Ines Anderie

Summary

Affiliation: Saarland University
Country: Germany

Publications

  1. ncbi Tyrosine phosphatase PTP1B interacts with TRPV6 in vivo and plays a role in TRPV6-mediated calcium influx in HEK293 cells
    Lutz Sternfeld
    Physiological Institute, University of the Saarland, Germany
    Cell Signal 17:951-60. 2005
  2. ncbi In vivo visualization of actin dynamics and actin interactions by BiFC
    Ines Anderie
    Department of Physiology, University of the Saarland, Homburg, Saar, Germany
    Cell Biol Int 31:1131-5. 2007
  3. ncbi Characterization of the C-terminal ER membrane anchor of PTP1B
    Ines Anderie
    Department of Physiology, University of the Saarland, D 66421 Homburg Saar, Germany
    Exp Cell Res 313:3189-97. 2007
  4. ncbi Direct interaction between ER membrane-bound PTP1B and its plasma membrane-anchored targets
    Ines Anderie
    Department of Physiology, University of the Saarland, D 66421, Homburg Saar, Germany
    Cell Signal 19:582-92. 2007
  5. ncbi Activation of muscarinic receptors reduces store-operated Ca2+ entry in HEK293 cells
    Lutz Sternfeld
    Department of Physiology, University of Saarland, Medical Faculty, Gebäude 58, D 66421 Homburg Saar, Germany
    Cell Signal 19:1457-64. 2007
  6. ncbi Tyrosine phosphatase PTP1B modulates store-operated calcium influx
    Shyuefang Hsu
    Department of Physiology, University of the Saarland, Building 58, D-66421, Homburg/Saar, Germany
    Cell Signal 15:1149-56. 2003

Collaborators

  • Lutz Sternfeld
  • Elmar Krause
  • Andreas Schmid
  • Irene Schulz
  • Hans Werner Hofer
  • Shyuefang Hsu
  • Diana Heinze
  • Monika Dudenhöffer
  • Anja Ludes
  • Hajaj Al-Shaldi
  • Anna Latas
  • Katrin Evers
  • Andreas Kohl
  • Gonzalo Solis

Detail Information

Publications6

  1. ncbi Tyrosine phosphatase PTP1B interacts with TRPV6 in vivo and plays a role in TRPV6-mediated calcium influx in HEK293 cells
    Lutz Sternfeld
    Physiological Institute, University of the Saarland, Germany
    Cell Signal 17:951-60. 2005
    ..These data indicate that tyrosine phosphorylation is involved in the regulation of the TRPV6 channel protein...
  2. ncbi In vivo visualization of actin dynamics and actin interactions by BiFC
    Ines Anderie
    Department of Physiology, University of the Saarland, Homburg, Saar, Germany
    Cell Biol Int 31:1131-5. 2007
    ..Our experiments demonstrate that the BiFC method might be a useful tool to investigate participation of the actin cytoskeleton in regulation of cell function...
  3. ncbi Characterization of the C-terminal ER membrane anchor of PTP1B
    Ines Anderie
    Department of Physiology, University of the Saarland, D 66421 Homburg Saar, Germany
    Exp Cell Res 313:3189-97. 2007
    ..TMD and tail length are variable and there are no sequence-specific features. Our data for PTP1B are consistent with a concept that explains the ER membrane anchor of tail-anchored proteins as a physicochemical structure...
  4. ncbi Direct interaction between ER membrane-bound PTP1B and its plasma membrane-anchored targets
    Ines Anderie
    Department of Physiology, University of the Saarland, D 66421, Homburg Saar, Germany
    Cell Signal 19:582-92. 2007
    ..The reported formation of membrane junctions is an important step towards the understanding of signal transmissions between the ER and the plasma membrane...
  5. ncbi Activation of muscarinic receptors reduces store-operated Ca2+ entry in HEK293 cells
    Lutz Sternfeld
    Department of Physiology, University of Saarland, Medical Faculty, Gebäude 58, D 66421 Homburg Saar, Germany
    Cell Signal 19:1457-64. 2007
    ..This mechanism may serve as a modulator of Ca2+ entry preventing a Ca2+ overload of the cytoplasm after Ca2+ store depletion...
  6. ncbi Tyrosine phosphatase PTP1B modulates store-operated calcium influx
    Shyuefang Hsu
    Department of Physiology, University of the Saarland, Building 58, D-66421, Homburg/Saar, Germany
    Cell Signal 15:1149-56. 2003
    ..It also decreases enzymatic activity of PTP1B as compared to non-transfected cells. Our data suggest that CRAC influx is modulated by tyrosine phosphorylation and dephosphorylation which involves the tyrosine phosphatase PTP1B...