Research Topics
| K ZacharowskiSummaryAffiliation: University of Bristol Country: UK Publications
| Collaborators
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Detail Information
Publications
CpG oligonucleotide activates Toll-like receptor 9 and causes lung inflammation in vivoPascal Knuefermann
Department for Anesthesiology and Intensive Care Medicine, University Hospital Bonn, Sigmund Freud Strasse 25, 53125 Bonn, Germany
Respir Res 8:72. 2007..Therefore, the hypothesis was tested whether CpG-ODN administration induces an inflammatory response in the lung via TLR9 in vivo...
[New reflections on inflammation and coagulation]K Zacharowski
Dept of Anaesthesia, BHI, Bristol Royal Infirmary, BS2 8HW, Bristol, United Kingdom
Anaesthesist 56:482-4. 2007..Great efforts are being undertaken worldwide to understand the regulation of inflammation in order to develop new drugs which can modulate the pathologic inflammation reaction...
The effects of the fibrin-derived peptide Bbeta(15-42) in acute and chronic rodent models of myocardial ischemia-reperfusionKai Zacharowski
Molecular Cardioprotection and Inflammation Group, Department of Anesthesia, University Hospital of Dusseldorf, Dusseldorf, Germany
Shock 27:631-7. 2007..In rodents, Bbeta(15-42) inhibits proinflammatory cytokine release and is cardioprotective during ischemia-reperfusion injury...
Inflammatory response and cardioprotection during open-heart surgery: the importance of anaestheticsM S Suleiman
Bristol Heart Institute and Department of Anaesthesia, Faculty of Medicine and Dentistry, Bristol Royal Infirmary, University of Bristol, Bristol, UK
Br J Pharmacol 153:21-33. 2008..This review describes the main mediators of myocardial injury during open heart surgery, explores available evidence of anaesthetics induced cardioprotection and addresses the efforts made to translate bench work into clinical practice...
Role of protein kinase C-epsilon (PKCepsilon) in isoflurane-induced cardioprotectionD Obal
Department of Anesthesiology, University Hospital, Moorenstrasse 5, , Germany
Br J Anaesth 94:166-73. 2005..0 or 1.75 MAC. CONCLUSIONS: Although isoflurane preconditioning resulted in a reduction in infarct size at all concentrations used, the protection was mediated by phosphorylation and translocation of PKCepsilon only at 0.4 MAC...
Biglycan is required for adaptive remodeling after myocardial infarctionD Westermann
Abteilung für Kardiologie und Pneumologie, Charité Universitätsklinikum Berlin, Campus Benjamin Franklin, Berlin, Germany
Circulation 117:1269-76. 2008..The present study investigates the role of the small leucine-rich proteoglycan biglycan during cardiac extracellular matrix remodeling and cardiac hemodynamics after MI...
