Erik Sundberg

Summary

Affiliation: Karolinska Institutet
Country: Sweden

Publications

  1. ncbi Systemic TNF blockade does not modulate synovial expression of the pro-inflammatory mediator HMGB1 in rheumatoid arthritis patients--a prospective clinical study
    Erik Sundberg
    Department of Woman and Child Health, Pediatric Rheumatology Research Unit, Karolinska Institutet Karolinska University Hospital, Stockholm, Sweden
    Arthritis Res Ther 10:R33. 2008
  2. ncbi High mobility group box chromosomal protein 1 acts as a proliferation signal for activated T lymphocytes
    Erik Sundberg
    Department of Woman and Child Health, Pediatric Rheumatology Research Unit, Karolinska Institutet Karolinska University Hospital, L 8 04 CMM, S 17176 Stockholm, Sweden
    Immunobiology 214:303-9. 2009
  3. ncbi Morphological characterization of intra-articular HMGB1 expression during the course of collagen-induced arthritis
    Karin Palmblad
    Department of Woman and Child Health, Karolinska Institutet, Astrid Lindgren Children s Hospital, SE 171 76 Stockholm, Sweden
    Arthritis Res Ther 9:R35. 2007

Detail Information

Publications3

  1. ncbi Systemic TNF blockade does not modulate synovial expression of the pro-inflammatory mediator HMGB1 in rheumatoid arthritis patients--a prospective clinical study
    Erik Sundberg
    Department of Woman and Child Health, Pediatric Rheumatology Research Unit, Karolinska Institutet Karolinska University Hospital, Stockholm, Sweden
    Arthritis Res Ther 10:R33. 2008
    ..It was, therefore, decided to investigate whether treatment based on TNF blockade in rheumatoid arthritis (RA) affects the expression of synovial HMGB1...
  2. ncbi High mobility group box chromosomal protein 1 acts as a proliferation signal for activated T lymphocytes
    Erik Sundberg
    Department of Woman and Child Health, Pediatric Rheumatology Research Unit, Karolinska Institutet Karolinska University Hospital, L 8 04 CMM, S 17176 Stockholm, Sweden
    Immunobiology 214:303-9. 2009
    ..0 microg/ml. HMGB1 had no effect on proliferation in the absence of anti-CD3 stimulation or during T-cell activation obtained using high doses of anti-CD3 mAb. Our results demonstrate a direct HMGB1-mediated effect in adaptive immunity...
  3. ncbi Morphological characterization of intra-articular HMGB1 expression during the course of collagen-induced arthritis
    Karin Palmblad
    Department of Woman and Child Health, Karolinska Institutet, Astrid Lindgren Children s Hospital, SE 171 76 Stockholm, Sweden
    Arthritis Res Ther 9:R35. 2007
    ..In conclusion, these new findings implicate a role for HMGB1 in both inducing and perpetuating inflammatory events of significant importance in the destructive processes in chronic arthritis...