Sabino Vesce

Summary

Affiliation: Buck Institute for Age Research
Country: USA

Publications

  1. ncbi Acute glutathione depletion restricts mitochondrial ATP export in cerebellar granule neurons
    Sabino Vesce
    Buck Institute for Age Research, Novato, California 94945, USA
    J Biol Chem 280:38720-8. 2005
  2. ncbi Relationships between superoxide levels and delayed calcium deregulation in cultured cerebellar granule cells exposed continuously to glutamate
    Sabino Vesce
    Buck Institute for Age Research, Novato, California 94945, USA
    J Neurochem 90:683-93. 2004
  3. ncbi Interactions between mitochondrial bioenergetics and cytoplasmic calcium in cultured cerebellar granule cells
    David G Nicholls
    Buck Institute for Age Research, 8001 Redwood Boulevard, Novato, CA 94945, USA
    Cell Calcium 34:407-24. 2003
  4. ncbi Bioenergetics of mitochondria in cultured neurons and their role in glutamate excitotoxicity
    David G Nicholls
    Buck Institute for Age Research, Novato, CA 94945, USA
    J Neurosci Res 85:3206-12. 2007
  5. ncbi Alternative, nonapoptotic programmed cell death: mediation by arrestin 2, ERK2, and Nur77
    Susana Castro-Obregon
    Buck Institute for Age Research, Novato, California 94945 1400, USA
    J Biol Chem 279:17543-53. 2004
  6. ncbi Barbiturates induce mitochondrial depolarization and potentiate excitotoxic neuronal death
    Christopher M Anderson
    Department of Neurology, University of California, San Francisco, USA
    J Neurosci 22:9203-9. 2002
  7. ncbi Glutamate release from astrocytes in physiological conditions and in neurodegenerative disorders characterized by neuroinflammation
    Sabino Vesce
    Department of Cell Biology and Morphology, University of Lausanne, Rue du Bugnon 9, 1005 Lausanne, Switzerland
    Int Rev Neurobiol 82:57-71. 2007

Collaborators

Detail Information

Publications7

  1. ncbi Acute glutathione depletion restricts mitochondrial ATP export in cerebellar granule neurons
    Sabino Vesce
    Buck Institute for Age Research, Novato, California 94945, USA
    J Biol Chem 280:38720-8. 2005
    ..The initial bioenergetic consequence of neuronal GSH depletion in this model is thus an inhibition of ATP export, which precedes other forms of mitochondrial dysfunction...
  2. ncbi Relationships between superoxide levels and delayed calcium deregulation in cultured cerebellar granule cells exposed continuously to glutamate
    Sabino Vesce
    Buck Institute for Age Research, Novato, California 94945, USA
    J Neurochem 90:683-93. 2004
    ..Furthermore, the majority of superoxide is produced in the cytoplasm rather than in mitochondria...
  3. ncbi Interactions between mitochondrial bioenergetics and cytoplasmic calcium in cultured cerebellar granule cells
    David G Nicholls
    Buck Institute for Age Research, 8001 Redwood Boulevard, Novato, CA 94945, USA
    Cell Calcium 34:407-24. 2003
    ....
  4. ncbi Bioenergetics of mitochondria in cultured neurons and their role in glutamate excitotoxicity
    David G Nicholls
    Buck Institute for Age Research, Novato, CA 94945, USA
    J Neurosci Res 85:3206-12. 2007
    ..This impacts on current hypotheses relating to the neuroprotective effects of mild mitochondrial uncoupling...
  5. ncbi Alternative, nonapoptotic programmed cell death: mediation by arrestin 2, ERK2, and Nur77
    Susana Castro-Obregon
    Buck Institute for Age Research, Novato, California 94945 1400, USA
    J Biol Chem 279:17543-53. 2004
    ..These results suggest that Nur77, which is regulated by a MAPK pathway activated via arrestin 2, modulates NK(1)R-mediated nonapoptotic pcd...
  6. ncbi Barbiturates induce mitochondrial depolarization and potentiate excitotoxic neuronal death
    Christopher M Anderson
    Department of Neurology, University of California, San Francisco, USA
    J Neurosci 22:9203-9. 2002
    ..Effects of barbiturates on neuronal mitochondria should be considered in experimental and clinical application of these drugs...
  7. ncbi Glutamate release from astrocytes in physiological conditions and in neurodegenerative disorders characterized by neuroinflammation
    Sabino Vesce
    Department of Cell Biology and Morphology, University of Lausanne, Rue du Bugnon 9, 1005 Lausanne, Switzerland
    Int Rev Neurobiol 82:57-71. 2007
    ....