Research Topics
Genomes and Genes | D R KaplanSummaryAffiliation: McGill University Country: Canada Publications
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Publications
Signal transduction by the neurotrophin receptorsD R Kaplan
Brain Tumor Research Centre, Montreal Neurological Institute, 3801 University Street, Montreal, PQ, Canada, H3A 2B4
Curr Opin Cell Biol 9:213-21. 1997..These differing responses point to a complex interplay between neurotrophin-stimulated survival, differentiation, and apoptosis pathways...
Neurotrophin signal transduction in the nervous systemD R Kaplan
Brain Tumor Research Center, Montreal Neurological Institute, Montreal, H3A 2B4, Canada
Curr Opin Neurobiol 10:381-91. 2000..In addition, a number of novel p75NTR-interacting proteins have been identified that transmit growth, survival, and apoptotic signals...
Neurotrophin signalling pathways regulating neuronal apoptosisF D Miller
Center for Neuronal Survival, Montreal Neurological Institute, PQ, Canada
Cell Mol Life Sci 58:1045-53. 2001..In this review, we focus on the proapoptotic actions of the p75 neurotrophin receptor (p75NTR), and on the interplay between Trk and p75NTR that determines neuronal survival...
On Trk for retrograde signalingF D Miller
Center for Neuronal Survival and Brain Tumor Research Center, Montreal Neurological Institute, McGill University, 3801 rue University, H3A 2B4, Montreal, Quebec, Canada
Neuron 32:767-70. 2001....
Isolation of multipotent adult stem cells from the dermis of mammalian skinJ G Toma
Center for Neuronal Survival, Montreal Neurological Institute, McGill University, 3801 rue University, Montreal, Quebec, Canada H3A 2B4
Nat Cell Biol 3:778-84. 2001....
An anti-apoptotic role for the p53 family member, p73, during developmental neuron deathC D Pozniak
Center for Neuronal Survival, Brain Tumor Research Center, Montreal Neurological Institute, McGill University, Montreal, Canada H3A 2B4
Science 289:304-6. 2000..Thus, truncated p73 is an essential anti-apoptotic protein in neurons, serving to counteract the pro-apoptotic function of p53...
Direct regulation of the Akt proto-oncogene product by phosphatidylinositol-3,4-bisphosphateT F Franke
ABL Basic Research Program, National Cancer Institute Frederick Cancer Research Facility and Development Center NCI FCRFDC, Frederick, MD 21702, USA
Science 275:665-8. 1997..Thus, Akt is apparently regulated by the direct interaction of PtdIns-3,4-P2 with the Akt PH domain...
Autophosphorylation of activation loop tyrosines regulates signaling by the TRK nerve growth factor receptorM E Cunningham
Department of Pathology and Center of Neurobiology and Behavior, College of Physicians and Surgeons, Columbia University, New York, New York 10032, USA
J Biol Chem 272:10957-67. 1997....
The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinaseT F Franke
Fox Chase Cancer Center, Philadelphia, Pennsylvania 19111, USA
Cell 81:727-36. 1995..These results identify Akt as a novel target of PI 3-kinase and suggest that the Akt PH domain may be a mediator of PI 3-kinase signaling...
A Trk nerve growth factor (NGF) receptor point mutation affecting interaction with phospholipase C-gamma 1 abolishes NGF-promoted peripherin induction but not neurite outgrowthD M Loeb
Department of Pathology, Columbia University College of Physicians and Surgeons, New York, New York 10032
J Biol Chem 269:8901-10. 1994..This supports the presence of multiple and separable signaling pathways in the NGF mechanism of action...
High-affinity NGF binding requires coexpression of the trk proto-oncogene and the low-affinity NGF receptorB L Hempstead
Department of Medicine, Cornell University Medical College, New York, New York 10021
Nature 350:678-83. 1991..These studies reveal a new growth factor receptor-mediated mechanism of cellular differentiation involving trk and the low-affinity NGF receptor...
The cytoplasmic and transmembrane domains of the p75 and Trk A receptors regulate high affinity binding to nerve growth factorD Esposito
Division of Hematology, Weill Medical College of Cornell University, New York, New York 10021, USA
J Biol Chem 276:32687-95. 2001..These results indicate that the transmembrane and cytoplasmic domains of Trk A and p75 are responsible for high affinity site formation and suggest that p75 alters the conformation of Trk A to generate high affinity NGF binding...
95-kilodalton B-Raf serine/threonine kinase: identification of the protein and its major autophosphorylation siteR M Stephens
Molecular Mechanisms of Carcinogenesis Laboratory, National Cancer Institute Frederick Cancer Research and Development Center, Maryland
Mol Cell Biol 12:3733-42. 1992..A threonine residue is present at similar positions in all three mammalian Raf family members and may represent a regulatory site for these proteins...
Trk receptors use redundant signal transduction pathways involving SHC and PLC-gamma 1 to mediate NGF responsesR M Stephens
ABL Basic Research Program, NCI Frederick Cancer Research and Development Center, Maryland 21702
Neuron 12:691-705. 1994..These results suggest a model whereby Trk receptors utilize at least partially redundant signal transduction pathways to mediate NGF responses...
AH/PH domain-mediated interaction between Akt molecules and its potential role in Akt regulationK Datta
Fox Chase Cancer Center, Philadelphia, Pennsylvania 19111
Mol Cell Biol 15:2304-10. 1995..Addition of maltose-binding protein-AH/PH fusion recombinant protein, which is expected to bind Akt, to the immunoprecipitates from serum-starved cells induced the activation of the Akt kinase...
Expression of a Trk high affinity nerve growth factor receptor in the human prostateB R Pflug
Department of Cell Biology, Georgetown University Medical Center, Washington, D C 20007
Endocrinology 136:262-8. 1995....
Direct activation of the serine/threonine kinase activity of Raf-1 through tyrosine phosphorylation by the PDGF beta-receptorD K Morrison
Department of Medicine, Howard Hughes Medical Institute, University of California, San Francisco 94143
Cell 58:649-57. 1989..This is the first demonstration of the direct modulation of a protein activity by a growth factor receptor tyrosine kinase...
Neurotrophin-5: a novel neurotrophic factor that activates trk and trkBL R Berkemeier
Department of Molecular Biology, Genentech, Inc, South San Francisco, California 94080
Neuron 7:857-66. 1991..Receptor sharing could enable neurotrophic factors emanating from distinct targets to cooperate in regulating neurons with multiple connections...
Tyrosine phosphorylation and tyrosine kinase activity of the trk proto-oncogene product induced by NGFD R Kaplan
Eukaryotic Signal Transduction Group, NCI Frederick Cancer Research and Development Center, Maryland 21701
Nature 350:158-60. 1991..Activation of the trk tyrosine kinase receptor provides a possible mechanism for signal transduction by NGF...
The neurotrophic factors brain-derived neurotrophic factor and neurotrophin-3 are ligands for the trkB tyrosine kinase receptorD Soppet
Molecular Embryology Section, NCI Frederick Cancer Research and Development Center, Maryland 21702
Cell 65:895-903. 1991..Thus, the trk family of receptors are likely to be important signal transducers of NGF-related trophic signals in the formation and maintenance of neuronal circuits...
Rap1 is involved in the signal transduction of myelin-associated glycoproteinJ Taniguchi
Department of Neurobiology, Graduate School of Medicine, Chiba University, Chuo Ku, Chiba, Japan
Cell Death Differ 15:408-19. 2008..Rap1 activity is required for survival of the DRG neurons as well as CGNs when treated with MAG. The transport of the signaling complex containing the p75 receptor and Rap1 may play a role in the effect of MAG...
