Research Topics
Species | Thomas MankeSummaryAffiliation: Max Planck Institute for Molecular Genetics Country: Germany Publications
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Detail Information
Publications
Quantifying the effect of sequence variation on regulatory interactionsThomas Manke
Max Planck Institute for Molecular Genetics, Computational Biology, Ihnestrasse 73, Berlin, Germany
Hum Mutat 31:477-83. 2010..Given the good performance of our method, we developed a publicly available tool that can serve as an important starting point for routine analysis of disease-associated sequence regions...
Lethality and entropy of protein interaction networksThomas Manke
Max Planck Institute for Molecular Genetics, Ihnestrasse 73, 14195 Berlin, Germany
Genome Inform 16:159-63. 2005..It is also applicable to networks in which the processes can be quantified and therefore serves as a link to study questions of structural and dynamical robustness in a unified way...
An entropic characterization of protein interaction networks and cellular robustnessThomas Manke
Max Planck Institute for Molecular Genetics, Ihnestr 73, 14195 Berlin, Germany
J R Soc Interface 3:843-50. 2006..One of its principal achievements is to provide a rationale to study proxies of cellular resilience and rank proteins according to their importance within the global network context...
Statistical modeling of transcription factor binding affinities predicts regulatory interactionsThomas Manke
Max Planck Institute for Molecular Genetics, Berlin, Germany
PLoS Comput Biol 4:e1000039. 2008..Its successful application to human promoter sequences serves as an encouraging example of how the method can be applied to other sequences...
Functional analysis and identification of cis-regulatory elements of human chromosome 21 gene promotersHans Jörg Warnatz
Department for Vertebrate Genomics, Max Planck Institute for Molecular Genetics, Ihnestrasse 63 73, 14195 Berlin, Germany
Nucleic Acids Res 38:6112-23. 2010..This study illustrates the modular functional architecture of chromosome 21 promoters and helps to reveal the complex mechanisms governing transcriptional regulation...
Predicting transcription factor affinities to DNA from a biophysical modelHelge G Roider
Max Planck Institute for Molecular Genetics Ihnestrasse 73, 14195 Berlin, Germany
Bioinformatics 23:134-41. 2007..The question arises to what extent these two approaches converge. In this paper, we adopt a physical binding model to predict the relative binding affinity of a transcription factor for a given sequence...
Prioritization of gene regulatory interactions from large-scale modules in yeastHo Joon Lee
Department of Computational Molecular Biology, Max Planck Institute for Molecular Genetics, 14195 Berlin, Germany
BMC Bioinformatics 9:32. 2008..In this work, we aim to prioritize regulator-target links within transcriptional modules based on three types of large-scale data sources...
PASTAA: identifying transcription factors associated with sets of co-regulated genesHelge G Roider
Max Planck Institute for Molecular Genetics, Ihnestrasse 73, 14195 Berlin
Bioinformatics 25:435-42. 2009..While, for a limited number of categories, the regulating TFs are already known, still for many functional categories the responsible factors remain to be elucidated...
The BTB and CNC homology 1 (BACH1) target genes are involved in the oxidative stress response and in control of the cell cycleHans Jörg Warnatz
Department of Vertebrate Genomics, Max Planck Institute for Molecular Genetics, 14195 Berlin, Germany
J Biol Chem 286:23521-32. 2011....
MicroRNAs differentially expressed in postnatal aortic development downregulate elastin via 3' UTR and coding-sequence binding sitesClaus Eric Ott
Institute for Medical Genetics, Charite Universitatsmedizin Berlin, Berlin, Germany
PLoS ONE 6:e16250. 2011..Our results demonstrate that multiple miR-29 and miR-15 family MREs are characteristic for some ECM genes and suggest that miR-29 and miR-15 family miRNAs are involved in the down-regulation of elastin in the adult aorta...
Transcription factor binding predictions using TRAP for the analysis of ChIP-seq data and regulatory SNPsMorgane Thomas-Chollier
Department of Computational Molecular Biology, Max Planck Institute for Molecular Genetics, Berlin, Germany
Nat Protoc 6:1860-9. 2011..All of the tools are fully available online and do not need any additional installation. The complete protocol takes about 45 min, but each individual tool runs in a few minutes...
Screening of human gene promoter activities using transfected-cell arraysXi Cheng
Department of Vertebrate Genomics, Max Planck Institute for Molecular Genetics, 14195 Berlin, Germany
Gene 450:48-54. 2010..Moreover, this is the first large-scale functional study of regulatory sequences to use a high-throughput transfected-cell array technique...
Integrating sequence, evolution and functional genomics in regulatory genomicsMartin Vingron
Computational Molecular Biology, Max Planck Institut fur Molekulare Genetik, Berlin, Germany
Genome Biol 10:202. 2009....
Correlating protein-DNA and protein-protein interaction networksThomas Manke
Max-Planck-Institute for Molecular Genetics, Abt Bioinformatik, Ihnestr. 73, 14195 Berlin, Germany
J Mol Biol 333:75-85. 2003..In particular we find that directly interacting transcription factors and those which are members of a protein complex are more likely to occur together as putative DNA-binding modules...
Promiscuous and depolarization-induced immediate-early response genes are induced by mechanical strain of osteoblastsClaus Eric Ott
Institute for Medical Genetics, Charite Universitatsmedizin Berlin, Berlin, Germany
J Bone Miner Res 24:1247-62. 2009....
Proteomic shifts in embryonic stem cells with gene dose modifications suggest the presence of balancer proteins in protein regulatory networksLei Mao
Institute for Human Genetics, Charite University Medicine Berlin, Germany
PLoS ONE 2:e1218. 2007..We propose that the "elasticity" of the proteomic regulatory network mediated by balancer proteins may compensate for changes that occur under diseased conditions...
Control of replication initiation and heterochromatin formation in Saccharomyces cerevisiae by a regulator of meiotic gene expressionHorst Irlbacher
Otto Warburg Laboratorium and Department for Computational Molecular Biology, Max Planck Institut fur Molekulare Genetik, D 14195 Berlin, Germany
Genes Dev 19:1811-22. 2005..Full initiation activity of these origins required Sum1, and their origin activity was decreased upon removal of the Sum1-binding site. Thus, Sum1 constitutes a novel global regulator of replication initiation in yeast...
