Sharon A Ross

Summary

Affiliation: National Institutes of Health
Country: USA

Publications

  1. ncbi Allyl sulfur compounds from garlic modulate aberrant crypt formation
    Sharon A Ross
    Nutritional Sciences Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, U S Department of Health and Human Services, Rockville, MD 20892 7328, USA
    J Nutr 136:852S-854S. 2006
  2. ncbi Epigenetic modulation and cancer: effect of metabolic syndrome?
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892 7328, USA
    Am J Clin Nutr 86:s872-7. 2007
  3. ncbi MicroRNA, Nutrition, and Cancer Prevention
    Sharon A Ross
    Nutritional Science Research Group, National Cancer Institute, Bethesda, MD
    Adv Nutr 2:472-85. 2011
  4. ncbi Nutritional genomic approaches to cancer prevention research
    S A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA
    Exp Oncol 29:250-6. 2007
  5. ncbi Evidence for the relationship between diet and cancer
    S A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, EPN 3157, National Institutes of Health, Department of Health and Human Services, 6130 Executive Blvd, MSC 7328 Bethesda, MD 20892 7328, USA
    Exp Oncol 32:137-42. 2010
  6. ncbi Dietary components impact histone modifications and cancer risk
    Cindy D Davis
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, 6130 Executive Boulevard, Suite 3159, Rockville, MD 20892 7328, USA
    Nutr Rev 65:88-94. 2007
  7. ncbi Evidence for dietary regulation of microRNA expression in cancer cells
    Cindy D Davis
    Nutritional Science Research Group, National Cancer Institute, 6130 Executive Blvd, Suite 3160, Rockville, MD 20892 7328, USA
    Nutr Rev 66:477-82. 2008
  8. ncbi New technologies for nutrition research
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892, USA
    J Nutr 134:681-5. 2004
  9. ncbi Diet and DNA methylation interactions in cancer prevention
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, Bethesda, Maryland 20892, USA
    Ann N Y Acad Sci 983:197-207. 2003
  10. ncbi Nutritional links to plausible mechanisms underlying pancreatic cancer: a conference report
    R Jean Hine
    Nutritional Sciences Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892-7328, USA
    Pancreas 27:356-66. 2003

Collaborators

Detail Information

Publications20

  1. ncbi Allyl sulfur compounds from garlic modulate aberrant crypt formation
    Sharon A Ross
    Nutritional Sciences Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, U S Department of Health and Human Services, Rockville, MD 20892 7328, USA
    J Nutr 136:852S-854S. 2006
    ..The response to these allyl sulfur compounds appears to depend on several factors, including the speciation, quantity, and duration provided...
  2. ncbi Epigenetic modulation and cancer: effect of metabolic syndrome?
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892 7328, USA
    Am J Clin Nutr 86:s872-7. 2007
    ..Thus, epigenetics may bridge both the thrifty genotype and thrifty phenotype hypotheses and provide a link between genes and the environment concerning disease predisposition to metabolic syndrome and its associated diseases...
  3. ncbi MicroRNA, Nutrition, and Cancer Prevention
    Sharon A Ross
    Nutritional Science Research Group, National Cancer Institute, Bethesda, MD
    Adv Nutr 2:472-85. 2011
    ..Understanding the affect of dietary factors on miRNA expression and function may provide insight on prevention strategies to reduce the burden of cancer...
  4. ncbi Nutritional genomic approaches to cancer prevention research
    S A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA
    Exp Oncol 29:250-6. 2007
    ..Recognition of the interplay between genes and diet could ultimately help identify modifiable molecular targets for preventing, delaying, or reducing the symptoms of cancer and other chronic diseases...
  5. ncbi Evidence for the relationship between diet and cancer
    S A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, EPN 3157, National Institutes of Health, Department of Health and Human Services, 6130 Executive Blvd, MSC 7328 Bethesda, MD 20892 7328, USA
    Exp Oncol 32:137-42. 2010
    ....
  6. ncbi Dietary components impact histone modifications and cancer risk
    Cindy D Davis
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, 6130 Executive Boulevard, Suite 3159, Rockville, MD 20892 7328, USA
    Nutr Rev 65:88-94. 2007
    ..This review focuses on several histone-modifying enzymes that are associated with cancer development and their modification by bioactive food components...
  7. ncbi Evidence for dietary regulation of microRNA expression in cancer cells
    Cindy D Davis
    Nutritional Science Research Group, National Cancer Institute, 6130 Executive Blvd, Suite 3160, Rockville, MD 20892 7328, USA
    Nutr Rev 66:477-82. 2008
    ..miRNAs may be useful as biomarkers of cancer prevention or nutritional status, as well as serve as potential molecular targets that are influenced by dietary interventions...
  8. ncbi New technologies for nutrition research
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD 20892, USA
    J Nutr 134:681-5. 2004
    ..Highlights from this symposium are presented briefly herein...
  9. ncbi Diet and DNA methylation interactions in cancer prevention
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, Bethesda, Maryland 20892, USA
    Ann N Y Acad Sci 983:197-207. 2003
    ..A third plausible mechanism may relate to DNA demethylation activity. Finally, the DNA methylation patterns may influence the response to a bioactive food component...
  10. ncbi Nutritional links to plausible mechanisms underlying pancreatic cancer: a conference report
    R Jean Hine
    Nutritional Sciences Research Group, Division of Cancer Prevention, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, Maryland 20892-7328, USA
    Pancreas 27:356-66. 2003
    ..The recommendations section suggests gaps in current knowledge and articulates future directions for this area of investigation...
  11. ncbi Histone H3 lysine 9 and H4 lysine 20 trimethylation and the expression of Suv4-20h2 and Suv-39h1 histone methyltransferases in hepatocarcinogenesis induced by methyl deficiency in rats
    Igor P Pogribny
    National Center for Toxicological Research, Jefferson, AR 72078, National Cancer Institute, Bethesda, MD 20892 and University of Tennessee, Knoxville, TN 37996, USA
    Carcinogenesis 27:1180-6. 2006
    ....
  12. ncbi Proceedings of the Trans-HHS Workshop: diet, DNA methylation processes and health
    Sharon A Ross
    Nutritional Science Research Group, Division of Cancer Prevention, National Cancer Institute, Bethesda, MD 20892, USA
    J Nutr 132:2329S-2332S. 2002
  13. ncbi Nanotechnology research: applications in nutritional sciences
    Pothur R Srinivas
    Atherothrombosis and Coronary Artery Diseases Branch, Division of Cardiovascular Sciences, National Heart, Lung, and Blood Institute, Bethesda, MD 20892, USA
    J Nutr 140:119-24. 2010
    ..These efforts could facilitate exploration of previously inaccessible cellular compartments and intracellular pathways and thus uncover strategies for new prevention and therapeutic modalities...
  14. ncbi Effect of long-term tamoxifen exposure on genotoxic and epigenetic changes in rat liver: implications for tamoxifen-induced hepatocarcinogenesis
    Volodymyr P Tryndyak
    Division of Biochemical Toxicology, National Center for Toxicological Research, Jefferson, AR 72079, USA
    Carcinogenesis 27:1713-20. 2006
    ..These data indicate the importance of genotoxic and epigenetic alterations in the etiology of tamoxifen-induced hepatocarcinogenesis...
  15. ncbi Dietary selenium affects homocysteine metabolism differently in Fisher-344 rats and CD-1 mice
    Eric O Uthus
    USDA, ARS, Grand Forks Human Nutrition Research Center, Grand Forks, ND 58202, USA
    J Nutr 137:1132-6. 2007
    ....
  16. ncbi Gene expression profiling reveals underlying molecular mechanisms of the early stages of tamoxifen-induced rat hepatocarcinogenesis
    Igor P Pogribny
    Division of Biochemical Toxicology, National Center for Toxicological Research, Jefferson, AR 72079, USA
    Toxicol Appl Pharmacol 225:61-9. 2007
    ..The early appearance of these alterations suggests their importance in the mechanism of neoplastic cell transformation induced by tamoxifen...
  17. ncbi Methyl deficiency, alterations in global histone modifications, and carcinogenesis
    Igor P Pogribny
    National Center for Toxicological Research, Jefferson, AR 72079, USA
    J Nutr 137:216S-222S. 2007
    ..These results illustrate the complexity and importance of histone modification changes in the etiology of hepatocarcinogenesis induced by MDD...
  18. ncbi Differential effects of dietary selenium (se) and folate on methyl metabolism in liver and colon of rats
    Eric O Uthus
    USDA ARS Grand Forks Human Nutrition Research Center, P O Box 9034, Grand Forks, ND 58202 9034, USA
    Biol Trace Elem Res 109:201-14. 2006
    ..This suggests that selenium deficiency appears to be a more important modifier of methyl metabolism than either adequate or supplemental selenium...
  19. ncbi Irreversible global DNA hypomethylation as a key step in hepatocarcinogenesis induced by dietary methyl deficiency
    Igor P Pogribny
    Division of Biochemical Toxicology, National Center for Toxicological Research, 3900 NCTR Dr, Jefferson, AR 72079, USA
    Mutat Res 593:80-7. 2006
    ..These results provide an experimental evidence and a mechanistic basis by which epigenetic alterations may contribute to the initiation and promotion steps of carcinogenesis...
  20. ncbi Epigenetic reprogramming of liver cells in tamoxifen-induced rat hepatocarcinogenesis
    Volodymyr P Tryndyak
    Division of Biochemical Toxicology, National Center for Toxicological Research, Jefferson, AR 72079, USA
    Mol Carcinog 46:187-97. 2007
    ....