Research Topics
| Yasunori OkadaSummaryAffiliation: Gunma University Country: Japan Publications
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Detail Information
Publications
Serum procalcitonin concentration in patients with Kawasaki diseaseYasunori Okada
Department of Pediatrics, Gunma University School of Medicine, Showa machi 3 39 22, Maebashi, Gunma 371 8511, Japan
J Infect 48:199-205. 2004..Procalcitonin (PCT) is a new parameter of inflammation, the clinical usefulness of which is currently being evaluated...- Effect of corticosteroids in addition to intravenous gamma globulin therapy on serum cytokine levels in the acute phase of Kawasaki disease in childrenYasunori Okada
Department of Pediatrics, Gunma University School of Medicine, Department of Cardiology, Maebashi, Gunma, Japan
J Pediatr 143:363-7. 2003..v.GG) therapy on serum cytokine levels, as well as to see its effect on the clinical course in children in the acute phase of Kawasaki disease (KD)... - A multicenter prospective randomized trial of corticosteroids in primary therapy for Kawasaki disease: clinical course and coronary artery outcomeYoshinari Inoue
Department of Pediatrics and Developmental Medicine, Gunma University Graduate School of Medicine, 3 29 22 Showa machi, Maebashi, Gunma 371 8511, Japan
J Pediatr 149:336-341. 2006..To investigate the role of corticosteroids in the initial treatment of Kawasaki disease (KD)... - Caudal regression and tracheoesophageal malformation induced by adriamycin: a novel chick model of VATER associationYoko Naito
Department of Pediatrics, Keio University School of Medicine, Tokyo, Japan
Pediatr Res 65:607-12. 2009.... - Identification of a prosencephalic-specific enhancer of SALL1: comparative genomic approach using the chick embryoKosuke Izumi
Department of Pediatrics, Keio University School of Medicine, Tokyo 160 8582, Japan
Pediatr Res 61:660-5. 2007..This report identifies, for the first time, an enhancer element of SALL1... - Sulfonated human immunoglobulin enhances CD16-linked CD11b expression on human neutrophilsHirokazu Kimura
Gunma Prefectural Institute of Public Health and Environmental Sciences, 378, Kamioki, Maebashi, Gunma 371 0052, Japan
Cell Biol Int 27:913-9. 2003..CD11b induction was inhibited by dexamethasone and by anti-CD16 antibody. These in vitro results suggest that aggregations and enhancement of CD11b on neutrophils by SHIG may induce excessive inflammatory responses in vivo... - Spontaneous closure of an unroofed coronary sinusMasayuki Watanabe
Department of Pediatrics, Saiseikai Maebashi General Hospital, Maebashi, Gunma, Japan
Pediatr Int 46:722-5. 2004 - Embryonic expression profile of chicken CHD7, the ortholog of the causative gene for CHARGE syndromeMichihiko Aramaki
Department of Pediatrics, Keio University School of Medicine, Tokyo, Japan
Birth Defects Res A Clin Mol Teratol 79:50-7. 2007..1 was identified as a causative gene for CHARGE syndrome. Because CHD7 was identified as a causative gene using a positional cloning approach, the role of CHD7 in early embryogenesis needs to be further investigated... - Atlanto-axial subluxation (Grisel's syndrome) associated with mumpsYasunori Okada
Department of Pediatrics, National Takasaki Hospital, Takasaki, Japan
Pediatr Int 44:192-4. 2002 - Prediction of intravenous immunoglobulin unresponsiveness in patients with Kawasaki diseaseTohru Kobayashi
Department of Pediatrics and Developmental Medicine, Gunma University Graduate School of Medicine, Gunma 377 8511, Japan
Circulation 113:2606-12. 2006..In the present study, we developed models to predict unresponsiveness to intravenous immunoglobulin (IVIG) in Kawasaki disease (KD)... - ADAM-12 (meltrin alpha) is involved in chondrocyte proliferation via cleavage of insulin-like growth factor binding protein 5 in osteoarthritic cartilageAiko Okada
School of Medicine, Keio University, Tokyo, Japan
Arthritis Rheum 58:778-89. 2008..ADAMs are a gene family of multifunctional proteins. We undertook this study to determine which ADAM species is up-regulated in osteoarthritic (OA) cartilage and to examine its pathobiologic function... - Expression of ADAM15 in rheumatoid synovium: up-regulation by vascular endothelial growth factor and possible implications for angiogenesisKoichiro Komiya
Department of Pathology, School of Medicine, Keio University, Tokyo, Japan
Arthritis Res Ther 7:R1158-73. 2005..These data demonstrate that ADAM15 is overexpressed in RA synovium and its expression is up-regulated by the action of VEGF165 through VEGFR-2, and suggest the possibility that ADAM15 is involved in angiogenesis in RA synovium... - Targeted deletion or pharmacological inhibition of MMP-2 prevents cardiac rupture after myocardial infarction in miceShin ichiro Matsumura
Department of Pathology, School of Medicine, Keio University, Tokyo, Japan
J Clin Invest 115:599-609. 2005..These data suggest that inhibition of MMP-2 activity improves the survival rate after acute MI by preventing cardiac rupture and delays post-MI remodeling through a reduction in macrophage infiltration... - Deficiency of inducible nitric oxide synthase exacerbates hepatic fibrosis in mice fed high-fat dietYi Chen
Department of Physiology, School of Medicine, Tokai University, Bohseidai, Isehara, Kanagawa 259 1193, Japan
Biochem Biophys Res Commun 326:45-51. 2005..iNOS may attenuate the progression of liver fibrosis in steatohepatitis, in part by inducing MMP-2 and MMP-9 expression and augmenting their activity... - Progressive adult-onset emphysema in transgenic mice expressing human MMP-1 in the lungRobert F Foronjy
Department of Medicine, Division of Molecular Medicine and Pulmonary Medicine, Columbia University, New York, New York 10032, USA
Am J Physiol Lung Cell Mol Physiol 284:L727-37. 2003..This study provides evidence that MMP-1 induces progressive adult-onset emphysema by the selective degradation of type III collagen within the alveolar wall... - Decreased expression of L-selectin on peripheral blood polymorphonuclear leukocytes in neonates with severe asphyxiaMasaki Hashimoto
Neonatal Unit, Children's Hospital Hachioji, Hachioji, Tokyo, Japan
Biol Neonate 81:95-8. 2002..These results suggest that the magnitude of the decrease in L-selectin expression reflects the severity of asphyxia in neonates...