Research Topics
Genomes and Genes | Darren L AsquithSummaryAffiliation: University of Glasgow Country: UK Publications
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Publications
Liver X receptor agonism promotes articular inflammation in murine collagen-induced arthritisDarren L Asquith
Glasgow Biomedical Research Center, Division of Infection, Immunity and Inflammation, University of Glasgow, 120 University Place, Glasgow, UK
Arthritis Rheum 60:2655-65. 2009..This study was therefore undertaken to explore the biologic mechanisms of LXRs in vivo and in vitro in an experimental inflammatory arthritis model...
Interleukin-33 induces protective effects in adipose tissue inflammation during obesity in miceAshley M Miller
Division of Immunology, Infection and Inflammation, GBRC, University of Glasgow, Glasgow, United Kingdom
Circ Res 107:650-8. 2010..The cytokine interleukin (IL)-33 and its receptor ST2 are expressed in adipose tissue, but their role in adipose tissue inflammation during obesity is unclear...
Apolipoprotein E-deficient mice are resistant to the development of collagen-induced arthritisDarren L Asquith
University of Glasgow, Glasgow, UK
Arthritis Rheum 62:472-7. 2010..To determine whether elevated serum lipid levels resulting from feeding animals a high-fat diet can affect the inflammatory process in C57BL/6 (B6) wild-type (WT) and B6 ApoE(-/-) mouse models of collagen-induced arthritis (CIA)...
IL-33 induces skin inflammation with mast cell and neutrophil activationAxel J Hueber
Glasgow Biomedical Research Centre, University of Glasgow, UK
Eur J Immunol 41:2229-37. 2011..These results therefore demonstrate that IL-33 may play a role in psoriasis-like plaque inflammation. IL-33 targeting may provide a new treatment strategy for psoriasis...
Simultaneous activation of the liver X receptors (LXR? and LXR?) drives murine collagen-induced arthritis disease pathologyDarren L Asquith
Correspondence to Dr Darren L Asquith, Glasgow Biomedical Research Centre, Institute of Immunity, Infection and Inflammation, College of Medical, Veterinary and Life Sciences, 120 University Place, University of Glasgow, Glasgow G12 8TA, UK
Ann Rheum Dis 70:2225-8. 2011..It has previously been shown that dual activation of the Liver X Receptors (LXR? and LXR?) by the agonist, GW3965, enhances pathology in a murine model of collagen-induced arthritis...
Mast cells express IL-17A in rheumatoid arthritis synoviumAxel J Hueber
Division of Infection, Immunity and Inflammation, Glasgow Biomedical Research Centre, University of Glasgow, Glasgow, United Kingdom
J Immunol 184:3336-40. 2010....
Animal models of rheumatoid arthritisDarren L Asquith
Division of Immunology, Infection and Inflammation, Glasgow Biomedical Research Centre, University of Glasgow, Glasgow, Scotland, UK
Eur J Immunol 39:2040-4. 2009..Here, we describe these animal models and discuss their advantages and limitations...
IL-33 reduces the development of atherosclerosisAshley M Miller
Division of Immunology, Infection and Inflammation, Glasgow Biomedical Research Centre, University of Glasgow, Glasgow G12 8TA, Scotland, UK
J Exp Med 205:339-46. 2008..In conclusion, IL-33 may play a protective role in the development of atherosclerosis via the induction of IL-5 and ox-LDL antibodies...
MicroRNA-155 as a proinflammatory regulator in clinical and experimental arthritisMariola Kurowska-Stolarska
Institute of Infection, Immunity and Inflammation, College of Medical, Veterinary and Life Sciences, University of Glasgow, Glasgow G12 8TA, United Kingdom
Proc Natl Acad Sci U S A 108:11193-8. 2011..Our data therefore identify a role of miR-155 in clinical and experimental arthritis and suggest that miR-155 may be an intriguing therapeutic target...
Emerging cytokine targets in rheumatoid arthritisDarren L Asquith
Glasgow Biomedical Research Centre, Division of Immunology, Infection and Inflammation, University of Glasgow, Glasgow, UK
Curr Opin Rheumatol 19:246-51. 2007..Partial and non-responses to tumour necrosis factor blocking agents, however, together with the increasing clinical drive to remission induction, requires that further therapeutic targets be identified...
