Laszlo Buday

Summary

Affiliation: Semmelweis University
Country: Hungary

Publications

  1. ncbi The Nck family of adapter proteins: regulators of actin cytoskeleton
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Str, 1088, Budapest, Hungary
    Cell Signal 14:723-31. 2002
  2. ncbi Many faces of Ras activation
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    Biochim Biophys Acta 1786:178-87. 2008
  3. ncbi Roles of cortactin in tumor pathogenesis
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    Biochim Biophys Acta 1775:263-73. 2007
  4. ncbi Phorbol ester-induced migration of HepG2 cells is accompanied by intensive stress fibre formation, enhanced integrin expression and transient down-regulation of p21-activated kinase 1
    Annamária Gujdár
    Department of Medical Chemistry, Molecular Biology and Pathobiochemistry, Semmelweis University of Medicine, PO Box 260, 1444 Budapest, Hungary
    Cell Signal 15:307-18. 2003
  5. ncbi Mechanism of epidermal growth factor regulation of Vav2, a guanine nucleotide exchange factor for Rac
    Peter Tamas
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    J Biol Chem 278:5163-71. 2003
  6. ncbi High levels of structural disorder in scaffold proteins as exemplified by a novel neuronal protein, CASK-interactive protein1
    Annamária Balázs
    Department of Medical Chemistry, Semmelweis University Medical School, Budapest, Hungary
    FEBS J 276:3744-56. 2009
  7. ncbi Intrinsic structural disorder confers cellular viability on oncogenic fusion proteins
    Hedi Hegyi
    Institute of Enzymology, Biological Research Center, Hungarian Academy of Sciences, Budapest, Hungary
    PLoS Comput Biol 5:e1000552. 2009
  8. ncbi Functional classification of scaffold proteins and related molecules
    Laszlo Buday
    Institute of Enzymology, Biological Research Center, Hungarian Academy of Sciences, Budapest, Hungary
    FEBS J 277:4348-55. 2010

Collaborators

  • Julian Downward
  • Szabolcs Sipeki
  • Hedi Hegyi
  • Annamária Balázs
  • Peter Tamas
  • Annamária Gujdár
  • Anna Farago
  • Marianna Rakacs
  • Veronika Csizmok
  • Robert Kiss
  • Kalman Tompa
  • Peter Tompa
  • Roopesh Udupa
  • Monika Bokor
  • Andras Bauer
  • Erzsébet Bander
  • Petra Bauer
  • Zita Solti
  • András Illés

Detail Information

Publications11

  1. ncbi The Nck family of adapter proteins: regulators of actin cytoskeleton
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Str, 1088, Budapest, Hungary
    Cell Signal 14:723-31. 2002
    ..The proteins of Nck family are implicated in organisation of actin cytoskeleton, cell movement or axon guidance in flies. In this review, the author attempts to summarise signalling pathways in which Nck plays a critical role...
  2. ncbi Many faces of Ras activation
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    Biochim Biophys Acta 1786:178-87. 2008
    ..Although all of them are able to activate Ras, their structure, tissue expression and regulation are significantly diverse. In this review we will summarize the various mechanisms by which these exchange factors activate Ras...
  3. ncbi Roles of cortactin in tumor pathogenesis
    Laszlo Buday
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    Biochim Biophys Acta 1775:263-73. 2007
    ..This review aims to focus on cortactin-mediated signaling pathways, with emphasis on its contribution to tumor progression and metastasis formation...
  4. ncbi Phorbol ester-induced migration of HepG2 cells is accompanied by intensive stress fibre formation, enhanced integrin expression and transient down-regulation of p21-activated kinase 1
    Annamária Gujdár
    Department of Medical Chemistry, Molecular Biology and Pathobiochemistry, Semmelweis University of Medicine, PO Box 260, 1444 Budapest, Hungary
    Cell Signal 15:307-18. 2003
    ..Protein kinase C stimulated the activation of p21-activated kinase (PAK), as well. However, it also stimulated the selective and transient down-regulation of PAK1, which coincided with the formation of stress fibres...
  5. ncbi Mechanism of epidermal growth factor regulation of Vav2, a guanine nucleotide exchange factor for Rac
    Peter Tamas
    Department of Medical Chemistry, Semmelweis University Medical School, 9 Puskin Street, 1088 Budapest, Hungary
    J Biol Chem 278:5163-71. 2003
    ..These results suggest that EGF regulates Vav2 activity basically through phosphatidylinositol 3-kinase activation, whereas tyrosine phosphorylation of Vav2 may rather be necessary for mediating protein-protein interactions...
  6. ncbi High levels of structural disorder in scaffold proteins as exemplified by a novel neuronal protein, CASK-interactive protein1
    Annamária Balázs
    Department of Medical Chemistry, Semmelweis University Medical School, Budapest, Hungary
    FEBS J 276:3744-56. 2009
    ....
  7. ncbi Intrinsic structural disorder confers cellular viability on oncogenic fusion proteins
    Hedi Hegyi
    Institute of Enzymology, Biological Research Center, Hungarian Academy of Sciences, Budapest, Hungary
    PLoS Comput Biol 5:e1000552. 2009
    ..g. EWS-ATF). Our findings also suggest novel strategies of intervention against the ensuing neoplastic transformations...
  8. ncbi Functional classification of scaffold proteins and related molecules
    Laszlo Buday
    Institute of Enzymology, Biological Research Center, Hungarian Academy of Sciences, Budapest, Hungary
    FEBS J 277:4348-55. 2010
    ..Thus, the field is in a state of continuous advance and expansion, which demands that the classification scheme be regularly updated and, if needed, revised...