Research Topics
Genomes and GenesSpecies | T JinSummaryCountry: Sweden Publications
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Publications
Intraarticular release and accumulation of defensins and bactericidal/permeability-increasing protein in patients with rheumatoid arthritisMaria I Bokarewa
Department of Rheumatology, Sahlgrenska University Hospital, Goteborg, Sweden
J Rheumatol 30:1719-24. 2003..We assessed concentrations of BPI and a-defensins in synovial fluid (SF) and matching blood samples of patients with rheumatoid arthritis (RA)...- Staphylococcus aureus: StaphylokinaseMaria I Bokarewa
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Guldhedsgatan 10, SE 41346 Göteborg, Sweden
Int J Biochem Cell Biol 38:504-9. 2006..Our observations indicate that staphylokinase favours symbiosis of staphylococci with the host that makes it an important colonization factor... - Decreased serum levels of TGF-?1 are associated with renal damage in female patients with systemic lupus erythematosusT Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska Academy at University of Gothenburg, Goteborg, Sweden
Lupus 21:310-8. 2012..Decreased levels of TGF-?1 might associate to disease susceptibility, activity and organ damage in SLE. However, the correlation between TGF-?1 levels and severity of renal damage in SLE has not been examined... - Impact of short-term therapies with biologics on prothrombotic biomarkers in rheumatoid arthritisT Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, S 413 46 Goteborg, Sweden
Clin Exp Rheumatol 27:491-4. 2009..There is no imformation about the influence of biological drugs, e.g. anti-CD20 and tumor necrosis factor (TNF) antibodies on these prothrombotic molecules... - Staphylokinase reduces plasmin formation by endogenous plasminogen activatorsTao Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Goteborg, Sweden
Eur J Haematol 81:8-17. 2008..In conclusion, SAK reduces tPA/uPA-mediated Plg activation by means of SAK.Plg complex formation, consequently downregulating tPA/uPA-induced fibrinolysis... - The role of urokinase in innate immunity against Staphylococcus aureusTao Jin
Department of Rheumatology and Inflammation Research, University of Goteborg, 41346 Göteborg, Sweden
Microbes Infect 7:1170-5. 2005..These findings indicate novel functions of HMW uPA in the host defense against staphylococcal infections... - Urokinase-type plasminogen activator, an endogenous antibioticTao Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Goteborg, Sweden
J Infect Dis 192:429-37. 2005..The decreased active uPA levels in infected organs might be due to the dramatically increased PAI-1 production during S. aureus infection... - Current status of pathogenetic mechanisms in staphylococcal arthritisAndrej Tarkowski
Department of Rheumatology and Inflammation Research, University of Goteborg, Guldhedsgatan 10, 413 46 Goteborg, Sweden
FEMS Microbiol Lett 217:125-32. 2002..This review deals with the intricate host-bacterium interactions that occur during experimental septic arthritis, and outlines potential preventive and treatment modalities... - Staphylococcus aureus resists human defensins by production of staphylokinase, a novel bacterial evasion mechanismTao Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Guldhedsgatan 10, , Sweden
J Immunol 172:1169-76. 2004..Staphylokinase production may therefore be responsible in vivo for defensin resistance during S. aureus infections... - Fatal outcome of bacteraemic patients caused by infection with staphylokinase-deficient Staphylococcus aureus strainsTao Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Guldhedsgatan 10, , Sweden
J Med Microbiol 52:919-23. 2003..aureus bacteraemia. The high frequency of SAK production in nasal isolates and in cases with uncomplicated bacteraemia suggests that SAK may be one of the adaptive mechanisms of S. aureus symbiosis with the host... - Urokinase, a constitutive component of the inflamed synovial fluid, induces arthritisTao Jin
Department of Rheumatology and Inflammation Research, Sahlgrenska University Hospital, Goteborg, Sweden
Arthritis Res Ther 5:R9-R17. 2003..Accumulation of uPA locally in the joint cavity is a typical finding in erosive RA. uPA exerts potent arthritogenic properties and thus may be viewed as one of the essential mediators of joint inflammation... - Single-stranded polyinosinic acid oligonucleotides trigger leukocyte production of proteins belonging to fibrinolytic and coagulation cascadesFariba Zare
Department of Rheumatology and Inflammation Research, University of Goteborg, Sweden
J Leukoc Biol 84:741-7. 2008..As fibrinolytic and coagulation cascades are important components of inflammatory homeostasis, these findings might have importance for development of new, targeted therapies... - Periodontal regeneration using novel glycidyl methacrylated dextran (Dex-GMA)/gelatin scaffolds containing microspheres loaded with bone morphogenetic proteinsFa Ming Chen
Department of Periodontology and Oral Medicine, School of Stomatology, Fourth Military Medical University, Xi an 710032, Shaanxi, China
J Control Release 121:81-90. 2007.... - [Protective effects of ginaton against ischemia-reperfusion injury on the autograft after lung autotransplantation: experiment with rabbits]He Yun Xu
Department of Thoracic and Cardiovascular Surgery, First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou 310003, China
Zhonghua Yi Xue Za Zhi 86:3211-4. 2006..To investigate the effects of ginaton against ischemia-reperfusion injury on the autograft after lung autotransplantation... - Novel glycidyl methacrylated dextran (Dex-GMA)/gelatin hydrogel scaffolds containing microspheres loaded with bone morphogenetic proteins: formulation and characteristicsFa Ming Chen
Department of Periodontology and Oral Medicine, School of Stomatology, Fourth Military Medical University, Xi an 710032, Shaanxi, China
J Control Release 118:65-77. 2007.... - Role of peritubular capillary loss and hypoxia in progressive tubulointerstitial fibrosis in a rat model of aristolochic acid nephropathyDong Sun
Department of Nephrology, First Affiliated Hospital of China Medical University, Shenyang City, Liaoning Province, PR China
Am J Nephrol 26:363-71. 2006..CONCLUSION: Following the reduction of PTCs density and up-regulation of HIF-1alpha, the tubulointerstitial fibrosis area increased. Ischemia and hypoxia are the important causes of severe tubulointerstitial fibrosis in AAN rats... - Effects of prostaglandin E1 on the progression of aristolochic acid nephropathyDong Sun
Department of Nephropathy, The First Affiliated Hospital, China Medical University, Shenyang 110001
Chin Med Sci J 20:67-9. 2005..To investigate the effects of prostaglandin E1 (PGE1) on the progression of aristolochic acid nephropathy (AAN)...