Research Topics
Species | Julia J InglisSummaryAffiliation: Imperial College Country: UK Publications
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Detail Information
Publications
Collagen-induced arthritis as a model of hyperalgesia: functional and cellular analysis of the analgesic actions of tumor necrosis factor blockadeJulia J Inglis
Kennedy Institute of Rheumatology, Imperial College London, London, UK
Arthritis Rheum 56:4015-23. 2007..We then validated the model using anti-tumor necrosis factor (anti-TNF) therapy, which has analgesic effects in models of inflammation as well as in human RA...
Regulation of pain sensitivity in experimental osteoarthritis by the endogenous peripheral opioid systemJulia J Inglis
Kennedy Institute of Rheumatology, Imperial College London, London, UK
Arthritis Rheum 58:3110-9. 2008..However, up to 40% of individuals with radiologic damage are asymptomatic. The purpose of this study was to assess the role of the endogenous opioid system in delaying the onset of pain in a murine model of osteoarthritis (OA)...
Blockade of tumor necrosis factor in collagen-induced arthritis reveals a novel immunoregulatory pathway for Th1 and Th17 cellsClare A Notley
Kennedy Institute of Rheumatology Division, Imperial College London, London, W6 8LH, England, UK
J Exp Med 205:2491-7. 2008....
Apremilast, a novel PDE4 inhibitor, inhibits spontaneous production of tumour necrosis factor-alpha from human rheumatoid synovial cells and ameliorates experimental arthritisFiona E McCann
The Kennedy Institute of Rheumatology, Imperial College London, 65 Aspenlea Road, London, W6 8LH, UK
Arthritis Res Ther 12:R107. 2010..We assessed the anti-inflammatory effects of a novel PDE4 inhibitor, apremilast, in human synovial cells from rheumatoid arthritis (RA) patients, as well as two murine models of arthritis...
Treatment of murine osteoarthritis with TrkAd5 reveals a pivotal role for nerve growth factor in non-inflammatory joint painKay E McNamee
Kennedy Institute of Rheumatology, Imperial College London, London W6 8LH, UK
Pain 149:386-92. 2010..They also suggest that, unlike post-operative pain, induction of pain in OA may not necessarily be driven by classical inflammatory processes...
Protocol for the induction of arthritis in C57BL/6 miceJulia J Inglis
Kennedy Institute of Rheumatology Division, Imperial College London, 1 Aspenlea Road, London W6 8LH, UK
Nat Protoc 3:612-8. 2008..The incidence of arthritis varies from 50 to 80% and is milder than the classical collagen-induced arthritis model. This procedure takes approximately 3 months to complete...
ANTI-CD3 therapy expands the numbers of CD4+ and CD8+ Treg cells and induces sustained amelioration of collagen-induced arthritisClare A Notley
Imperial College London, London, UK
Arthritis Rheum 62:171-8. 2010..To assess the therapeutic potential of anti-CD3 monoclonal antibodies (mAb) for rheumatoid arthritis, using collagen-induced arthritis as an animal model...
Collagen-induced arthritis in C57BL/6 mice is associated with a robust and sustained T-cell response to type II collagenJulia J Inglis
Kennedy Institute of Rheumatology, Imperial College London, 1 Aspenlea Road, London W6 8LH, UK
Arthritis Res Ther 9:R113. 2007....
Indoleamine 2,3 dioxygenase-mediated tryptophan catabolism regulates accumulation of Th1/Th17 cells in the joint in collagen-induced arthritisGabriel Criado
Kennedy Institute of Rheumatology, Imperial College London, London, UK
Arthritis Rheum 60:1342-51. 2009..The aim of this study was to investigate the regulation of collagen-induced arthritis by tryptophan catabolism mediated by IDO...
IL-17 induces hyperalgesia via TNF-dependent neutrophil infiltrationKay E McNamee
Kennedy Institute of Rheumatology, Imperial College London, London W6 8LH, UK
Pain 152:1838-45. 2011..The subsequent production of keratinocyte chemoattractant then triggers neutrophil chemotaxis to the plantar tissue, releasing algesic mediators locally to sensitise the nerve...
The differential contribution of tumour necrosis factor to thermal and mechanical hyperalgesia during chronic inflammationJulia J Inglis
Bone and Joint Research Unit, Barts and the London School of Medicine and Dentistry, John Vane Science Centre, London, UK
Arthritis Res Ther 7:R807-16. 2005..These results highlight the potential for TNF to play a vital role in inflammatory hyperalgesia, both by a direct action on neurons via TNFR1 and by facilitating the accumulation of macrophages in the DRG via a TNFR2-mediated pathway...
Nonsteroidal anti-inflammatory drugs increase TNF production in rheumatoid synovial membrane cultures and whole bloodTheresa H Page
The Kennedy Institute of Rheumatology Division, Faculty of Medicine, Imperial College of Science, Technology and Medicine, London, United Kingdom
J Immunol 185:3694-701. 2010..Thus, by reducing the level of PGE(2), NSAIDs can increase TNF production and may exacerbate the proinflammatory environment both within the rheumatoid arthritis joint and the systemic environment...
Analysing the effect of novel therapies on cytokine expression in experimental arthritisRichard O Williams
Kennedy Institute of Rheumatology Division, Imperial College London, London, UK
Int J Exp Pathol 86:267-78. 2005..In this review, we outline the various techniques used to detect cytokines in experimental arthritis and describe how these techniques have been used to quantify changes in cytokine expression following therapeutic intervention...
In vivo fluorescence imaging of E-selectin: quantitative detection of endothelial activation in a mouse model of arthritisLuke L Gompels
Kennedy Institute of Rheumatology, Imperial College London, London, UK
Arthritis Rheum 63:107-17. 2011..The purpose of this study was to demonstrate E-selectin-targeted fluorescence imaging in vivo in a mouse model of paw edema generated by local injection of TNF? as well as in mice with CIA...
Inhibition of inflammation and hyperalgesia in NK-1 receptor knock-out miceBruce L Kidd
Barts and the London School of Medicine, Queen Mary University of London, London, UK
Neuroreport 14:2189-92. 2003..The results point to a previously unrecognised influence of substance P on peripheral tissue injury and the maintenance of hyperalgesia during more severe or more chronic phases of inflammatory disease...
