Jean Philippe Haymann

Summary

Country: France

Publications

  1. ncbi Glomerular hyperfiltration in adult sickle cell anemia: a frequent hemolysis associated feature
    Jean Philippe Haymann
    Universite Pierre et Marie Curie, Universite Paris 6, Institut National de la Santé et de la Recherche Médicale Unit 702, Physiology, Paris, France
    Clin J Am Soc Nephrol 5:756-61. 2010
  2. ncbi Calpain activation and secretion promote glomerular injury in experimental glomerulonephritis: evidence from calpastatin-transgenic mice
    Julie Peltier
    INSERM U702, 7Université Pierre et Marie Curie Paris6, UMRS702, Paris, France
    J Am Soc Nephrol 17:3415-23. 2006
  3. ncbi Strong association between a new marker of hemolysis and glomerulopathy in sickle cell anemia
    Micheline Maier-Redelsperger
    Service d Hematologie Biologique, Hopital Tenon, Assistance Publique Hopitaux de Paris, F 75020, Paris, France
    Blood Cells Mol Dis 45:289-92. 2010
  4. ncbi Critical role of the calpain/calpastatin balance in acute allograft rejection
    Emmanuel Letavernier
    INSERM, UMR S 702, Paris, France
    Eur J Immunol 41:473-84. 2011
  5. ncbi Spectrum of mutations in Gitelman syndrome
    Rosa Vargas-Poussou
    Assistance Publique Hopitaux de Paris, Hopital Europeen Georges Pompidou, Service de Genetique, Paris, France
    J Am Soc Nephrol 22:693-703. 2011
  6. ncbi Determinants of osteopenia in male renal-stone-disease patients with idiopathic hypercalciuria
    Emmanuel Letavernier
    Service des explorations fonctionnelles multidisciplinaires, Hopital Tenon, 4 rue de la Chine, 75020 Paris, France
    Clin J Am Soc Nephrol 6:1149-54. 2011
  7. ncbi Targeting the calpain/calpastatin system as a new strategy to prevent cardiovascular remodeling in angiotensin II-induced hypertension
    Emmanuel Letavernier
    Institut National de la Sante et de la Recherche Medicale, U702, Paris, France
    Circ Res 102:720-8. 2008
  8. ncbi Aggregated IgG bind to glomerular epithelial cells to stimulate urokinase release through an endocytosis-independent process
    Jean Philippe Haymann
    INSERM U 489, Université Paris 6 and Association Claude Bernard, Hopital Tenon, Paris, France
    Nephron Exp Nephrol 98:e13-21. 2004
  9. ncbi Sirolimus interacts with pathways essential for podocyte integrity
    Emmanuel Letavernier
    INSERM, U702 and Université Pierre et Marie Curie Paris 6, UMRS702, Paris, France
    Nephrol Dial Transplant 24:630-8. 2009
  10. ncbi [Metabolic syndrome, obesity and urolithiasis]
    Olivier Traxer
    Service d Urologie, Hopital Tenon, Paris, France
    Prog Urol 16:418-20. 2006

Collaborators

Detail Information

Publications10

  1. ncbi Glomerular hyperfiltration in adult sickle cell anemia: a frequent hemolysis associated feature
    Jean Philippe Haymann
    Universite Pierre et Marie Curie, Universite Paris 6, Institut National de la Santé et de la Recherche Médicale Unit 702, Physiology, Paris, France
    Clin J Am Soc Nephrol 5:756-61. 2010
    ..Glomerular damage is a common feature revealed by a microalbuminuria or a macroalbuminuria. Although glomerular hyperfiltration has been described for decades in this population, its prevalence in young adults is unknown...
  2. ncbi Calpain activation and secretion promote glomerular injury in experimental glomerulonephritis: evidence from calpastatin-transgenic mice
    Julie Peltier
    INSERM U702, 7Université Pierre et Marie Curie Paris6, UMRS702, Paris, France
    J Am Soc Nephrol 17:3415-23. 2006
    ..Thus, both activation and secretion of calpains participate in the development of immune glomerular injury...
  3. ncbi Strong association between a new marker of hemolysis and glomerulopathy in sickle cell anemia
    Micheline Maier-Redelsperger
    Service d Hematologie Biologique, Hopital Tenon, Assistance Publique Hopitaux de Paris, F 75020, Paris, France
    Blood Cells Mol Dis 45:289-92. 2010
    ..Furthermore, by combining this parameter with LDH, we defined a composite variable, which we called CVar, that is highly correlated with albuminuria and might constitute a powerful new marker of risk for this complication...
  4. ncbi Critical role of the calpain/calpastatin balance in acute allograft rejection
    Emmanuel Letavernier
    INSERM, UMR S 702, Paris, France
    Eur J Immunol 41:473-84. 2011
    ..This is the first study establishing that calpain inhibition delays allograft rejection by slowing down T-cell migration rather than proliferation...
  5. ncbi Spectrum of mutations in Gitelman syndrome
    Rosa Vargas-Poussou
    Assistance Publique Hopitaux de Paris, Hopital Europeen Georges Pompidou, Service de Genetique, Paris, France
    J Am Soc Nephrol 22:693-703. 2011
    ....
  6. ncbi Determinants of osteopenia in male renal-stone-disease patients with idiopathic hypercalciuria
    Emmanuel Letavernier
    Service des explorations fonctionnelles multidisciplinaires, Hopital Tenon, 4 rue de la Chine, 75020 Paris, France
    Clin J Am Soc Nephrol 6:1149-54. 2011
    ..Although this pathologic link has been recognized for decades, the underlying mechanisms and risk factors associated with osteopenia/osteoporosis in this population remain partially understood...
  7. ncbi Targeting the calpain/calpastatin system as a new strategy to prevent cardiovascular remodeling in angiotensin II-induced hypertension
    Emmanuel Letavernier
    Institut National de la Sante et de la Recherche Medicale, U702, Paris, France
    Circ Res 102:720-8. 2008
    ..These results indicate a critical role for calpains as downstream mediators in Ang II-induced cardiovascular remodeling and, thus, highlight an attractive therapeutic target...
  8. ncbi Aggregated IgG bind to glomerular epithelial cells to stimulate urokinase release through an endocytosis-independent process
    Jean Philippe Haymann
    INSERM U 489, Université Paris 6 and Association Claude Bernard, Hopital Tenon, Paris, France
    Nephron Exp Nephrol 98:e13-21. 2004
    ..We therefore determined whether this binding could be responsible for a modification in the fibrinolytic activity of GEC...
  9. ncbi Sirolimus interacts with pathways essential for podocyte integrity
    Emmanuel Letavernier
    INSERM, U702 and Université Pierre et Marie Curie Paris 6, UMRS702, Paris, France
    Nephrol Dial Transplant 24:630-8. 2009
    ..Podocyte injury and focal segmental glomerulosclerosis have been related to sirolimus therapy in some patients but the pathways underlying these lesions remain hypothetical...
  10. ncbi [Metabolic syndrome, obesity and urolithiasis]
    Olivier Traxer
    Service d Urologie, Hopital Tenon, Paris, France
    Prog Urol 16:418-20. 2006
    ..The objective of this article is to review the metabolic syndrome in order to help urologists to recognize this syndrome so that they can identify patients requiring more specific management and medical follow-up...