Research Topics
| Adam AmeurSummaryAffiliation: Uppsala University Country: Sweden Publications
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Detail Information
Publications
Differential binding and co-binding pattern of FOXA1 and FOXA3 and their relation to H3K4me3 in HepG2 cells revealed by ChIP-seqMehdi Motallebipour
Department of Genetics and Pathology, Uppsala University, Rudbeck Laboratory, Dag Hammarskjölds väg 20, Uppsala SE 75185, Sweden
Genome Biol 10:R129. 2009..The forkhead box/winged helix family members FOXA1, FOXA2, and FOXA3 are of high importance in development and specification of the hepatic linage and the continued expression of liver-specific genes...
Identification of novel exons and transcribed regions by chimpanzee transcriptome sequencingAnna Wetterbom
Department of Genetics and Pathology, Rudbeck Laboratory, Uppsala University, SE 751 85 Uppsala, Sweden
Genome Biol 11:R78. 2010..We profile the chimpanzee transcriptome by using deep sequencing of cDNA from brain and liver, aiming to quantify expression of known genes and to identify novel transcribed regions...
Global and unbiased detection of splice junctions from RNA-seq dataAdam Ameur
Department of Genetics and Pathology, Rudbeck Laboratory, Uppsala University, Uppsala, Sweden
Genome Biol 11:R34. 2010..Our method also reports genomic rearrangements such as insertions and deletions...
Total RNA sequencing reveals nascent transcription and widespread co-transcriptional splicing in the human brainAdam Ameur
Department of Immunology, Genetics and Pathology, Science for Life Laboratory Uppsala, Rudbeck Laboratory, Uppsala University, Uppsala, Sweden
Nat Struct Mol Biol 18:1435-40. 2011..Our data show that sequencing of total RNA provides unique insight into the transcriptional processes in the cell, with particular importance for normal brain development...
Ultra-deep sequencing of mouse mitochondrial DNA: mutational patterns and their originsAdam Ameur
Department of Immunology, Genetics, and Pathology, SciLifeLab Uppsala, Rudbeck Laboratory, Uppsala University, Uppsala, Sweden
PLoS Genet 7:e1002028. 2011..Our results from studies of mice thus indicate that most somatic mtDNA mutations occur as replication errors during development and do not result from damage accumulation in adult life...
