Research Topics
Species | Tetsuya OginoSummaryAffiliation: Okayama University Country: Japan Publications
| Collaborators
|
Detail Information
Publications
Serum-dependent export of protoporphyrin IX by ATP-binding cassette transporter G2 in T24 cellsTetsuya Ogino
Department of Pathology and Experimental Medicine, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikata, Okayama 700 8558, Japan
Mol Cell Biochem 358:297-307. 2011..Inhibition of ABCG2 transporter system could be a new target for the improvement of photodynamic therapy...- Activation of c-Jun N-terminal kinase is essential for oxidative stress-induced Jurkat cell apoptosis by monochloramineTetsuya Ogino
Pathology and Experimental Medicine, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikata, Okayama 700 8558, Japan
Leuk Res 33:151-8. 2009..In contrast, Ca(2+) chelation by EGTA+acetoxymethyl-EGTA had no effects on apoptosis. Our results indicated that JNK activation contributed most importantly to the NH(2)Cl-induced apoptosis... - Oxidative modification of IkappaB by monochloramine inhibits tumor necrosis factor alpha-induced NF-kappaB activationTetsuya Ogino
Department of Pathological Research, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikata, Japan
Biochim Biophys Acta 1746:135-42. 2005..These results indicated that NH(2)Cl inhibited TNFalpha-induced NF-kappaB activation through the oxidation of IkappaB, and that NH(2)Cl is physiologically more relevant than Tau-NHCl in modifying NF-kappaB-mediated cellular responses... - Physiological oxidants induce apoptosis and cell cycle arrest in a multidrug-resistant natural killer cell line, NK-YSTin Aung Than
Department of Pathological Research, Okayama University Graduate School of Medicine and Dentistry, 2-5-1 Shikata, Okayama 700-8558, Japan
Leuk Lymphoma 44:2109-16. 2003..These observations suggest that NH2Cl may induce apoptotic cell death and growth arrest in multidrug-resistant NK cell tumors... - Mechanism of cell death by 5-aminolevulinic acid-based photodynamic action and its enhancement by ferrochelatase inhibitors in human histiocytic lymphoma cell line U937Takashi Amo
Department of Cytology and Histology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama, Japan
Cell Biochem Funct 27:503-15. 2009..These results indicated that ALA-based-PDA induced apoptotic cell death through a mitochondrial pathway and that ferrochelatase inhibitors might enhanced the effect of PDT for tumors even at low concentrations of ALA... - Cell cycle arrest by monochloramine through the oxidation of retinoblastoma proteinMutsumi Hosako
Department of Pathological Research, Okayama University Graduate School of Medicine and Dentistry, Shikata, Okayama, Japan
Free Radic Biol Med 36:112-22. 2004..As protein methionine oxidation has been reported in inflammation and aging, cell cycle modulation by pRB oxidation may occur in various pathological conditions... - Regulation of 5-aminolevulinic acid-dependent protoporphyrin IX accumulations in human histiocytic lymphoma U937 cellsYuya Okimura
Department of Cytology and Histology, Okayama University Graduate School, Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikatacho, Okayama, Okayama 700 8558, Japan
Physiol Chem Phys Med NMR 39:69-82. 2007..These results suggest that ALA-enhanced accumulation of PpIX in U937 cells was regulated by cellular uptake and conversion of ALA to PpIX and by degradation of Heme... - Alpha-tocopheryl succinate induces rapid and reversible phosphatidylserine externalization in histiocytic lymphoma through the caspase-independent pathwayHirofumi Fujita
Department of Cytology and Histology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikatacho, Okayama, 700 8558, Japan
Mol Cell Biochem 333:137-49. 2010..These results suggested that the mechanism of TOS- and CS-induced PS externalization in the absence of FBS was different from it occurring during typical apoptosis... - Taurine chloramine: a possible oxidant reservoirTetsuya Ogino
Pathology and Experimental Medicine, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama, Japan
Adv Exp Med Biol 643:451-61. 2009..These results indicate that Tau-NHCl can serve as an oxidant reservoir, exhibiting either delayed oxidant effects or acting as an oxidant at a distant site... - Oxidative stress enhances granulocytic differentiation in HL 60 cells, an acute promyelocytic leukemia cell lineTetsuya Ogino
Pathology and Experimental Medicine, Okayama University, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikata, Okayama 700 8558, Japan
Free Radic Res 44:1328-37. 2010..These observations suggested that oxidative stress enhanced granulocytic differentiation of HL 60 cells and that leukaemic cell differentiation was affected by cellular redox status... - Monochloramine inhibits the expression of E-selectin and intercellular adhesion molecule-1 induced by TNF-alpha through the suppression of NF-kappaB activation in human endothelial cellsMasako Omori
Department of Pathological Research, Faculty of Medicine, Okayama University Graduate School of Medicine and Dentistry, Japan
Free Radic Res 36:845-52. 2002..We speculate that neutrophil-derived chloramines may have a regulatory role in the recruitment of leukocytes... - Monochloramine inhibits ultraviolet B-induced p53 activation and DNA repair response in human fibroblastsKazuhisa Hiramatsu
Department of Pathological Research, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2-5-1 Shikata, Okayama 700-8558, Japan
Biochim Biophys Acta 1763:188-96. 2006..NH2Cl may facilitate carcinogenesis through the inhibition of NER that repairs DNA damages from various carcinogens... - Alpha-lipoic acid suppresses 6-hydroxydopamine-induced ROS generation and apoptosis through the stimulation of glutathione synthesis but not by the expression of heme oxygenase-1Hirofumi Fujita
Department of Cytology and Histology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, 2 5 1 Shikatacho, Okayama 700 8558, Japan
Brain Res 1206:1-12. 2008..These results suggested that LA suppressed the 6-OHDA induced-apoptosis by the increase in cellular glutathione through stimulation of the GSH synthesis system but not by the expression of HO-1... - Effect of risedronate on osteoblast differentiation, expression of receptor activator of NF-?B ligand and apoptosis in mesenchymal stem cellsHirofumi Fujita
Department of Cytology and Histology, Okayama University Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Japan
Basic Clin Pharmacol Toxicol 109:78-84. 2011.... - Forced expression of suppressor of cytokine signaling 3 in T cells protects the development of concanavalin A-induced hepatitis in miceSoichiro Fushimi
Department of Pathology and Experimental Medicine, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, 2 5 1 Shikata, Okayama 700 8558, Japan
Clin Immunol 133:437-46. 2009..These results suggest that forced expression of SOCS3 in T cells prevents ConA-induced liver injury by inhibiting several phases of Th1 responses... - Cell-permeable cAMP analog suppresses 6-hydroxydopamine-induced apoptosis in PC12 cells through the activation of the Akt pathwayHirofumi Fujita
Institute of Medical Science, Kurashiki Medical Center, Kurashiki 710 8522
Brain Res 1113:10-23. 2006..It is likely that pCPT-cAMP prevented the 6-OHDA-induced apoptosis via activation of the PI3-kinase/Akt pathway without any effect on superoxide generation or mitochondrial membrane depolarization... - Immunohistochemical staining of liver grafts with a monoclonal antibody against HCV-Envelope 2 for recurrent hepatitis C after living donor liver transplantationHiroshi Sadamori
Department of Gastroenterological Surgery, Okayama University Graduate School of Medicine and Dentistry, 2 5 1 Shikata, Okayama 700 8558, Japan
J Gastroenterol Hepatol 24:574-80. 2009..We evaluated the expression of hepatitis C virus (HCV) antigen on liver grafts by immunohistochemical staining (IHS) using IG222 monoclonal antibody (mAb) against HCV-envelope 2 (E2)... - Angiomyofibroblastoma of the vulva: a large pedunculated mass formationMasako Omori
Department of Pathology, Okayama University Hospital, Okayama 700 8558, Japan
Acta Med Okayama 60:237-42. 2006..These histological findings are consistent with angiomyofibroblastoma and support the hypothesis that angiomyofibroblastoma originates from perivascular stem cells with a capacity for myofibroblastic and fatty differentiation... - Ex vivo adenoviral gene transfer of constitutively activated STAT3 reduces post-transplant liver injury and promotes regeneration in a 20% rat partial liver transplant modelKamrul A S M Huda
Department of Gastroenterological Surgery, Transplant, and Surgical Oncology, Okayama University Graduate School of Medicine and Dentistry, Shikata, Okayama, Japan
Transpl Int 19:415-23. 2006..Thus, the activation of STAT3 in the liver graft may be a potentially effective clinical strategy for improving the outcome of small-for-size liver transplantation... - Mechanism of impaired regeneration of fatty liver in mouse partial hepatectomy modelHiroshi Murata
Department of Gastroenterological Surgery, Transplant and Surgical Oncology, Okayama University Graduate School of Medicine and Dentistry, Shikata, Okayama, Japan
J Gastroenterol Hepatol 22:2173-80. 2007..However, the mechanism of an impaired regeneration is still not well understood. The aim of this study was to analyze the mechanism of impaired regeneration of steatotic liver after partial hepatectomy (PH)... - Stat3 protects against Fas-induced liver injury by redox-dependent and -independent mechanismsSanae Haga
Department of Innovative Surgery, National Research Institute for Child Health and Development, Tokyo, Japan
J Clin Invest 112:989-98. 2003..These findings indicate that Stat3 activation protects against Fas-mediated liver injury by inhibiting caspase activities in redox-dependent and -independent mechanisms... - Mechanism and characteristics of stimuli-dependent ROS generation in undifferentiated HL-60 cellsShikibu Muranaka
Institute of Medical Science, Kurashiki Medical Center, Kurashiki, Japan
Antioxid Redox Signal 7:1367-76. 2005..These results suggested that the mechanism of ROS generation in undifferentiated HL-60 cells is the same as that in primed neutrophils... - Compensatory recovery of liver mass by Akt-mediated hepatocellular hypertrophy in liver-specific STAT3-deficient miceSanae Haga
Department of Surgery, Hokkaido University Graduate School of Medicine, Faculty of Medicine, Sapporo, Japan
J Hepatol 43:799-807. 2005..The present study was designed to study the roles of proliferation and hypertrophy of hepatocytes in liver regeneration following hepatectomy in liver-specific STAT3-knockout (LS3-KO) mice lacking mitogenic activity...