mistaken identifiers: gene name errors can be introduced inadvertently when using Excel in bioinformaticsBarry R Zeeberg
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research CCR, National Cancer Institute NCI, National Institutes of Health NIH, Bethesda, MD 20892 USA
BMC Bioinformatics 5:80. 2004..When processing microarray data sets, we recently noticed that some gene names were being changed inadvertently to non-gene names...
Signal pathway profiling of epithelial and stromal compartments of colonic carcinoma reveals epithelial-mesenchymal transitionK M Sheehan
NCI FDA Clinical Proteomics Program, Laboratory of Pathology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Oncogene 27:323-31. 2008..Given recent findings of epithelial-mesenchymal transition in therapy-resistant tumour epithelium, these findings could have therapeutic implications for colon cancer...
In vitro differential sensitivity of melanomas to phenothiazines is based on the presence of codon 600 BRAF mutationOgechi N Ikediobi
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, Bethesda, Maryland, USA
Mol Cancer Ther 7:1337-46. 2008..The findings reported here have potential implications for the use of phenothiazines in the treatment of V600E BRAF mutant melanoma...
SpliceCenter: a suite of web-based bioinformatic applications for evaluating the impact of alternative splicing on RT-PCR, RNAi, microarray, and peptide-based studiesMichael C Ryan
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA
BMC Bioinformatics 9:313. 2008..In addition, the critical roles of alternative splice forms in biological function and in disease suggest that assay results may be more informative if analyzed in the context of the targeted splice variant...
Detailed DNA methylation profiles of the E-cadherin promoter in the NCI-60 cancer cellsWilliam C Reinhold
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, NIH, Building 37, Room 5056, Bethesda, MD 20892 4255, USA
Mol Cancer Ther 6:391-403. 2007..As has been shown in recent years, DNA methylation status can serve as a biomarker for use in choosing therapy...
Apoptotic susceptibility of cancer cells selected for camptothecin resistance: gene expression profiling, functional analysis, and molecular interaction mappingWilliam C Reinhold
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Cancer Res 63:1000-11. 2003..This model is analogous to one suggested previously for the relationship between oncogene function and apoptosis in carcinogenesis...
Predicting cisplatin and trabectedin drug sensitivity in ovarian and colon cancersEllen V Stevens
National Cancer Institute, Building 37, Room 5068, Bethesda, MD 20892, USA
Mol Cancer Ther 7:10-8. 2008..The work reported here provides motivation for larger proteomic studies with more cell types focused on potential biomarkers in additional pharmacologically pertinent pathways...
Cancers as wounds that do not heal: differences and similarities between renal regeneration/repair and renal cell carcinomaJoseph Riss
Laboratory of Biosystems and Cancer, Comparative Oncology Program, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 66:7216-24. 2006..The observations reported here provide a conceptual framework for further efforts to understand the biology and to develop more effective diagnostic biomarkers and therapeutic strategies for renal tumors and renal ischemia...
Diagnostic markers that distinguish colon and ovarian adenocarcinomas: identification by genomic, proteomic, and tissue array profilingSatoshi Nishizuka
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, NIH, Bethesda, Maryland 20814, USA
Cancer Res 63:5243-50. 2003..The multistep process introduced here has the potential to produce additional markers for cancer diagnosis, prognosis, and therapy...
Selective toxicity of NSC73306 in MDR1-positive cells as a new strategy to circumvent multidrug resistance in cancerJoseph A Ludwig
Laboratory of Cell Biology, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 66:4808-15. 2006..This article shows that NSC73306 kills cells with intrinsic or acquired P-gp-induced MDR and indirectly acts to eliminate resistance to MDR1 substrates...
Exon array analyses across the NCI-60 reveal potential regulation of TOP1 by transcription pausing at guanosine quartets in the first intronWilliam C Reinhold
Laboratory of Molecular Pharmacology and Developmental Therapeutics Program, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20894, USA
Cancer Res 70:2191-203. 2010..The observations reported here suggest the hypothesis that there is a conserved negative transcription regulator within intron 1 of the TOP1 gene associated with a quadruplex-prone region...
Evaluation of current methods used to analyze the expression profiles of ATP-binding cassette transporters yields an improved drug-discovery databaseJosiah N Orina
Laboratory of Cell Biology, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, MD 20892 4256, USA
Mol Cancer Ther 8:2057-66. 2009..This study also led to an improved database by revealing previously unidentified substrates for ABCB1, ABCC1, and ABCG2, transporters that contribute to MDR...
CellMiner: a relational database and query tool for the NCI-60 cancer cell linesUma T Shankavaram
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Centre for Cancer Research, National Cancer Institute, NIH, Bethesda, MD, USA
BMC Genomics 10:277. 2009..S. National Cancer Institute to screen compounds for anticancer activity. To our knowledge, CellMiner is the first online database resource for integration of the diverse molecular types of NCI-60 and related meta data...
Multiplexing siRNAs to compress RNAi-based screen size in human cellsScott E Martin
Gene Silencing Section, Office of Science and Technology Partnership, OD, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Nucleic Acids Res 35:e57. 2007..This approach is likely to be especially applicable where assay costs or platform limitations are prohibitive...
Karyotypic complexity of the NCI-60 drug-screening panelAnna V Roschke
Genetics Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland 20889-5105, USA
Cancer Res 63:8634-47. 2003....
Asparagine synthetase is a predictive biomarker of L-asparaginase activity in ovarian cancer cell linesPhilip L Lorenzi
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland, USA
Mol Cancer Ther 7:3123-8. 2008..These findings provide rationale for evaluation of ASNS protein expression as a predictive biomarker of clinical L-ASP activity in ovarian cancer...
Integrating data on DNA copy number with gene expression levels and drug sensitivities in the NCI-60 cell line panelKimberly J Bussey
Laboratory of Molecular Pharmacology, National Cancer Institute, Building 37, Room 5056, NIH, MSC 4255, 9000 Rockville Pike, Bethesda, MD 20892-4255, USA
Mol Cancer Ther 5:853-67. 2006..The DNA copy number database presented here will enable other investigators to explore DNA transcript-drug relationships in their own domains of research focus...
Transcriptional regulation of mitotic genes by camptothecin-induced DNA damage: microarray analysis of dose- and time-dependent effectsYi Zhou
Laboratory of Molecular Pharmacology, Division of Basic Sciences, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA
Cancer Res 62:1688-95. 2002....
DNA fingerprinting of the NCI-60 cell line panelPhilip L Lorenzi
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Mol Cancer Ther 8:713-24. 2009..As expected, DNA fingerprints were not able to distinguish different tissues-of-origin. The fingerprints serve principally as a barcodes...
A stromal gene signature associated with inflammatory breast cancerBrenda J Boersma
Laboratory of Human Carcinogenesis, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892 4258, USA
Int J Cancer 122:1324-32. 2008..We identified multiple pathways related to the endoplasmic stress response that could be functionally significant in IBC. Our findings suggest that the gene expression in the tumor stroma may play a role in determining the IBC phenotype...
Asparagine synthetase: a new potential biomarker in ovarian cancerPhilip L Lorenzi
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research CCR, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA
Drug News Perspect 22:61-4. 2009....
UPLC-ESI-TOFMS-based metabolomics and gene expression dynamics inspector self-organizing metabolomic maps as tools for understanding the cellular response to ionizing radiationAndrew D Patterson
Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland, USA
Anal Chem 80:665-74. 2008..quot;Radiation metabolomics," the application of metabolomic analysis to the field of radiobiology, promises to increase our understanding of cellular responses to stressors such as radiation...
Nonclassic functions of human topoisomerase I: genome-wide and pharmacologic analysesZe Hong Miao
Laboratories of Molecular Pharmacology, National Institute of Diabetes and Digestive and Kidney Diseases, NIH, Bethesda, Maryland, USA
Cancer Res 67:8752-61. 2007..The reported cell lines and approaches described in this article provide new tools to perform detailed functional analyses related to Top1 function...
Depicting combinatorial complexity with the molecular interaction map notationKurt W Kohn
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, MD, USA
Mol Syst Biol 2:51. 2006..These comparisons may help cell and systems biologists adopt a graphical language that is unambiguous and generally understood...
AffyProbeMiner: a web resource for computing or retrieving accurately redefined Affymetrix probe setsHongfang Liu
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Bioinformatics 23:2385-90. 2007..Otherwise, analysis and interpretation of an Affymetrix microarray experiment will be in error...
Molecular interaction maps--a diagrammatic graphical language for bioregulatory networksMirit I Aladjem
Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, Bethesda, MD 20892, USA
Sci STKE 2004:pe8. 2004....
Transcriptomic analysis of the NCI-60 cancer cell linesJohn N Weinstein
Laboratory of Molecular Pharmacology, Center for Cancer Research, US National Cancer Institute, NIH, Department of Health and Human Services, 9000 Rockville Pike, Bethesda, MD 20892, USA
C R Biol 326:909-20. 2003..We discuss conceptual and experimental aspects of the profiling, as well as a number of bioinformatic computer programs that we have developed for biological interpretation of the profiles...
Analysis of ATP-binding cassette transporter expression in drug-selected cell lines by a microarray dedicated to multidrug resistanceJean-Philippe Annereau
Laboratory of Cell Biology, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892-4256, USA
Mol Pharmacol 66:1397-405. 2004..The custom-designed ABC-Tox microarray presented here will be helpful to elucidate mechanisms leading to anticancer drug resistance...
Karyotypic "state" as a potential determinant for anticancer drug discoveryAnna V Roschke
Genetics Branch and Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA
Proc Natl Acad Sci U S A 102:2964-9. 2005..Thus, we delineate an approach for the identification of "lead compounds" for anticancer drug discovery complementary to those that are focused at the outset on a given gene or pathway...
The p53 tumor suppressor network is a key responder to microenvironmental components of chronic inflammatory stressFrank Staib
Laboratories of Human Carcinogenesis, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892-4255, USA
Cancer Res 65:10255-64. 2005..In summary, the inflammatory stress response is a complex, integrated biological network in which p53 is a key molecular node regulating gene expression...
Biomarkers in cancer staging, prognosis and treatment selectionJoseph A Ludwig
Genomics and Bioinformatics Group, Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892, USA
Nat Rev Cancer 5:845-56. 2005..Understanding how and when biomarkers can be integrated into clinical care is crucial if we want to translate the promise into reality...
Workshop on cancer biometrics: identifying biomarkers and surrogates of cancer in patients: a meeting held at the Masur Auditorium, National Institutes of HealthMichael T Lotze
Translational Research, University of Pittsburgh Molecular Medicine Institute, Pittsburgh, Pennsylvania, USA
J Immunother (1997) 28:79-119. 2005..Concrete recommendations for current application and enabling further development in cancer biometrics are summarized. This will allow a more informed, rapid, and accurate assessment of novel cancer therapies...
Integrating global gene expression and radiation survival parameters across the 60 cell lines of the National Cancer Institute Anticancer Drug ScreenSally A Amundson
Center for Radiological Research, Columbia University Medical Center, New York, New York 10032, USA
Cancer Res 68:415-24. 2008..The response of those genes to gamma-rays seems to be unaffected by the myriad of genetic differences across this diverse cell set; it represents the most penetrant gene expression response to ionizing radiation yet observed...
Nova regulates brain-specific splicing to shape the synapseJernej Ule
Howard Hughes Medical Institute and Laboratory of Molecular Neuro Oncology, The Rockefeller University, New York, New York, USA
Nat Genet 37:844-52. 2005..Validating a large set of Nova RNA targets has led us to identify a multi-tiered network in which Nova regulates the exon content of RNAs encoding proteins that interact in the synapse...
Comparison of methods for sequential screening of large compound setsPaul E Blower
Leadscope, Inc, 1393 Dublin Road, Columbus, OH 43215, USA
Comb Chem High Throughput Screen 9:115-22. 2006....
Membrane transporters and channels: role of the transportome in cancer chemosensitivity and chemoresistanceYing Huang
Program of Pharmacogenomics, Department of Pharmacology, College of Medicine and Public Health, The Ohio State University, Columbus, 43210, USA
Cancer Res 64:4294-301. 2004..Measurement of transporter gene expression may prove useful in predicting anticancer drug response...
A strategy for predicting the chemosensitivity of human cancers and its application to drug discoveryJae K Lee
Department of Public Health Sciences, University of Virginia, Charlottesville, VA 22908, USA
Proc Natl Acad Sci U S A 104:13086-91. 2007..Furthermore, we used COXEN for in silico screening of 45,545 compounds and identify an agent with activity against human bladder cancer...
Impact of p53 knockout and topotecan treatment on gene expression profiles in human colon carcinoma cells: a pharmacogenomic studySayed S Daoud
Department of Pharmaceutical Sciences, Washington State University, Pullman, Washington 99164, USA
Cancer Res 63:2782-93. 2003..The new experimental design and gene expression map analysis introduced here are applicable to a wide range of studies that encompass both treatment effects and genotypic or phenotypic differences...
Mutation analysis of 24 known cancer genes in the NCI-60 cell line setOgechi N Ikediobi
Cancer Genome Project, Wellcome Trust Sanger Institute, Hinxton, Cambridge CB10 1SA, United Kingdom
Mol Cancer Ther 5:2606-12. 2006....
Connecting genes, drugs and diseasesJohn N Weinstein
Nat Biotechnol 24:1365-6. 2006
Sequencing and analysis of 10,967 full-length cDNA clones from Xenopus laevis and Xenopus tropicalis reveals post-tetraploidization transcriptome remodelingRyan D Morin
British Columbia Genome Sciences Centre, BCCA, Vancouver, BC V5Z 1L3 Canada
Genome Res 16:796-803. 2006..Approximately 14% of the paralogous pairs analyzed here also show differential expression indicative of subfunctionalization...
MicroRNAs modulate the chemosensitivity of tumor cellsPaul E Blower
Program of Pharmacogenomics, Department of Pharmacology, Ohio State University, 333 West Tenth Street, Columbus, OH 43210, USA
Mol Cancer Ther 7:1-9. 2008..Ten of those microRNAs have already been implicated in cancer biology. Our results support a substantial role for microRNAs in anticancer drug response, suggesting novel potential approaches to the improvement of chemotherapy...
MicroRNA expression profiles for the NCI-60 cancer cell panelPaul E Blower
Program of Pharmacogenomics, Department of Pharmacology and the Comprehensive Cancer Center, College of Medicine, The Ohio State University, 5072 Graves Hall, 333 West 10th Avenue, Columbus, OH 43210, USA
Mol Cancer Ther 6:1483-91. 2007..Combined with gene expression and other biological data using multivariate analysis, microRNA expression profiles may provide a critical link for understanding mechanisms involved in chemosensitivity and chemoresistance...
Biochemistry. A postgenomic visual iconJohn N Weinstein
Department of Bioinformatics and Computational Biology, M. D. Anderson Cancer Center, 1515 Holcombe Boulevard, Houston, TX 77030, USA
Science 319:1772-3. 2008
