Luc Härter

Summary

Affiliation: University Hospital
Country: Switzerland

Publications

  1. ncbi In vitro norepinephrine significantly activates isolated platelets from healthy volunteers and critically ill patients following severe traumatic brain injury
    Christoph Tschuor
    Surgical Intensive Care Medicine, University Hospital Zuerich, Raemistrasse 100, CH 8091 Zuerich, Switzerland
    Crit Care 12:R80. 2008
  2. ncbi Increased expression of toll-like receptor-2 and -4 on leukocytes from patients with sepsis
    Luc Härter
    Division of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    Shock 22:403-9. 2004
  3. ncbi Delayed inhibition of agonist-induced granulocyte-platelet aggregation after low-dose sevoflurane inhalation in humans
    Johannes Wacker
    Institute of Anesthesiology, University Hospital Zurich, University of Zurich, Zurich, Switzerland
    Anesth Analg 106:1749-58. 2008
  4. ncbi Molecular evidence of late preconditioning after sevoflurane inhalation in healthy volunteers
    Eliana Lucchinetti
    Institute of Anesthesiology, University Hospital Zurich, Switzerland
    Anesth Analg 105:629-40. 2007
  5. ncbi Vacuum-assisted closure therapy increases local interleukin-8 and vascular endothelial growth factor levels in traumatic wounds
    Ludwig Labler
    Division of Trauma Surgery, Department of Surgery, University Hospital Zurich, Zurich, Switzerland
    J Trauma 66:749-57. 2009
  6. ncbi Pancreatic stone protein is highly increased during posttraumatic sepsis and activates neutrophil granulocytes
    Marius Keel
    Departments of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    Crit Care Med 37:1642-8. 2009
  7. ncbi Thiopental-induced apoptosis in lymphocytes is independent of CD95 activation
    Marius Keel
    Department of Trauma Surgery, Division of Trauma Surgery, University Hospital Zurich, Switzerland
    Anesthesiology 103:576-84. 2005
  8. ncbi Endotoxin reduces CD95-induced neutrophil apoptosis by cIAP-2-mediated caspase-3 degradation
    Ladislav Mica
    Division of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    J Am Coll Surg 199:595-602. 2004
  9. ncbi Sevoflurane inhalation at sedative concentrations provides endothelial protection against ischemia-reperfusion injury in humans
    Eliana Lucchinetti
    Institute of Anesthesiology, University Hospital Zurich, Switzerland
    Anesthesiology 106:262-8. 2007

Collaborators

Detail Information

Publications11

  1. ncbi In vitro norepinephrine significantly activates isolated platelets from healthy volunteers and critically ill patients following severe traumatic brain injury
    Christoph Tschuor
    Surgical Intensive Care Medicine, University Hospital Zuerich, Raemistrasse 100, CH 8091 Zuerich, Switzerland
    Crit Care 12:R80. 2008
    ..This, in turn, could promote microthrombosis formation and induce additional brain damage...
  2. ncbi Increased expression of toll-like receptor-2 and -4 on leukocytes from patients with sepsis
    Luc Härter
    Division of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    Shock 22:403-9. 2004
    ..Therefore, the endotoxin tolerance seen in patients with sepsis does not depend solely on TLR-2 or TLR-4 expression, and other mechanisms must be involved...
  3. ncbi Delayed inhibition of agonist-induced granulocyte-platelet aggregation after low-dose sevoflurane inhalation in humans
    Johannes Wacker
    Institute of Anesthesiology, University Hospital Zurich, University of Zurich, Zurich, Switzerland
    Anesth Analg 106:1749-58. 2008
    ..Sevoflurane can be used as sedative-analgesic drug with endothelial protective properties. We tested whether low-dose sevoflurane inhalation provides sustained inhibition of detrimental granulocyte-platelet aggregation in humans...
  4. ncbi Molecular evidence of late preconditioning after sevoflurane inhalation in healthy volunteers
    Eliana Lucchinetti
    Institute of Anesthesiology, University Hospital Zurich, Switzerland
    Anesth Analg 105:629-40. 2007
    ..We hypothesized that sevoflurane inhalation would modify the transcriptome in human blood and modulate the expression of adhesion molecules in white blood cells consistent with the occurrence of a late preconditioning phase...
  5. ncbi Vacuum-assisted closure therapy increases local interleukin-8 and vascular endothelial growth factor levels in traumatic wounds
    Ludwig Labler
    Division of Trauma Surgery, Department of Surgery, University Hospital Zurich, Zurich, Switzerland
    J Trauma 66:749-57. 2009
    ..Aim of this study was to determine the impact of VAC therapy versus alternative Epigard application on local inflammation and neovascularization in traumatic soft tissue wounds...
  6. ncbi Pancreatic stone protein is highly increased during posttraumatic sepsis and activates neutrophil granulocytes
    Marius Keel
    Departments of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    Crit Care Med 37:1642-8. 2009
    ..However, after stress (e.g., anesthesia), PSP/reg levels are increased transiently in animals without pancreatic injury. Therefore, we aimed to determine whether PSP/reg is an acute-phase protein after nonpancreatic trauma...
  7. ncbi Thiopental-induced apoptosis in lymphocytes is independent of CD95 activation
    Marius Keel
    Department of Trauma Surgery, Division of Trauma Surgery, University Hospital Zurich, Switzerland
    Anesthesiology 103:576-84. 2005
    ..However, barbiturate-mediated neuroprotection correlates with lymphopenia, which increases the risk of infection. The mechanisms by which barbiturates lead to lymphopenia remain to be determined...
  8. ncbi Endotoxin reduces CD95-induced neutrophil apoptosis by cIAP-2-mediated caspase-3 degradation
    Ladislav Mica
    Division of Trauma Surgery, University Hospital Zurich, Zurich, Switzerland
    J Am Coll Surg 199:595-602. 2004
    ..7 +/- 2.6%) apoptosis and degradation of caspase-3. CONCLUSIONS: Induction of cIAP-2 by endotoxins and accelerated degradation of activated caspase-3 by the proteasome might be responsible for reduced apoptosis in PMN during sepsis...
  9. ncbi Sevoflurane inhalation at sedative concentrations provides endothelial protection against ischemia-reperfusion injury in humans
    Eliana Lucchinetti
    Institute of Anesthesiology, University Hospital Zurich, Switzerland
    Anesthesiology 106:262-8. 2007
    ..The authors tested whether inhalation of sevoflurane at subanesthetic concentrations provides protection against postocclusive endothelial dysfunction induced by ischemia-reperfusion injury of the forearm in humans...