Research Topics
Species | Frederick J RaalSummaryAffiliation: University of the Witwatersrand Country: South Africa Publications
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Detail Information
Publications
A longitudinal study of stavudine-associated toxicities in a large cohort of South African HIV infected subjectsColin N Menezes
Infectious Diseases Unit, Department of Medicine, Helen Joseph Hospital, University of the Witwatersrand, Johannesburg, South Africa
BMC Infect Dis 11:244. 2011..This has brought new challenges which include the effects of stavudine-related toxicities...
Pathogenesis and management of the dyslipidemia of the metabolic syndromeFrederick J Raal
Division of Endocrinology and Metabolism, Department of Medicine, Johannesburg Hospital, Johannesburg, South Africa
Metab Syndr Relat Disord 7:83-8. 2009..However, in very high-risk patients, such as those with established cardiovascular disease, an LDL-C goal of below 70 mg/dL is recommended...
Mipomersen, an apolipoprotein B synthesis inhibitor, for lowering of LDL cholesterol concentrations in patients with homozygous familial hypercholesterolaemia: a randomised, double-blind, placebo-controlled trialFrederick J Raal
Department of Medicine, University of the Witwatersrand, Johannesburg, South Africa
Lancet 375:998-1006. 2010..This study investigated whether an antisense inhibitor of apolipoprotein B synthesis, mipomersen, is effective and safe as an adjunctive agent to lower LDL cholesterol concentrations in patients with this disease...
CEPHEUS SA: a South African survey on the undertreatment of hypercholesterolaemiaF Raal
Department of Medicine, Charlotte Maxeke Johannesburg Academic Hospital, Johannesburg, South Africa
Cardiovasc J Afr 22:234-40. 2011....
Reduction in mortality in subjects with homozygous familial hypercholesterolemia associated with advances in lipid-lowering therapyFrederick J Raal
Department of Medicine, University of the Witwatersrand Medical School, 7 York Rd, Parktown 2193, Johannesburg, South Africa
Circulation 124:2202-7. 2011..9±3.9 to 11.7±3.4 mmol/L; P<0.0001) with lipid-lowering therapy. Conclusions- Lipid-lowering therapy is associated with delayed cardiovascular events and prolonged survival in patients with homozygous familial hypercholesterolemia...
High-dose atorvastatin therapy is required for significant improvement of endothelial function in heterozygous familial hypercholesterolaemic patientsSusan L Brown
Carbohydrate and Lipid Metabolism Research Unit, Department of Medicine, University of the Witwatersrand, Johannesburg
Cardiovasc J S Afr 15:70-5. 2004..There was no correlation between the changes in FMD and LDL-C, suggesting either a LDLC-independent effect on ED, or that a marked reduction in LDL-C is required to normalise ED in FH...
Leptin, adiponectin, and high-sensitivity C-reactive protein in relation to the metabolic syndrome in urban South African blacks with and without coronary artery diseaseLucas Ntyintyane
Carbohydrate and Lipid Metabolism Research Unit, Department of Medicine, University of the Witwatersrand, Johannesburg, South Africa
Metab Syndr Relat Disord 7:243-8. 2009..Inflammation is frequently associated with metabolic syndrome and CAD. This study evaluated markers of inflammation in black CAD patients, some of whom had metabolic syndrome...
Avasimibe, an ACAT inhibitor, enhances the lipid lowering effect of atorvastatin in subjects with homozygous familial hypercholesterolemiaFrederick J Raal
Carbohydrate and Lipid Metabolism Research Unit, University of the Witwatersrand, Johannesburg Hospital, Johannesburg, South Africa
Atherosclerosis 171:273-9. 2003..Avasimibe may modestly enhance the lipid-reducing effect of atorvastatin by further inhibiting the production of intracellular cholesterol through mechanisms that appear to be compatible in this population...
Autosomal recessive hypercholesterolaemia: discrimination of ARH protein and LDLR function in the homozygous FH phenotypeAron B Abera
Department of Medicine, University of Cape Town, UCT NHLS, Cape Town, South Africa
Clin Chim Acta 378:33-7. 2007..An effective strategy is thus required to identify the appropriate mechanism for the disorder...
