Research Topics
Species | J C MansonSummaryAffiliation: Institute for Animal Health Country: UK Publications
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Detail Information
Publications
A single amino acid alteration (101L) introduced into murine PrP dramatically alters incubation time of transmissible spongiform encephalopathyJ C Manson
BBSRC Neuropathogenesis Unit, Institute for Animal Health, Ogston Building, West Mains Road, Edinburgh EH9 3JF
EMBO J 18:6855-64. 1999..Additionally, a rapid TSE transmission was demonstrated despite extremely low levels of disease-associated PrP...
The transmissible spongiform encephalopathies: emerging and declining epidemicsJ C Manson
Institute for Animal Health, Neuropathogenesis Unit, Ogston Building, West Mains Road, Edinburgh EH9 3JF, Scotland, UK
Biochem Soc Trans 34:1155-8. 2006..This review examines the emergence of various TSE strains and their transmission, and discusses disease surveillance and control...
Changing a single amino acid in the N-terminus of murine PrP alters TSE incubation time across three species barriersR M Barron
Institute for Animal Health, Neuropathogenesis Unit, Edinburgh, UK
EMBO J 20:5070-8. 2001..These findings suggest a critical role for the structurally 'flexible' region of PrP in agent replication and targeting of TSE pathology...
Comparison of expression patterns of PrP mRNA in the developing sheep and mouseN Hunter
Institute for Animal Health, AFRC/MRC Neuropathogenesis Unit, Edinburgh, Scotland
Ann N Y Acad Sci 724:353-4. 1994
Predicting susceptibility and incubation time of human-to-human transmission of vCJDM T Bishop
National CJD Surveillance Unit, Bryan Matthews Building, Western General Hospital, Edinburgh, UK
Lancet Neurol 5:393-8. 2006..A lengthy preclinical disease is predicted by these models, which may represent a risk for further disease transmission and thus a significant public-health issue...
PrP gene dosage determines the timing but not the final intensity or distribution of lesions in scrapie pathologyJ C Manson
Institute for Animal Health, BBSRC and MRC Neuropathogenesis Unit, Edinburgh
Neurodegeneration 3:331-40. 1994..These results show that signs of disease, vacuolation and PrP deposition are dependent upon PrPc in a rate dependent manner...
Double replacement gene targeting for the production of a series of mouse strains with different prion protein gene alterationsR C Moore
Institute of Cell and Molecular Biology, University of Edinburgh, Scotland
Biotechnology (N Y) 13:999-1004. 1995..We anticipate that this procedure will have applications to the study of human inherited diseases and the development of therapies...
Mice with gene targetted prion protein alterations show that Prnp, Sinc and Prni are congruentR C Moore
Institute of Cell and Molecular Biology, University of Edinburgh, UK
Nat Genet 18:118-25. 1998..Challenge with a mouse-adapted BSE strain results in dramatically shortened incubation times and demonstrates that PrP dimorphisms at codon 108 and/or 189 control incubation time, and that Sinc/Prni and Prnp are congruent...
Altered circadian activity rhythms and sleep in mice devoid of prion proteinI Tobler
Institute of Pharmacology, University of Zurich, Switzerland
Nature 380:639-42. 1996....
129/Ola mice carrying a null mutation in PrP that abolishes mRNA production are developmentally normalJ C Manson
Institute for Animal Health, Edinburgh
Mol Neurobiol 8:121-7. 1994..The mice are being used in experiments designed to address the role of PrP in the pathogenesis of scrapie and the replication of infectivity...
In vitro amplification and detection of variant Creutzfeldt-Jakob disease PrPScM Jones
National CJD Surveillance Unit, School of Molecular and Clinical Medicine Pathology, University of Edinburgh, Western General Hospital, Edinburgh EH4 2XU, UK
J Pathol 213:21-6. 2007....
Increased levels of oxidative stress markers detected in the brains of mice devoid of prion proteinB S Wong
Institute of Pathology, Case Western Reserve University School of Medicine, Cleveland, Ohio 44106, USA
J Neurochem 76:565-72. 2001..Taken together, these findings are indicative of a role for PrP in oxidative homeostasis in vivo...
