Research Topics
| Ute E SchwabSummaryAffiliation: Cornell University Country: USA Publications
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Detail Information
Publications
Alternative sigma factor sigmaB is not essential for listeria monocytogenes surface attachmentUte Schwab
Department of Food Science, Cornell University, Ithaca, New York 14853, USA
J Food Prot 68:311-7. 2005..Our data suggest that sigmaB is not required for initial surface attachment of L. monocytogenes...
Equine bronchial epithelial cells differentiate into ciliated and mucus producing cells in vitroUte E Schwab
Department of Microbiology and Immunology, College of Veterinary Medicine, Cornell University, Ithaca, NY, USA
In Vitro Cell Dev Biol Anim 46:102-6. 2010..Furthermore, the demonstration of the two major gel-forming mucin species, Muc5ac and Muc5b, in our bronchial epithelial cell culture system validates this method for studies of respiratory tract disease of the horse...
Transcriptional responses of Mycobacterium tuberculosis to lung surfactantUte Schwab
Department of Microbiology and Immunology, Cornell University College of Veterinary Medicine, Ithaca, NY 14853, USA
Microb Pathog 46:185-93. 2009....
The Listeria monocytogenes prfAP2 promoter is regulated by sigma B in a growth phase dependent mannerUte Schwab
Department of Food Science, Cornell University, 415 Stocking Hall, Ithaca, NY 14853, USA
FEMS Microbiol Lett 245:329-36. 2005..We conclude that prfAP2 is directly regulated by sigma(B)...
Role of actin filament network in Burkholderia multivorans invasion in well-differentiated human airway epitheliaUte E Schwab
Cystic Fibrosis Pulmonary Research and Treatment Center, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599 7248, USA
Infect Immun 71:6607-9. 2003..Our data suggest that an intact actin cytoskeleton is required for biofilm formation but not single cell entry or paracytosis...
A physical linkage between cystic fibrosis airway surface dehydration and Pseudomonas aeruginosa biofilmsHirotoshi Matsui
Cystic Fibrosis/Pulmonary Research and Treatment Center, University of North Carolina, Chapel Hill, NC 27599, USA
Proc Natl Acad Sci U S A 103:18131-6. 2006..aeruginosa biofilms in CF via changes in the hydration-dependent physical-chemical properties of mucus and suggest that the thickened mucus gel model will be useful to develop therapies of P. aeruginosa biofilms in CF airways...
Reduced three-dimensional motility in dehydrated airway mucus prevents neutrophil capture and killing bacteria on airway epithelial surfacesHirotoshi Matsui
Cystic Fibrosis Pulmonary Research and Treatment Center and Department of Biochemistry and Biophysics, University of North Carolina, Chapel Hill, NC 27599, USA
J Immunol 175:1090-9. 2005..We conclude that concentrated ("thick") mucus inhibits neutrophil migration and killing and is a key component in the failure of defense against chronic airways infection in CF...
Disposition and intracellular levels of moxifloxacin in human THP-1 monocytes in unstimulated and stimulated conditionsIris H Hall
Division of Medicinal Chemistry, School of Pharmacy, University of North Carolina, Chapel Hill, NC 27599 7360, USA
Int J Antimicrob Agents 22:579-87. 2003..Increased amounts of the drug were found in the THP-1 monocytes as well as the endoplasmic reticulum and the isolated phagosomes after stimulation by zymogen A, Staphylococcus aureus or Streptococcus pneumoniae...
Human THP-1 monocyte uptake and cellular disposition of 14C-grepafloxacinIris H Hall
Division of Medicinal Chemistry, School of Pharmacy, University of North Carolina, Chapel Hill 27599-7360, USA
J Infect Chemother 10:11-8. 2004..Metabolic inhibitors, suppressors of K(+)/Cl(-) and Cl(-) transporters, inhibitors of the phagocytic process, low temperature, and low pH inhibited grepafloxacin uptake by THP-1 monocytes...
Disposition and intracellular activity of azithromycin in human THP-1 acute monocytesIris H Hall
Division of Medicinal Chemistry, School of Pharmacy, University of North Carolina, Chapel Hill, NC 27599 7360, USA
Int J Antimicrob Agents 20:348-60. 2002..Nevertheless, suppression of these chemical mediators and hydrolytic enzyme activities would reduce the infection and the spread to adjacent areas...
