Research Topics
| Edward J FilardoSummaryAffiliation: Brown University Country: USA Publications
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Detail Information
Publications
Epidermal growth factor receptor (EGFR) transactivation by estrogen via the G-protein-coupled receptor, GPR30: a novel signaling pathway with potential significance for breast cancerEdward J Filardo
Department of Medicine, Division of Clinical Pharmacology, Rhode Island Hospital and Brown University, Aldrich Bldg Rm 718, 593 Eddy Street, Providence, RI 02903, USA
J Steroid Biochem Mol Biol 80:231-8. 2002..This novel mechanism by which estrogen activates growth factor-dependent signaling and its implications for breast cancer biology are discussed further in this review...
Association of the membrane estrogen receptor, GPR30, with breast tumor metastasis and transactivation of the epidermal growth factor receptorEdward J Filardo
Department of Medicine, Rhode Island Hospital and Brown University School of Medicine, Providence, RI 02903, USA
Steroids 73:870-3. 2008....
Coordinate regulation of estrogen-mediated fibronectin matrix assembly and epidermal growth factor receptor transactivation by the G protein-coupled receptor, GPR30Jeffrey A Quinn
Department of Medicine, Brown University School of Medicine, Providence, Rhode Island 02903, USA
Mol Endocrinol 23:1052-64. 2009..Our results suggest that GPR30 coordinates estrogen-mediated FN matrix assembly and growth factor release in human breast cancer cells via a Shc-dependent signaling mechanism that activates integrin alpha5beta1...
Estrogen action via the G protein-coupled receptor, GPR30: stimulation of adenylyl cyclase and cAMP-mediated attenuation of the epidermal growth factor receptor-to-MAPK signaling axisEdward J Filardo
Department of Surgery, Rhode Island Hospital, and Brown University, Providence, Rhode Island 02903, USA
Mol Endocrinol 16:70-84. 2002....
Retrograde transport of the transmembrane estrogen receptor, G-protein-coupled-receptor-30 (GPR30/GPER) from the plasma membrane towards the nucleusShi Bin Cheng
Division of Hematology and Oncology Rhode Island Hospital and Brown University School of Medicine Providence, RI 02903, USA
Steroids 76:892-6. 2011..g. ?1AR) or are degraded in lysosomes (e.g. CXCR4). The accumulation of GPER in the perinuclear space and its possible significance for attenuating estrogen action via this newly recognized membrane estrogen receptor is discussed herein...
GPR30: a seven-transmembrane-spanning estrogen receptor that triggers EGF releaseEdward J Filardo
Department of Medicine, Rhode Island Hospital and Brown University School of Medicine, Providence, RI 02903, USA
Trends Endocrinol Metab 16:362-7. 2005..Thus, estrogen can signal by the same mechanism as various other hormones, through a specific 7TM receptor...
Distribution of GPR30, a seven membrane-spanning estrogen receptor, in primary breast cancer and its association with clinicopathologic determinants of tumor progressionEdward J Filardo
Department of Medicine, Rhode Island Hospital, and Department of Pathology, Brown University School of Medicine, Providence, Rhode Island 02903, USA
Clin Cancer Res 12:6359-66. 2006..Here, the significance of GPR30 in human breast cancer was evaluated by comparing its relationship to steroid hormone receptor expression and tumor progression variables...
Down-modulation of the G-protein-coupled estrogen receptor, GPER, from the cell surface occurs via a trans-Golgi-proteasome pathwayShi Bin Cheng
Division of Hematology and Oncology, Rhode Island Hospital and Brown University, Providence, Rhode Island 02903, USA
J Biol Chem 286:22441-55. 2011..Our results provide an explanation as to why GPER is not readily detected on the cell surface in some cell types and further suggest that TGN serves as the checkpoint for degradation of endocytosed GPER...
