T H Mogensen

Summary

Affiliation: University of Copenhagen
Country: Denmark

Publications

  1. ncbi Pathogen recognition and inflammatory signaling in innate immune defenses
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, Aarhus, Denmark
    Clin Microbiol Rev 22:240-73, Table of Contents. 2009
  2. ncbi Chronic hepatitis caused by persistent parvovirus B19 infection
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, DK 8200 Aarhus N, Denmark
    BMC Infect Dis 10:246. 2010
  3. ncbi Innate immune recognition and activation during HIV infection
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, DK 8200, Aarhus N, Denmark
    Retrovirology 7:54. 2010
  4. ncbi Streptococcus pneumoniae stabilizes tumor necrosis factor alpha mRNA through a pathway dependent on p38 MAPK but independent of Toll-like receptors
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital Aarhus University Hospital, DK 8200, Aarhus N, Denmark
    BMC Immunol 9:52. 2008
  5. ncbi Mechanisms of dexamethasone-mediated inhibition of Toll-like receptor signaling induced by Neisseria meningitidis and Streptococcus pneumoniae
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Brendstrupgaardsvej, DK 8200 Aarhus N, Denmark
    Infect Immun 76:189-97. 2008
  6. ncbi Acute demyelinizating encephalomyelitis (ADEM) as initial presentation of primary HIV infection
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, University Hospital of Aarhus, Aarhus N, Denmark
    Scand J Infect Dis 39:630-4. 2007
  7. ncbi Two neisseria meningitidis strains with different ability to stimulate toll-like receptor 4 through the MyD88-independent pathway
    T H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Aarhus, Denmark
    Scand J Immunol 64:646-54. 2006
  8. ncbi Aseptic meningitis caused by reactivation of varicella-zoster virus in two immunocompetent patients
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, University Hospital of Aarhus, Aarhus, Denmark
    Scand J Infect Dis 38:815-8. 2006
  9. ncbi Live Streptococcus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis activate the inflammatory response through Toll-like receptors 2, 4, and 9 in species-specific patterns
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Brendstrupgaardsvej, DK 8200 Aarhus N, Denmark
    J Leukoc Biol 80:267-77. 2006
  10. ncbi Important role for Toll-like receptor 9 in host defense against meningococcal sepsis
    Hong Sjölinder
    Department of Medical Biochemistry and Microbiology, Biomedical Center, Uppsala University, P O Box 582, Uppsala, Sweden
    Infect Immun 76:5421-8. 2008

Detail Information

Publications15

  1. ncbi Pathogen recognition and inflammatory signaling in innate immune defenses
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, Aarhus, Denmark
    Clin Microbiol Rev 22:240-73, Table of Contents. 2009
    ....
  2. ncbi Chronic hepatitis caused by persistent parvovirus B19 infection
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, DK 8200 Aarhus N, Denmark
    BMC Infect Dis 10:246. 2010
    ..This has prompted important questions regarding the tropism of the virus and its possible involvement in a broad range of infectious and autoimmune medical conditions...
  3. ncbi Innate immune recognition and activation during HIV infection
    Trine H Mogensen
    Department of Infectious Diseases, Aarhus University Hospital, Skejby, DK 8200, Aarhus N, Denmark
    Retrovirology 7:54. 2010
    ....
  4. ncbi Streptococcus pneumoniae stabilizes tumor necrosis factor alpha mRNA through a pathway dependent on p38 MAPK but independent of Toll-like receptors
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital Aarhus University Hospital, DK 8200, Aarhus N, Denmark
    BMC Immunol 9:52. 2008
    ..Infections with S. pneumoniae evoke a strong inflammatory response, which plays a major role in the pathogenesis of pneumococcal disease...
  5. ncbi Mechanisms of dexamethasone-mediated inhibition of Toll-like receptor signaling induced by Neisseria meningitidis and Streptococcus pneumoniae
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Brendstrupgaardsvej, DK 8200 Aarhus N, Denmark
    Infect Immun 76:189-97. 2008
    ..meningitidis and S. pneumoniae, which may contribute to our understanding of the clinical effect and the importance of timing with respect to corticosteroid treatment during bacterial meningitis...
  6. ncbi Acute demyelinizating encephalomyelitis (ADEM) as initial presentation of primary HIV infection
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, University Hospital of Aarhus, Aarhus N, Denmark
    Scand J Infect Dis 39:630-4. 2007
    ..The aetiology, pathophysiology, diagnosis, and treatment of ADEM is reviewed, and the sparse existing literature on ADEM and HIV infection is discussed...
  7. ncbi Two neisseria meningitidis strains with different ability to stimulate toll-like receptor 4 through the MyD88-independent pathway
    T H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Aarhus, Denmark
    Scand J Immunol 64:646-54. 2006
    ..These data therefore provide molecular insight into activation of the inflammatory response by N. meningitidis, which is one of the key events in the pathogenesis of meningococcal disease...
  8. ncbi Aseptic meningitis caused by reactivation of varicella-zoster virus in two immunocompetent patients
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, University Hospital of Aarhus, Aarhus, Denmark
    Scand J Infect Dis 38:815-8. 2006
    ....
  9. ncbi Live Streptococcus pneumoniae, Haemophilus influenzae, and Neisseria meningitidis activate the inflammatory response through Toll-like receptors 2, 4, and 9 in species-specific patterns
    Trine H Mogensen
    Department of Infectious Diseases, Skejby Hospital, Brendstrupgaardsvej, DK 8200 Aarhus N, Denmark
    J Leukoc Biol 80:267-77. 2006
    ..pneumoniae, H. influenzae, and N. meningitidis each activate several TLRs in species-specific patterns and show that infection with live pathogens may lead to activation of PRR not targeted by inactivated bacteria...
  10. ncbi Important role for Toll-like receptor 9 in host defense against meningococcal sepsis
    Hong Sjölinder
    Department of Medical Biochemistry and Microbiology, Biomedical Center, Uppsala University, P O Box 582, Uppsala, Sweden
    Infect Immun 76:5421-8. 2008
    ..In contrast, plasmacytoid dendritic cells relied entirely on TLR9 to induce these activities. Thus, our data demonstrate an important role for TLR9 in host defense against N. meningitidis...
  11. ncbi p38 MAPK autophosphorylation drives macrophage IL-12 production during intracellular infection
    Leesun Kim
    Department of Microbiology and Immunology, College of Veterinary Medicine, Cornell University, Ithaca, NY 14853 6401, USA
    J Immunol 174:4178-84. 2005
    ..Furthermore, a synthetic p38 catalytic-site inhibitor blocked tachyzoite-induced p38alpha MAPK phosphorylation. These data are the first to demonstrate p38 MAPK autophosphorylation triggered by intracellular infection...
  12. ncbi Long-term renal effects of a neutralizing RAGE antibody in obese type 2 diabetic mice
    Allan Flyvbjerg
    Medical Department M and Medical Research Laboratories, Institute of Experimental Clinical Research, Aarhus University Hospital, Aarhus, Denmark
    Diabetes 53:166-72. 2004
    ..In conclusion, RAGE is an important pathogenetic factor in the renal changes in an animal model of type 2 diabetes...
  13. ncbi Activation of NF-kappa B in virus-infected macrophages is dependent on mitochondrial oxidative stress and intracellular calcium: downstream involvement of the kinases TGF-beta-activated kinase 1, mitogen-activated kinase/extracellular signal-regulated kin
    Trine H Mogensen
    Department of Medical Microbiology and Immunology, University of Aarhus, Aarhus, Denmark
    J Immunol 170:6224-33. 2003
    ....
  14. ncbi [Toll-like receptors, a new way of treatment?]
    Claus Vinter Bødker Pedersen
    Arhus Universitetshospital, Infektionsmedicinsk Afdeling Q, Skejby, Arhus N
    Ugeskr Laeger 169:2193-8. 2007
    ....
  15. ncbi Suppression of proinflammatory cytokine expression by herpes simplex virus type 1
    Trine H Mogensen
    Department of Medical Microbiology and Immunology, The Bartholin Building, University of Aarhus, DK-8000 Aarhus C, Denmark
    J Virol 78:5883-90. 2004
    ..Thus, HSV is able to suppress expression of proinflammatory cytokines by decreasing the stability of mRNAs, thereby potentially impeding the antiviral host response to infection...