Research Topics
Species | Beata PeplonskaSummaryAffiliation: University of Lodz Country: Poland Publications
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Detail Information
Publications
Estimating age-specific breast cancer risks: a descriptive tool to identify age interactionsWilliam F Anderson
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institute of Health, Executive Plaza South 8070, 6120 Executive Plaza Blvd, Rockville, MD 20892 7242, USA
Cancer Causes Control 18:439-47. 2007..Clarifying age-specific female breast cancer risks and interactions may provide important etiologic clues...- Night shift work characteristics and 6-sulfatoxymelatonin (MT6s) in rotating night shift nurses and midwivesBeata Peplonska
Department of Environmental Epidemiology, Nofer Institute of Occupational Medicine, 8 Teresy St, 91 348 Lodz, Poland
Occup Environ Med 69:339-46. 2012..9 vs 47.4 ng/mg Cr, respectively). Total night shift work history was not associated with MT6s. Conclusions The results of this study indicate that working eight or more night shifts per month may disrupt the synthesis of melatonin... - Genetic variation in PRL and PRLR, and relationships with serum prolactin levels and breast cancer risk: results from a population-based case-control study in PolandSarah J Nyante
Division of Cancer Epidemiology and Genetics, National Cancer Institute, 6120 Executive Boulevard, Rockville, MD 20852, USA
Breast Cancer Res 13:R42. 2011..Thus, we examined the relationship between single nucleotide polymorphisms (SNPs) in PRL and PRLR, serum prolactin levels and breast cancer risk in a population-based case-control study... - Ovarian cancer risk and common variation in the sex hormone-binding globulin gene: a population-based case-control studyMontserrat Garcia-Closas
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Rockville, MD 20952 7234, USA
BMC Cancer 7:60. 2007..We evaluated whether common genetic variation in SHBG and its 3' neighbor ATP1B2, in linkage disequilibrium, is associated with the risk of epithelial ovarian cancer... - Occupational exposure to organic solvents and breast cancer in womenBeata Peplonska
Department of Environmental Epidemiology, Nofer Institute of Occupational Medicine, Teresy 8 St, Lodz 91 348, Poland
Occup Environ Med 67:722-9. 2010..Although studies in rodents suggest possible associations between exposure to organic solvents and breast cancer, the evidence in humans is limited... - Adulthood lifetime physical activity and breast cancerBeata Peplonska
Department of Occupational and Environmental Epidemiology, Nofer Institute of Occupational Medicine, Lodz, Poland
Epidemiology 19:226-36. 2008..However, the effects of moderate activities, timing of activities, and intervening effects of other risk factors remain less clear... - [Occupational diseases among personnel of Polish hospitals, 2001]Beata Pepłońska
Zakładu Epidemiologii Srodowiskowej, Instytutu Medycyny Pracy im prof J Nofera w Łodzi
Med Pr 53:369-74. 2002..The decrease in the number of the registered occupational diseases seen in the hospital employees was mostly due to the effective anti HBV prevention programs carried out in Poland among health care personnel since 1989... - Occupation and breast cancer risk in Polish women: a population-based case-control studyBeata Peplonska
Department of Occupational and Environmental Epidemiology, Nofer Institute of Occupational Medicine, Łódź, Poland
Am J Ind Med 50:97-111. 2007..The etiology of breast cancer is not well understood and the role of occupational exposures in breast carcinogenesis is still uncertain... - Occupational diseases in Poland, 2001Beata Pepłońska
Department of Occupational and Environmental Epidemiology, Nofer Institute of Occupational Medicine, Lodz, Poland
Int J Occup Med Environ Health 15:337-45. 2002.... - [Epidemiologic analysis of infectious diseases identified as occupational diseases in Poland in the years 1998-2002]Beata Pepłońska
Zakładu Epidemiologii Srodowiskowej, Instytutu Medycyny Pracy im prof J Nofera w Łodzi
Med Pr 54:521-8. 2003..During the last thirty years overall as many as 41,000 cases of occupational contagious and invasive diseases were identified... - Earlier onset of Alzheimer's disease: risk polymorphisms within PRNP, PRND, CYP46, and APOE genesEwa Golanska
Department of Molecular Pathology and Neuropathology, Medical University of Lodz, Lodz, Poland
J Alzheimers Dis 17:359-68. 2009..Moreover, there may be multiple sufficient risk sets for AD. Looking at multiple genes together rather than analyzing them individually, may improve identification of risk alleles... - Genetic polymorphisms in the one-carbon metabolism pathway and breast cancer risk: a population-based case-control study and meta-analysesJolanta Lissowska
Department of Cancer Epidemiology and Prevention, Cancer Center and M Sklodowska Curie Institute of Oncology, Warsaw, Poland
Int J Cancer 120:2696-703. 2007..In conclusion, this report does not support a substantial overall association between the evaluated polymorphisms in the one-carbon metabolism pathway and breast cancer risk... - Common genetic variation in TP53 and its flanking genes, WDR79 and ATP1B2, and susceptibility to breast cancerMontserrat Garcia-Closas
Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, Maryland, USA
Int J Cancer 121:2532-8. 2007..However, data suggested that common variation in TP53 or WDR79 could be associated with ER negative breast cancers... - Genome-wide association study identifies novel breast cancer susceptibility lociDouglas F Easton
CR UK Genetic Epidemiology Unit, Department of Public Health and Primary Care, University of Cambridge, Cambridge CB1 8RN, UK
Nature 447:1087-93. 2007..05 level compared with an estimated 1,343 that would be expected by chance, indicating that many additional common susceptibility alleles may be identifiable by this approach... - Heterogeneity of breast cancer associations with five susceptibility loci by clinical and pathological characteristicsMontserrat Garcia-Closas
Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, Marylan, United States of America
PLoS Genet 4:e1000054. 2008..Understanding the etiologic heterogeneity of breast cancer may ultimately result in improvements in prevention, early detection, and treatment... - Polymorphisms in DNA double-strand break repair genes and risk of breast cancer: two population-based studies in USA and Poland, and meta-analysesMontserrat Garcia-Closas
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, 6120 Executive Boulevard, Room 7076, Rockville, MD 20952 7234, USA
Hum Genet 119:376-88. 2006..Evaluation of potential underlying gene-gene interactions or associations in population subgroups will require even larger sample sizes... - Common genetic variation in GATA-binding protein 3 and differential susceptibility to breast cancer by estrogen receptor alpha tumor statusMontserrat Garcia-Closas
Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, 6120 Executive Boulevard, Room 7076, MSC 7234, Rockville, MD 20852 7234, USA
Cancer Epidemiol Biomarkers Prev 16:2269-75. 2007..These differential associations are consistent with markedly different levels of GATA3 protein by ER status. Additional epidemiologic studies are needed to clarify these intriguing relationships... - Genetic polymorphisms in base-excision repair pathway genes and risk of breast cancerYawei Zhang
Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, Department of Health and Human Services, Rockville, Maryland, USA
Cancer Epidemiol Biomarkers Prev 15:353-8. 2006..6; 95% CI, 1.1-2.4; P = 0.02). In conclusion, our results do not support that the polymorphisms evaluated in six BER pathway genes play a major role in breast carcinogenesis, particularly in Caucasian populations... - Genetic variation in tumor necrosis factor and lymphotoxin-alpha (TNF-LTA) and breast cancer riskMia M Gaudet
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, Department of Health and Human Services, Bethesda, MD, USA
Hum Genet 121:483-90. 2007..Additional studies are required to replicate these findings and to determine whether rs361525 is a causative SNP or is a marker of a causative SNP... - Comprehensive assessment of genetic variation of catechol-O-methyltransferase and breast cancer riskMia M Gaudet
Division of Cancer Epidemiology, National Cancer Institute, 6120 Executive Boulevard, EPS 7055, Rockville, MD 20852, USA
Cancer Res 66:9781-5. 2006..The suggestion of increased risk associated with a haplotype in the 3' UTR of COMT needs to be confirmed in independent study populations... - Genetic variation of Cytochrome P450 1B1 (CYP1B1) and risk of breast cancer among Polish womenMia M Gaudet
Division of Cancer Epidemiology and Genetics, National Cancer Institute, Department of Health and Human Services, Bethesda, Maryland 20852, USA
Pharmacogenet Genomics 16:547-53. 2006..The present study provides strong evidence against the existence of a substantial overall association between common genetic variation in CYP1B1 and breast cancer risk... - The ATM missense mutation p.Ser49Cys (c.146C>G) and the risk of breast cancerDenise L Stredrick
Laboratory of Population Genetics, National Cancer Institute, National Institutes of Health, Bethesda, Maryland 20892 5060, USA
Hum Mutat 27:538-44. 2006..S49C, may be breast cancer susceptibility alleles. Because of their low frequency, even larger sample sizes are required to more firmly establish these associations... - Genetic variation in CYP17 and endometrial cancer riskMia M Gaudet
National Cancer Institute, National Institutes of Health, Bethesda, MD, USA
Hum Genet 123:155-62. 2008..Further evaluation in consortia is necessary to confirm potential weak associations between common variation in CYP17 and endometrial cancer risk and to address the concern of publication bias... - Simultaneous analysis of five genetic risk factors in Polish patients with Alzheimer's diseaseMaria Styczynska
Department of Neurodegenerative Disorders, Medical Research Centre, Polish Academy of Sciences, 5 Pawinskiego Str, 02 106 Warsaw, Poland
Neurosci Lett 344:99-102. 2003..Our results suggest that the APOE epsilon 4 allele is the only known genetic risk factor for late-onset, sporadic AD... - Hormonal markers in breast cancer: coexpression, relationship with pathologic characteristics, and risk factor associations in a population-based studyXiaohong R Yang
Genetic Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, NIH, Bethesda, MD 20892 7236, USA
Cancer Res 67:10608-17. 2007.... - Variation in breast cancer hormone receptor and HER2 levels by etiologic factors: a population-based analysisMark E Sherman
Division of Cancer Epidemiology and Genetics, National Cancer Institute, National Institutes of Health, DHHS, Bethesda, MD, USA
Int J Cancer 121:1079-85. 2007..07) for low PR and HER2 expression vs. OR = 1.78 (95% CI = 1.25-2.55) for high expression (p-heterogeneity = 0.001). PR and HER2 levels in breast cancer vary by BMI, suggesting a heterogeneous etiology for tumors related to these markers... - CYP46: a risk factor for Alzheimer's disease or a coincidence?Ewa Golanska
Department of Molecular Pathology and Neuropathology, Medical University of Lodz, Poland
Neurosci Lett 383:105-8. 2005..However, considering the extreme divergence of results obtained by different authors, a clear connection between the CYP46 gene and AD is questionable... - Tagging single nucleotide polymorphisms in cell cycle control genes and susceptibility to invasive epithelial ovarian cancerSimon A Gayther
Translational Research Laboratories, University College London, London, United Kingdom
Cancer Res 67:3027-35. 2007..This study highlights the need for multicenter collaborations for genetic association studies... - Differences in risk factors for breast cancer molecular subtypes in a population-based studyXiaohong R Yang
Genetic Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Room 7014, 6120 Executive Boulevard, Bethesda, MD 20892 7236, USA
Cancer Epidemiol Biomarkers Prev 16:439-43. 2007..Results from this study have shown that breast cancer risk factors may vary by molecular subtypes identified in expression studies, suggesting etiologic, in addition to clinical, heterogeneity of breast cancer... - A common coding variant in CASP8 is associated with breast cancer riskAngela Cox
Sheffield University Medical School, Sheffield S10 2RX, UK
Nat Genet 39:352-8. 2007..02-1.13) and 1.16 (95% c.i.: 1.08-1.25), respectively; P(trend) = 2.8 x 10(-5)). These results demonstrate that common breast cancer susceptibility alleles with small effects on risk can be identified, given sufficiently powerful studies... - Tobacco smoking, NAT2 acetylation genotype and breast cancer riskJolanta Lissowska
Department of Cancer Epidemiology and Prevention, Cancer Center and M Sklodowska Curie Institute of Oncology, Warsaw, Poland
Int J Cancer 119:1961-9. 2006..In conclusion, our data indicate that passive smoking is not associated with breast cancer risk; however, active smoking might be associated with an increased risk for early onset breast cancers... - Field comparison of inhalable aerosol samplers applied in the european rubber manufacturing industryFrank de Vocht
Environmental and Occupational Health Division, Institute for Risk Assessment Sciences, Utrecht University, PO Box 80 176, 3508 TD, Utrecht, The Netherlands
Int Arch Occup Environ Health 79:621-9. 2006..To compare measurements from different studies and countries, correction factors should be applied to all but the IOM sampler, which was the only sampler that performed similar to the CALTOOL sampler...