ALISON SCHECTER

Summary

Affiliation: Mount Sinai School of Medicine
Country: USA

Publications

  1. ncbi Chemokine receptors in vascular smooth muscle
    Alison D Schecter
    The Zena and Michael A Wiener Cardiovascular Institute, New York, NY 14642, USA
    Microcirculation 10:265-72. 2003
  2. ncbi MCP-1-dependent signaling in CCR2(-/-) aortic smooth muscle cells
    Alison D Schecter
    The Cardiovascular Institute, Department of Medicine, Mount Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
    J Leukoc Biol 75:1079-85. 2004
  3. ncbi CCL11 (Eotaxin) induces CCR3-dependent smooth muscle cell migration
    Ravindra B Kodali
    Zena and Michael A Wiener Cardiovascular Institute and Department of Medicine, The Mount Sinai School of Medicine, New York, NY, USA
    Arterioscler Thromb Vasc Biol 24:1211-6. 2004
  4. ncbi CXCR4 modulates contractility in adult cardiac myocytes
    Robert T Pyo
    Zena and Michael A Wiener Cardiovascular Institute, The Mount Sinai School of Medicine, New York, NY 10029, USA
    J Mol Cell Cardiol 41:834-44. 2006
  5. ncbi Ambient air particulate matter exposure and tissue factor expression in atherosclerosis
    Qinghua Sun
    Division of Environmental Health Sciences, The Ohio State University, Columbus, Ohio, USA
    Inhal Toxicol 20:127-37. 2008
  6. ncbi Human immunodeficiency virus (HIV) infects human arterial smooth muscle cells in vivo and in vitro: implications for the pathogenesis of HIV-mediated vascular disease
    Eliseo A Eugenin
    Department of Pathology, Albert Einstein College of Medicine, Bronx, NY, USA
    Am J Pathol 172:1100-11. 2008

Research Grants

  1. MCP-1, TISSUE FACTOR, AND VASCULAR SMOOTH MUSCLE
    ALISON SCHECTER; Fiscal Year: 2002
  2. HIV, Chemokine Receptors, and Vascular Smooth Muscle
    ALISON SCHECTER; Fiscal Year: 2007

Collaborators

Detail Information

Publications6

  1. ncbi Chemokine receptors in vascular smooth muscle
    Alison D Schecter
    The Zena and Michael A Wiener Cardiovascular Institute, New York, NY 14642, USA
    Microcirculation 10:265-72. 2003
    ..It is likely that SMC are increasingly recognized as potential targets for chemokines, and that these effects may influence a variety of normal and pathological processes involving SMC such as atherosclerosis and arterial injury...
  2. ncbi MCP-1-dependent signaling in CCR2(-/-) aortic smooth muscle cells
    Alison D Schecter
    The Cardiovascular Institute, Department of Medicine, Mount Sinai School of Medicine, One Gustave L Levy Place, New York, NY 10029, USA
    J Leukoc Biol 75:1079-85. 2004
    ..This alternative receptor may be important in mediating some of the effects of MCP-1 in atherosclerotic arteries and in other inflammatory processes...
  3. ncbi CCL11 (Eotaxin) induces CCR3-dependent smooth muscle cell migration
    Ravindra B Kodali
    Zena and Michael A Wiener Cardiovascular Institute and Department of Medicine, The Mount Sinai School of Medicine, New York, NY, USA
    Arterioscler Thromb Vasc Biol 24:1211-6. 2004
    ..The major receptor for CCL11 is CCR3, which is found on leukocytes and on some nonleukocytic cells. We sought to determine whether vascular smooth muscle cells (SMCs) possessed functional CCR3...
  4. ncbi CXCR4 modulates contractility in adult cardiac myocytes
    Robert T Pyo
    Zena and Michael A Wiener Cardiovascular Institute, The Mount Sinai School of Medicine, New York, NY 10029, USA
    J Mol Cell Cardiol 41:834-44. 2006
    ..The specific mechanism of action involves alterations of calcium channel activity on the membrane. The presence of functional CXCR4 on cardiac myocytes introduces a new target for treating cardiac dysfunction...
  5. ncbi Ambient air particulate matter exposure and tissue factor expression in atherosclerosis
    Qinghua Sun
    Division of Environmental Health Sciences, The Ohio State University, Columbus, Ohio, USA
    Inhal Toxicol 20:127-37. 2008
    ..TF mRNA expression increased rapidly (within 1 h) in response to SRM PM. We conclude that in vivo and in vitro exposure to ambient air PM(2.5) induces TF expression...
  6. ncbi Human immunodeficiency virus (HIV) infects human arterial smooth muscle cells in vivo and in vitro: implications for the pathogenesis of HIV-mediated vascular disease
    Eliseo A Eugenin
    Department of Pathology, Albert Einstein College of Medicine, Bronx, NY, USA
    Am J Pathol 172:1100-11. 2008
    ..Our data suggest that direct infection of human arterial SMCs by HIV represents a potential mechanism in a multifactorial paradigm to explain the exacerbated atherosclerosis and vasculopathy reported in individuals infected with HIV...

Research Grants6

  1. MCP-1, TISSUE FACTOR, AND VASCULAR SMOOTH MUSCLE
    ALISON SCHECTER; Fiscal Year: 2002
    ..The receptor will be expressed in COS cells to study the properties of the new receptor in mammalian cells and, in particular, to examine its coupling to TF induction. ..
  2. HIV, Chemokine Receptors, and Vascular Smooth Muscle
    ALISON SCHECTER; Fiscal Year: 2007
    ..Furthermore, given that HIV patients appear to have accelerated atherosclerosis, these studies will provide potential mechanism(s) responsible for HIV-associated vasculopathies. ..