Manoj Kumar Jaiswal

Summary

Country: Germany

Publications

  1. ncbi Expression of a Cu,Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of neuroblastoma cells to infectious injury
    Miriam Goos
    Department of Neurology, Georg August University of Gottingen, Gottingen, Germany
    BMC Infect Dis 7:131. 2007
  2. ncbi Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease
    Manoj Kumar Jaiswal
    Center of Physiology, Georg August University, Goettingen, Germany
    BMC Neurosci 10:64. 2009
  3. ncbi Cu/Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of mitochondria and perturbs Ca2+ homeostasis in SOD1G93A mice
    Manoj Kumar Jaiswal
    Center of Physiology, University of Goettingen, Goettingen, Germany
    Mol Pharmacol 75:478-89. 2009

Detail Information

Publications3

  1. ncbi Expression of a Cu,Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of neuroblastoma cells to infectious injury
    Miriam Goos
    Department of Neurology, Georg August University of Gottingen, Gottingen, Germany
    BMC Infect Dis 7:131. 2007
    ..Streptococcus pneumoniae, the most frequent respiratory pathogen, causes damage by the action of the cholesterol-binding virulence factor pneumolysin and by stimulation of the innate immune system, particularly via Toll-like-receptor 2...
  2. ncbi Impairment of mitochondrial calcium handling in a mtSOD1 cell culture model of motoneuron disease
    Manoj Kumar Jaiswal
    Center of Physiology, Georg August University, Goettingen, Germany
    BMC Neurosci 10:64. 2009
    ..In this study, we used a fluorescence charged cool device (CCD) imaging system to separate and simultaneously monitor cytosolic and mitochondrial calcium concentrations in individual cells in an established cellular model of ALS...
  3. ncbi Cu/Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of mitochondria and perturbs Ca2+ homeostasis in SOD1G93A mice
    Manoj Kumar Jaiswal
    Center of Physiology, University of Goettingen, Goettingen, Germany
    Mol Pharmacol 75:478-89. 2009
    ..The stabilization of mitochondria-related signal cascades may represent a useful strategy for clinical neuroprotection in ALS...