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Genomes and GenesSpecies | Norman LatovSummaryAffiliation: Cornell University Country: USA Publications
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Publications
Biomarkers of CIDP in patients with diabetes or CMT1Norman Latov
Department of Neurology and Neuroscience, Weill Medical College of Cornell University, 1305 York Avenue, New York, NY 10021, USA
J Peripher Nerv Syst 16:14-7. 2011..Studies are needed to establish electrodiagnostic criteria for CIDP in patients with diabetes, and to identify biomarkers that distinguish between inflammatory and non-inflammatory causes of demyelinating neuropathy...- Timing and course of clinical response to intravenous immunoglobulin in chronic inflammatory demyelinating polyradiculoneuropathyNorman Latov
Peripheral Neuropathy Center, Cornell University, New York, New York 10021, USA
Arch Neurol 67:802-7. 2010..To investigate the timing, course, and clinical characteristics of the response to intravenous immunoglobulin in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP)... - Neuropathy and cognitive impairment following vaccination with the OspA protein of Borrelia burgdorferiNorman Latov
Department of Neurology and Neurosciences, Weill Medical College of Cornell University, New York, NY 10022, USA
J Peripher Nerv Syst 9:165-7. 2004.... - Effector mechanisms in anti-MAG antibody-mediated and other demyelinating neuropathiesNorman Latov
Department of Neurology and Neuroscience, Peripheral Neuropathy Center, Weill Medical College of Cornell University, New York, NY 10022, USA
J Neurol Sci 220:127-9. 2004 - Sensory CIDP presenting as cryptogenic sensory polyneuropathyRussell L Chin
Weill Medical College of Cornell University, Department of Neurology, New York, NY 10022, USA
J Peripher Nerv Syst 9:132-7. 2004..Sural nerve biopsy should be considered in patients with progressive, predominantly large fiber sensory neuropathy of otherwise unknown etiology, as they may have sensory CIDP that responds to therapy... - Characterization of neuropathies associated with elevated IgM serum levelsAdina R Goldfarb
Peripheral Neuropathy Center, Department of Neurology, Weill Medical College of Cornell University, 635 Madison Ave, Suite 400, New York, NY 10022, USA
J Neurol Sci 228:155-60. 2005..In contrast to the IgM monoclonal gammopathies the neuropathy associated with polyclonal IgM gammopathy has not been well characterized... - Immune cross-reactivity in celiac disease: anti-gliadin antibodies bind to neuronal synapsin IArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, and St Vincent s Hospital Manhattan, New York, NY 10021, USA
J Immunol 178:6590-5. 2007..Anti-gliadin Abs from patients with celiac disease also bound to the protein. Such cross-reactivity may provide clues into the pathogenic mechanism of the neurologic deficits that are associated with gluten sensitivity... - Small-fiber neuropathy/neuronopathy associated with celiac disease: skin biopsy findingsThomas H Brannagan
Department of Neurology, Weill Medical College of Cornell University, New York, NY 10022, USA
Arch Neurol 62:1574-8. 2005..Celiac disease (CD) is increasingly recognized in North America and is associated with a peripheral neuropathy... - Differential gene expression in chronic inflammatory demyelinating polyneuropathy (CIDP) skin biopsiesGrace Lee
Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York City, NY 10021, USA
J Neurol Sci 290:115-22. 2010..The findings indicate the presence of inflammatory changes in the skin of patients with CIDP... - Comparison of electrodiagnostic abnormalities and criteria in a cohort of patients with chronic inflammatory demyelinating polyneuropathyPaul Magda
Peripheral Neuropathy Center, Department of Neurology, Weill Medical College of Cornell University, New York, NY 10022, USA
Arch Neurol 60:1755-9. 2003..Application of such criteria in clinical practice may miss the diagnosis in potentially treatable patients... - Anti-neural antibody reactivity in patients with a history of Lyme borreliosis and persistent symptomsAbhishek Chandra
Department of Neurology and Neuroscience, Cornell University, New York, NY 10065, USA
Brain Behav Immun 24:1018-24. 2010..The results provide evidence for the existence of a differential immune system response in PLS, offering new clues about the etiopathogenesis of the disease that may prove useful in devising more effective treatment strategies... - Gene expression profiling in chronic inflammatory demyelinating polyneuropathySusanne Renaud
Department of Neurology and Neurosciences, Weill Medical College, Cornell University, New York, NY, USA
J Neuroimmunol 159:203-14. 2005..Differential gene expression may help distinguish between CIDP, VAS and NN in sural nerve biopsies and identify genes that may be involved in disease pathogenesis... - Cerebellar ataxia and coeliac diseaseHoward W Sander
Peripheral Neuropathy Center, College of Physicians and Surgeons, Columbia University, New York, NY 10022, USA
Lancet 362:1548. 2003 - Etanercept (Enbrel) therapy for chronic inflammatory demyelinating polyneuropathyRussell L Chin
Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, NY 10022, USA
J Neurol Sci 210:19-21. 2003.... - Antiganglioside antibodies in multifocal acquired sensory and motor neuropathyArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, New York, NY 10021, USA
Arch Neurol 60:42-6. 2003..Multifocal acquired demyelinating sensory and motor neuropathies are considered autoimmune and responsive to immunotherapy. In the absence of demyelination, however, they are considered idiopathic if no other cause is found... - Transglutaminase-independent binding of gliadin to intestinal brush border membrane and GM1 gangliosideArmin Alaedini
Department of Neurology and Neuroscience, 1300 York Ave, Weill Medical College of Cornell University, New York, NY 10021, USA
J Neuroimmunol 177:167-72. 2006..Furthermore, binding of gliadin to GM1 on the intestinal epithelium might have a role in the anti-gliadin immune response and contribute to the intestinal inflammatory reaction in celiac disease... - Demyelinating neuropathy in patients with hepatitis C virus infectionRussell L Chin
Peripheral Neuropathy Center, Weill Cornell Medical College, New York, NY 10021, USA
J Clin Neuromuscul Dis 11:209-12. 2010..Immunomodulatory therapy may be beneficial in some cases. Testing for HCV should be considered, especially in patients with hypergammaglobulinemia or IgM monoclonal gammopathy... - Epitope mapping of antibodies to VlsE protein of Borrelia burgdorferi in post-Lyme disease syndromeAbhishek Chandra
Department of Neurology and Neuroscience, Weill Cornell Medical College, Cornell University, New York, NY, USA
Clin Immunol 141:103-10. 2011..The crystal structure of VlsE indicated that the newly described epitopes form a contiguous region in the surface-exposed membrane-proximal part of the monomeric form of the protein... - Antibodies against OspA epitopes of Borrelia burgdorferi cross-react with neural tissueArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, 1300 York Ave, New York, NY 10021, USA
J Neuroimmunol 159:192-5. 2005..Antibodies against two of the homologous OspA peptides were found to react with neurons in human brain, spinal cord and dorsal root ganglia by immunohistochemistry... - Anti-Borrelia burgdorferi antibody profile in post-Lyme disease syndromeAbhishek Chandra
Department of Neurology and Neuroscience, Weill Cornell Medical College, Cornell University, New York, New York, USA
Clin Vaccine Immunol 18:767-71. 2011..005). The described antiborrelia antibody profile in PLDS offers clues about the course of the antecedent infection in affected patients, which may be useful for understanding the pathogenic mechanism of the disease... - Research criteria for defining patients with CIDPHoward W Sander
Peripheral Neuropathy Center, Department of Neurology, Weill Medical College of Cornell University, New York, NY 10022, USA
Neurology 60:S8-15. 2003..Therefore, although patients may not meet the diagnostic criteria for inclusion in clinical trials of CIDP, they may still benefit from current and future treatments used in CIDP... - Up-regulation of apoptosis and regeneration genes in the dorsal root ganglia during cisplatin treatmentArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, New York, NY 10065, USA
Exp Neurol 210:368-74. 2008..As such, the identified genes may represent candidate processes and pathways that should be considered as targets for therapeutic intervention in cisplatin-induced neuropathy... - Induction of human IgM and IgG anti-GM1 antibodies in transgenic mice in response to lipopolysaccharides from Campylobacter jejuniGrace Lee
Department of Neurology and Neuroscience, Weill Medical College, Cornell University, New York, NY, USA
J Neuroimmunol 146:63-75. 2004.... - Detection of anti-ganglioside antibodies in Guillain-Barré syndrome and its variants by the agglutination assayArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, 525 E 68th St, Room LC 807, New York, NY 10021, USA
J Neurol Sci 196:41-4. 2002..Some of the sera reacted with more than one ganglioside. Antibodies were not found in the control sera that were studied. The agglutination assay may be useful for rapid screening of GBS sera for antibodies to multiple gangliosides... - Prolongation of the tibialis anterior CMAP duration in chronic inflammatory demyelinating polyneuropathyHiroyuki Nodera
Peripheral Neuropathy Center, Department of Neurology, Weill Cornell Medical College, New York, NY, USA
Clin Neurophysiol 123:393-8. 2012..To assess the duration of the distal compound muscle action potential (dCMAP) recording from the tibialis anterior (TA) as a supportive electrodiagnostic feature in chronic inflammatory demyelinating polyneuropathy (CIDP)... - Diagnosis of CIDPNorman Latov
Peripheral Neuropathy Center, Weill Medical College of Cornell University, New York, NY 10022, USA
Neurology 59:S2-6. 2002..A favorable response to therapy, consisting of stabilization or improvement of the neuropathy, would confirm the diagnosis... - Specific electron transport chain abnormalities in amyotrophic lateral sclerosisJerry Lin
Multiple Sclerosis Research Center of New York, 521 West 57th Street, 4th Fl, New York, NY 10019, USA
J Neurol 256:774-82. 2009..It is possible that this may have an effect on oxygen-dependent metabolic pathways. Human motor neurons may be particularly susceptible to injury if there is sub-optimal oxidative metabolism... - Ganglioside reactive antibodies in the neuropathy associated with celiac diseaseArmin Alaedini
Department of Neurology and Neuroscience, Cornell University, 525 E 68th St, LC 807, New York, NY 10021, USA
J Neuroimmunol 127:145-8. 2002..The neuropathy of celiac disease may be autoimmune and associated with anti-ganglioside antibodies. The presence of IgG reactivity furthermore implicates a T cell-mediated response to ganglioside antigens... - Quantitative sensory testing: high sensitivity in small fiber neuropathy with normal NCS/EMGPaul Magda
Peripheral Neuropathy Center, Department of Neurology and Neuroscience, Weill Medical College of Comell University, New York, NY 10022, USA
J Peripher Nerv Syst 7:225-8. 2002..QST is therefore highly effective in documenting dysfunction in small fiber neuropathy patients... - Central nervous system manifestations of rheumatologic diseasesRussell L Chin
Peripheral Neuropathy Center, Department of Neurology, Department of Neurology and Neuroscience, Weill Medical College of Cornell University, New York, New York 10022, USA
Curr Opin Rheumatol 17:91-9. 2005..To summarize the current literature on central nervous system manifestations of vasculitides and connective tissue diseases... - Altered subicular MAP2 immunoreactivity in schizophreniaGorazd Rosoklija
Department of Neuroscience, New York State Psychiatric Institute, New York, NY 10032, USA
Prilozi 26:13-34. 2005..The chemical or structural abnormalities underlying decreased MAP2 immunoreactivity in schizophrenia remain to be determined...