Research Topics
Species | Bente A Talseth-PalmerSummaryAffiliation: University of Newcastle Country: Australia Publications
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Detail Information
Publications
Whole genome amplification and its impact on CGH array profilesBente A Talseth-Palmer
School of Biomedical Sciences, Faculty of Health, University of Newcastle and the Hunter Medical Research Institute, NSW, Australia
BMC Res Notes 1:56. 2008..Four experiments were conducted on amplified DNA compared to unamplified DNA and two experiments on unamplified DNA compared to unamplified DNA...
A 1q44 deletion, paternal UPD of chromosome 2 and a deletion due to a complex translocation detected in children with abnormal phenotypes using new SNP array technologyB A Talseth-Palmer
Faculty of Health, School of Biomedical Sciences, University of Newcastle and the Hunter Medical Research Institute, Newcastle, NSW, Australia
Cytogenet Genome Res 124:94-101. 2009..The results of this study suggest that it may be desirable to investigate idiopathic mental retardation using genome-wide SNP arrays, in conjunction with other cytogenetic and molecular techniques...
MSH6 and PMS2 mutation positive Australian Lynch syndrome families: novel mutations, cancer risk and age of diagnosis of colorectal cancerBente A Talseth-Palmer
School of Biomedical Sciences and Pharmacy, University of Newcastle, NSW 2308, Australia
Hered Cancer Clin Pract 8:5. 2010..This study reports on our findings on mutation type, cancer risk and age of diagnosis in MSH6 and PMS2 families...
Colorectal cancer susceptibility loci on chromosome 8q23.3 and 11q23.1 as modifiers for disease expression in Lynch syndromeBente A Talseth-Palmer
School of Biomedical Sciences and Pharmacy, University of Newcastle, HMRI, Room 3642, Level 3, John Hunter Hospital, Newcastle, Australia
J Med Genet 48:279-84. 2011..The objective of this study was to genotype nine SNPs within these six loci to confirm previous findings and investigate whether they act as modifiers of disease risk in patients with Lynch syndrome...
Haemochromatosis HFE gene polymorphisms as potential modifiers of hereditary nonpolyposis colorectal cancer risk and onset ageZumin Shi
Research Centre for Gender, Health and Ageing, University of Newcastle, Callaghan, Australia
Int J Cancer 125:78-83. 2009..Understanding the mechanisms by which HFE interrelates with colorectal malignancies could lead to reduction of disease risk in HNPCC...
P53 in human melanoma fails to regulate target genes associated with apoptosis and the cell cycle and may contribute to proliferationKelly A Avery-Kiejda
Oncology and Immunology, Calvary Mater Newcastle Hospital, University of Newcastle, Newcastle, NSW, Australia
BMC Cancer 11:203. 2011..While mutation of the P53 tumour suppressor gene is a common feature of many types of cancer, mutational inactivation of P53 in melanoma is uncommon; however, its function often appears abnormal...
