Research Topics
Species | S KunishimaSummaryAffiliation: Fujita Health University Country: Japan Publications
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Detail Information
Publications
Platelet glycoprotein (GP) V polymorphisms in JapaneseS Kunishima
Japanese Red Cross Aichi Blood Center, Seto
Tissue Antigens 54:285-7. 1999..Flow cytometric analysis showed that the 341Arg variant is expressed normally on the platelet membranes, suggesting the possibility of the involvement of the 341Gly/Arg polymorphism as a platelet alloantigen...
Cys97-->Tyr mutation in the glycoprotein IX gene associated with Bernard-Soulier syndromeS Kunishima
Japanese Red Cross Aichi Blood Centre, Seto, Aichi, Japan
Br J Haematol 107:539-45. 1999..Transient transfection studies confirmed that mutant GPIX was not expressed on the transfected cells, showing that the mutation was responsible for the BSS phenotype observed in the patient...
Homozygous Pro74-->Arg mutation in the platelet glycoprotein Ibbeta gene associated with Bernard-Soulier syndromeS Kunishima
Japanese Red Cross Aichi Blood Center, Seto, Japan
Thromb Haemost 84:112-7. 2000..The mutant GPIbbeta was normally transcribed. Transient transfection studies confirmed that mutant GPIbbeta impairs surface expression of GPIb/IX, showing that the mutation is responsible for a BSS phenotype observed in the patient...
Novel heterozygous missense mutation in the platelet glycoprotein Ib beta gene associated with isolated giant platelet disorderS Kunishima
Japanese Red Cross Aichi Blood Center, Seto, Japan
Am J Hematol 68:249-55. 2001....
Expression of low-frequency Ala108Pro substitution in the platelet glycoprotein Ibbeta geneS Kunishima
Japanese Red Cross Aichi Blood Center, Seto, Japan
Eur J Immunogenet 30:159-61. 2003..In vitro expression studies showed surface expression of the GPIbbeta Pro108 variant, suggesting the possibility of the involvement of the substitution as an alloantigen...
Presence of Propionibacterium acnes in blood componentsS Kunishima
Japanese Red Cross Aichi Blood Center, Minamiyamaguchi, Seto, Japan
Transfusion 41:1126-9. 2001..18%), all of which were RBC concentrates, were contaminated with bacteria. Nine isolates were Propionibacterium acnes and one was Staphylococcus capitis...
A new polymorphism in the HLA-F gene (67Ala[GCC] to Ala[GCG])S Kunishima
Japanese Red Cross Aichi Blood Center, Seto, Japan
Immunogenetics 49:147-8. 1999
A missense mutation (Tyr88 to Cys) in the platelet membrane glycoprotein Ibbeta gene affects GPIb/IX complex expression--Bernard-Soulier syndrome in the homozygous form and giant platelets in the heterozygous formY Kurokawa
Department of Pediatrics, Shinshu University School of Medicine, Matsumoto, Japan
Thromb Haemost 86:1249-56. 2001..Tyr88 in the GPIbbeta gene plays a significant role in the GPIb/IX expression; the defect causes BSS in a homozygous form and possibly giant platelets in a heterozygous form...
Missense mutations of the glycoprotein (GP) Ib beta gene impairing the GPIb alpha/beta disulfide linkage in a family with giant platelet disorderS Kunishima
Department of Medicine, Nagoya University Branch Hospital, Higashi ku, Japan
Blood 89:2404-12. 1997..Furthermore, the phenotype caused by mutations in the subunits of the GPIb/IX complex could span the spectrum from a normal phenotype, to isolated GPD, to a full-blown bleeding disorder, such as Bernard-Soulier syndrome...
Giant platelet syndromeH Saito
Nagoya Medical Center and Nagoya University School of Medicine, Nagoya, Japan
Hematology 10:41-6. 2005
