Thomas Moeslinger

Summary

Affiliation: University of Vienna
Country: Austria

Publications

  1. ncbi Urea induces macrophage proliferation by inhibition of inducible nitric oxide synthesis
    T Moeslinger
    Institute for Medical Physiology, Vienna, Austria
    Kidney Int 56:581-8. 1999
  2. ncbi Inhibition of inducible nitric oxide synthesis by oxidized lipoprotein(a) in a murine macrophage cell line
    T Moeslinger
    Institute for Physiology, Schwarzspanierstrasse 17, 1090, Vienna, Austria
    FEBS Lett 478:95-9. 2000
  3. ncbi Inhibition of inducible nitric oxide synthesis by the herbal preparation Padma 28 in macrophage cell line
    T Moeslinger
    Institute of Physiology, Vienna, Austria
    Can J Physiol Pharmacol 78:861-6. 2000
  4. ncbi Inhibition of inducible nitric oxide synthesis by azathioprine in a macrophage cell line
    Thomas Moeslinger
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstrasse 17, 1090 Vienna, Austria
    Life Sci 79:374-81. 2006
  5. ncbi Aqueous extracts of Cimicifuga racemosa and phenolcarboxylic constituents inhibit production of proinflammatory cytokines in LPS-stimulated human whole blood
    Diethart Schmid
    Institute of Physiology, Centre for Physiology, Pathophysiology and Immunology, Medical University of Vienna, Schwarzspanierstrasse 17, 1090 Vienna, Austria
    Can J Physiol Pharmacol 87:963-72. 2009
  6. ncbi Pinacidil-primed ATP-sensitive potassium channels mediate feedback control of mechanical power output in isolated myocardium of rats and guinea pigs
    Diethart Schmid
    Institute of Physiology, Centre for Physiology and Pharmacology, Medical University of Vienna, Vienna, Austria
    Eur J Pharmacol 628:116-27. 2010
  7. ncbi Inhibition of inducible nitric oxide synthesis by Cimicifuga racemosa (Actaea racemosa, black cohosh) extracts in LPS-stimulated RAW 264.7 macrophages
    Diethart Schmid
    Institute for Physiology, Section of Vegetative Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Austria
    J Pharm Pharmacol 61:1089-96. 2009
  8. ncbi Oxidized phospatidylcholine but not native phosphatidylcholine inhibits inducible nitric oxide synthase in RAW 264.7 macrophages
    Roswitha Friedl
    Medical University of Vienna, Institute of Physiology, Schwarzspanierstrasse 17, A 1090 Vienna, Austria
    Life Sci 78:1586-91. 2006
  9. ncbi ATP-sensitive potassium channels expressed by human monocytes play a role in stasis-induced thrombogenesis via tissue factor pathway
    Diethart Schmid
    Center of Physiology and Pathophysiology, Department of Physiology, Medical University of Vienna, Schwarzspanierstrasse 17, A 1090, Austria
    Life Sci 80:989-98. 2007
  10. ncbi An abundant, truncated human sulfonylurea receptor 1 splice variant has prodiabetic properties and impairs sulfonylurea action
    Diethart Schmid
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Vienna, Austria
    Cell Mol Life Sci 69:129-48. 2012

Detail Information

Publications10

  1. ncbi Urea induces macrophage proliferation by inhibition of inducible nitric oxide synthesis
    T Moeslinger
    Institute for Medical Physiology, Vienna, Austria
    Kidney Int 56:581-8. 1999
    ..Atherosclerosis is a major cause of morbidity and mortality in chronic renal failure and is associated with the proliferation of macrophages within atherosclerotic lesions...
  2. ncbi Inhibition of inducible nitric oxide synthesis by oxidized lipoprotein(a) in a murine macrophage cell line
    T Moeslinger
    Institute for Physiology, Schwarzspanierstrasse 17, 1090, Vienna, Austria
    FEBS Lett 478:95-9. 2000
    ..Ox-Lp(a) induced iNOS inhibition might contribute to the development of atherosclerotic lesions by reducing the anti-atherogenic effects of nitric oxide...
  3. ncbi Inhibition of inducible nitric oxide synthesis by the herbal preparation Padma 28 in macrophage cell line
    T Moeslinger
    Institute of Physiology, Vienna, Austria
    Can J Physiol Pharmacol 78:861-6. 2000
    ..The inhibition of inducible nitric oxide synthesis might contribute to the anti-inflammatory activities of Padma 28...
  4. ncbi Inhibition of inducible nitric oxide synthesis by azathioprine in a macrophage cell line
    Thomas Moeslinger
    Institute of Physiology, Center for Physiology and Pathophysiology, Medical University of Vienna, Schwarzspanierstrasse 17, 1090 Vienna, Austria
    Life Sci 79:374-81. 2006
    ..The inhibition of inducible nitric oxide synthesis might contribute to the anti-inflammatory activities of azathioprine...
  5. ncbi Aqueous extracts of Cimicifuga racemosa and phenolcarboxylic constituents inhibit production of proinflammatory cytokines in LPS-stimulated human whole blood
    Diethart Schmid
    Institute of Physiology, Centre for Physiology, Pathophysiology and Immunology, Medical University of Vienna, Schwarzspanierstrasse 17, 1090 Vienna, Austria
    Can J Physiol Pharmacol 87:963-72. 2009
    ..The effect of this compound may explain the antiinflammatory activities of CRE and its beneficial actions in rheumatism and other inflammatory diseases...
  6. ncbi Pinacidil-primed ATP-sensitive potassium channels mediate feedback control of mechanical power output in isolated myocardium of rats and guinea pigs
    Diethart Schmid
    Institute of Physiology, Centre for Physiology and Pharmacology, Medical University of Vienna, Vienna, Austria
    Eur J Pharmacol 628:116-27. 2010
    ..This effect is associated with a reduced development of hypoxic areas inside the heart muscle by adapting cardiac function to a limited energy supply...
  7. ncbi Inhibition of inducible nitric oxide synthesis by Cimicifuga racemosa (Actaea racemosa, black cohosh) extracts in LPS-stimulated RAW 264.7 macrophages
    Diethart Schmid
    Institute for Physiology, Section of Vegetative Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Austria
    J Pharm Pharmacol 61:1089-96. 2009
    ..The present study focuses on the effects of C. racemosa root extracts on inducible nitric oxide synthase (iNOS) in lipopolysaccharide-stimulated murine macrophages (RAW 264.7)...
  8. ncbi Oxidized phospatidylcholine but not native phosphatidylcholine inhibits inducible nitric oxide synthase in RAW 264.7 macrophages
    Roswitha Friedl
    Medical University of Vienna, Institute of Physiology, Schwarzspanierstrasse 17, A 1090 Vienna, Austria
    Life Sci 78:1586-91. 2006
    ..Additionally, chromatin immunoprecipitation assay revealed that oxPAPC reduced the interaction of the active NF-kappaB subunit p65 with the iNOS promoter region when compared to native PAPC...
  9. ncbi ATP-sensitive potassium channels expressed by human monocytes play a role in stasis-induced thrombogenesis via tissue factor pathway
    Diethart Schmid
    Center of Physiology and Pathophysiology, Department of Physiology, Medical University of Vienna, Schwarzspanierstrasse 17, A 1090, Austria
    Life Sci 80:989-98. 2007
    ..These channels are able to enhance Ca(2+)-dependent intracellular signalling and can increase TF-activity and phosphatidylserine exposure thereby accelerating clot formation during stasis by monocytes...
  10. ncbi An abundant, truncated human sulfonylurea receptor 1 splice variant has prodiabetic properties and impairs sulfonylurea action
    Diethart Schmid
    Institute of Physiology, Center for Physiology and Pharmacology, Medical University of Vienna, Vienna, Austria
    Cell Mol Life Sci 69:129-48. 2012
    ..Thus, SUR1?2 represents an endogenous K(ATP)-channel modulator with prodiabetic properties in islet cells. Its predominance in heart may explain why high-affinity sulfonylurea receptors are not found in human cardiac tissue...