Mohammed A Junaid

Summary

Publications

  1. ncbi Folic acid supplementation dysregulates gene expression in lymphoblastoid cells--implications in nutrition
    Mohammed A Junaid
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, NY 10314, USA
    Biochem Biophys Res Commun 412:688-92. 2011
  2. ncbi Glyoxalase I polymorphism rs2736654 causing the Ala111Glu substitution modulates enzyme activity--implications for autism
    Madhabi Barua
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York, NY 10314, USA
    Autism Res 4:262-70. 2011
  3. ncbi A critical tryptophan and Ca2+ in activation and catalysis of TPPI, the enzyme deficient in classic late-infantile neuronal ceroid lipofuscinosis
    Salomon Kuizon
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York, United States of America
    PLoS ONE 5:e11929. 2010
  4. ncbi Differential expression of small heat shock protein 27 (Hsp27) in Ataxia telangiectasia brains
    Wenqiang Chen
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, NY 10314, USA
    Neurochem Res 34:1658-67. 2009

Collaborators

Detail Information

Publications4

  1. ncbi Folic acid supplementation dysregulates gene expression in lymphoblastoid cells--implications in nutrition
    Mohammed A Junaid
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, NY 10314, USA
    Biochem Biophys Res Commun 412:688-92. 2011
    ..Aberrant expression of genes during early brain development may have an impact on behavioural characteristics...
  2. ncbi Glyoxalase I polymorphism rs2736654 causing the Ala111Glu substitution modulates enzyme activity--implications for autism
    Madhabi Barua
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York, NY 10314, USA
    Autism Res 4:262-70. 2011
    ..We suggest that accumulation of MG results in the formation of AGEs, which induce expression of the RAGE that during crucial neuronal development may be a factor in the pathology of autism...
  3. ncbi A critical tryptophan and Ca2+ in activation and catalysis of TPPI, the enzyme deficient in classic late-infantile neuronal ceroid lipofuscinosis
    Salomon Kuizon
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, New York, United States of America
    PLoS ONE 5:e11929. 2010
    ..Recent X-ray crystallographic studies have provided insights into the structural/functional aspects of TPPI catalysis, and indicated presence of an octahedrally coordinated Ca(2+)...
  4. ncbi Differential expression of small heat shock protein 27 (Hsp27) in Ataxia telangiectasia brains
    Wenqiang Chen
    Department of Developmental Biochemistry, New York State Institute for Basic Research in Developmental Disabilities, Staten Island, NY 10314, USA
    Neurochem Res 34:1658-67. 2009
    ..We propose that neurons in the frontal lobe are protected by the expression of Hsp27, which scavenges the oxidative stress molecules formed consequent to the primary loss of ATM function...