Research Topics
Genomes and Genes
Species | Jun ichi SatohSummaryAffiliation: Meiji Pharmaceutical University Country: Japan Publications
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Detail Information
Publications
Phosphorylated Syk expression is enhanced in Nasu-Hakola disease brainsJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University Department of Clinical Neuropathology, Tokyo, Japan
Neuropathology 32:149-57. 2012..These observations indicate that neuronal expression of pSyk was greatly enhanced in the cerebral cortex and the hippocampus of NHD brains, possibly via non-TREM2/DAP12 signaling pathways involved in Syk activation...
Gene expression profile of THP-1 monocytes following knockdown of DAP12, a causative gene for Nasu-Hakola diseaseJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo, Japan
Cell Mol Neurobiol 32:337-43. 2012..These results suggest that a molecular defect of DAP12 in human monocytes deregulates the gene network pivotal for maintenance of myeloid cell function in NHD...
MicroRNAs and their therapeutic potential for human diseases: aberrant microRNA expression in Alzheimer's disease brainsJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Japan
J Pharmacol Sci 114:269-75. 2010..This review will briefly summarize recent studies that focus attention on aberrant miRNA expression in Alzheimer's disease brains...
Protein microarray analysis identifies human cellular prion protein interactorsJ Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo, Japan
Neuropathol Appl Neurobiol 35:16-35. 2009..To obtain an insight into the function of cellular prion protein (PrPC), we studied PrPC-interacting proteins (PrPIPs) by analysing a protein microarray...
Immunohistochemical characterization of microglia in Nasu-Hakola disease brainsJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University Department of Clinical Neuropathology, Tokyo, Japan
Neuropathology 31:363-75. 2011....
Molecular network analysis of T-cell transcriptome suggests aberrant regulation of gene expression by NF-kappaB as a biomarker for relapse of multiple sclerosisJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Japan
Dis Markers 25:27-35. 2008....
[Recent progress in bioinformatics for microarray analysis]Jun ichi Satoh
Department of Bioinformatics, Faculty of Pharmaceutical Sciences, Meiji Pharmaceutical University, Kiyose City, Tokyo, Japan
Yakugaku Zasshi 128:1537-45. 2008..Thus, the molecular network analysis is highly valuable to extract biological implications from a huge amount of microarray data...
Molecular network of the comprehensive multiple sclerosis brain-lesion proteomeJ I Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo, Japan Department of Immunology, National Institute of Neuroscience, NCNP, Tokyo, Japan
Mult Scler 15:531-41. 2009..However, among thousands of proteins examined, nearly all of remaining proteins are yet to be characterized in terms of their implications in MS brain-lesion development...
Stable expression of neurogenin 1 induces LGR5, a novel stem cell marker, in an immortalized human neural stem cell line HB1.F3Jun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo, 204 8588, Japan
Cell Mol Neurobiol 30:415-26. 2010....
Molecular network analysis suggests aberrant CREB-mediated gene regulation in the Alzheimer disease hippocampusJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Noshio, Kiyose, Tokyo, Japan
Dis Markers 27:239-52. 2009....
Human astrocytes express aquaporin-1 and aquaporin-4 in vitro and in vivoJun ichi Satoh
Department of Bioinformatics, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo 204 8588, Japan
Neuropathology 27:245-56. 2007....
Aberrant transcriptional regulatory network in T cells of multiple sclerosisJun ichi Satoh
Department of Bioinformatics, Meiji Pharmaceutical University, Tokyo, Japan
Neurosci Lett 422:30-3. 2007..This novel bioinformatic approach proposes the logical hypothesis that aberrant regulation of the complex transcriptional regulatory network contributes to development of pathogenic T cells in MS...
TROY and LINGO-1 expression in astrocytes and macrophages/microglia in multiple sclerosis lesionsJ Satoh
Department of Bioinformatics, Meiji Pharmaceutical University, Tokyo, Japan
Neuropathol Appl Neurobiol 33:99-107. 2007....
Comprehensive analysis of human microRNA target networksJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo 204 8588, Japan
BioData Min 4:17. 2011..abstract:..
Human astrocytes express 14-3-3 sigma in response to oxidative and DNA-damaging stressesJun ichi Satoh
Department of Bioinformatics and Neuroinformatics, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo, Japan
Neurosci Res 56:61-72. 2006..These observations suggest that 14-3-3sigma might serve as a marker of oxidative and DNA-damaging stresses inducing the mitotic checkpoint dysfunction in reactive astrocytes under pathological conditions...
Protein microarray analysis identifies cyclic nucleotide phosphodiesterase as an interactor of Nogo-AKenta Sumiyoshi
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo 204 8588, Japan
Neuropathology 30:7-14. 2010..Although CNP located chiefly in the cytoplasm of oligodendrocytes might not serve as a cell-surface NIG receptor, it could act as a conformational stabilizer for the intrinsically unstructured large segment of Amino-Nogo...
Gene expression profiling of human neural progenitor cells following the serum-induced astrocyte differentiationShinya Obayashi
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo, 204 8588, Japan
Cell Mol Neurobiol 29:423-38. 2009....
Neuromyelitis optica/Devic's disease: gene expression profiling of brain lesionsJun ichi Satoh
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo, Japan
Neuropathology 28:561-76. 2008..These results suggest that profound activation of the macrophage-mediated proinflammatory immune mechanism plays a pivotal role in development of NMO brain lesions...
Nogo-A and nogo receptor expression in demyelinating lesions of multiple sclerosisJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, Tokyo, Japan
J Neuropathol Exp Neurol 64:129-38. 2005....
Close association of water channel AQP1 with amyloid-beta deposition in Alzheimer disease brainsTamako Misawa
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo, 204 8588, Japan
Acta Neuropathol 116:247-60. 2008..These observations suggest the possible association of astrocytic AQP1 with Abeta deposition in AD brains...
The 14-3-3 protein epsilon isoform expressed in reactive astrocytes in demyelinating lesions of multiple sclerosis binds to vimentin and glial fibrillary acidic protein in cultured human astrocytesJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, 4 1 1 Ogawahigashi, Kodaira, Tokyo 187 8502, Japan
Am J Pathol 165:577-92. 2004..These results suggest that the 14-3-3 protein plays an organizing role in the intermediate filament network in reactive astrocytes at the site of demyelinating lesions in MS...
The 14-3-3 protein forms a molecular complex with heat shock protein Hsp60 and cellular prion proteinJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, Tokyo, Japan
J Neuropathol Exp Neurol 64:858-68. 2005....
Microarray analysis identifies a set of CXCR3 and CCR2 ligand chemokines as early IFNbeta-responsive genes in peripheral blood lymphocytes in vitro: an implication for IFNbeta-related adverse effects in multiple sclerosisJun ichi Satoh
Department of Bioinformatics, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo 204 8588, Japan
BMC Neurol 6:18. 2006..The aim of this study is to identify a comprehensive list of early IFNbeta-responsive genes (IRGs) in peripheral blood mononuclear cells (PBMC) that may play a key role in induction of adverse effects...
BmDJ-1 is a key regulator of oxidative modification in the development of the silkworm, Bombyx moriHiroko Tabunoki
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, Tokyo, Japan
PLoS ONE 6:e17683. 2011..These results suggest that BmDJ-1 might control oxidative stress in the cell due to NO and serves as a development modulation factor in B. mori...
Non-phosphorylated FTY720 induces apoptosis of human microglia by activating SREBP2Takashi Yoshino
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo 204 8588, Japan
Cell Mol Neurobiol 31:1009-20. 2011..These observations suggest that FTY720-non-P-induced apoptosis of HMO6 human microglia is independent of S1P receptor binding, and positively regulated by the SREBP2-dependent proapoptotic signaling pathway...
The constitutive and inducible expression of Nurr1, a key regulator of dopaminergic neuronal differentiation, in human neural and non-neural cell linesJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, Kodaira, Tokyo, Japan
Neuropathology 22:219-32. 2002....
Rapid identification of 14-3-3-binding proteins by protein microarray analysisJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, 4 1 1 Ogawahigashi, Kodaira, Tokyo 187 8502, Japan
J Neurosci Methods 152:278-88. 2006..These results suggest that protein microarray is a powerful tool for rapid and comprehensive profiling of 14-3-3-binding proteins...
Gene expression profile following stable expression of the cellular prion proteinJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, Tokyo, Japan
Cell Mol Neurobiol 24:793-814. 2004..This represents a possible mechanism underlying PrPC-mediated selective neurodegeneration...
Detection of the 14-3-3 protein in the cerebrospinal fluid of Japanese multiple sclerosis patients presenting with severe myelitisJun ichi Satoh
Department of Immunology, National Institute of Neuroscience, NCNP, 4 1 1 Ogawahigashi, Kodaira, Tokyo 187 8502, Japan
J Neurol Sci 212:11-20. 2003....
[Molecular biomarkers for prediction of multiple sclerosis relapse]Jun ichi Satoh
Department of Bioinformatics, Meiji Pharmaceutical University
Nihon Rinsho 66:1103-11. 2008..Thus, DNA microarray technology is highly valuable to identify molecular mechanism-based biomarkers for classification of MS subgroups and prediction of MS relapse...
TDP-43 dimerizes in human cells in cultureYuki Shiina
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio Kiyose, Tokyo, 204 8588, Japan
Cell Mol Neurobiol 30:641-52. 2010..These results suggest that the 86-kDa band represents dimerized TDP-43 expressed constitutively in normal cells under physiological conditions...
Identification of Bombyx mori 14-3-3 orthologs and the interactor Hsp60Hiroko Tabunoki
Department of Bioinformatics and Molecular Neuropathology, Meiji Pharmaceutical University, 2 522 1 Noshio, Kiyose, Tokyo 204 8588, Japan
Neurosci Res 61:271-80. 2008..These observations suggest that a molecular adaptor 14-3-3 and a molecular chaperone Hsp60 cooperate to achieve a wide range of cellular functions in B. mori...
[Molecular network analysis of multiple sclerosis brain lesion proteome]Jun ichi Satoh
Department of Bioinformatics, Meiji Pharmaceutical University, Tokyo, Japan
Nihon Rinsho Meneki Gakkai Kaishi 33:182-8. 2010..These observations indicate that the selective blockade of the interaction between ECM and integrins would be a rational approach for designing inhibitors of chronic inflammatory demyelination in MS brain lesions...
