Research Topics
Species | Jeremy B M JowettSummaryCountry: Australia Publications
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Detail Information
Publications
Combining target enrichment with barcode multiplexing for high throughput SNP discoveryNik Cummings
Baker IDI Heart and Diabetes Institute, Melbourne, Australia
BMC Genomics 11:641. 2010....
Genetic influences on type 2 diabetes and metabolic syndrome related quantitative traits in MauritiusJeremy B Jowett
Baker IDI Heart and Diabetes Institute, Melbourne, Victoria, Australia
Twin Res Hum Genet 12:44-52. 2009..A high heritability for key type 2 diabetes and metabolic syndrome related phenotypes (range 0.23 to 0.68), suggest the cohort will have utility in identifying genes that influence these quantitative traits...
GATTACA--are we there yet?Jeremy B M Jowett
Baker IDI Heart and Diabetes Institute, 75 Commercial Road, Melbourne, Victoria 3004, Australia
Nat Rev Endocrinol 5:187-8. 2009..In the past few years, new genetic risk factors for type 2 diabetes mellitus have been identified in large-scale studies of genetic associations. But can their inclusion in clinically based risk models improve disease prediction?..
Genetic variation at the FTO locus influences RBL2 gene expressionJeremy B M Jowett
Department of Genomics and Systems Biology, Baker IDI Heart and Diabetes Institute, Melbourne, Victoria, Australia
Diabetes 59:726-32. 2010..We combined gene variation and expression data in a human cohort to identify causal genes...
Genome-wide scan identifies a quantitative trait locus at 4p15.3 for serum urateNik Cummings
Genomics and Systems Biology, Baker IDI Heart and Diabetes Institute, Melbourne, Victoria, Australia
Eur J Hum Genet 18:1243-7. 2010..Our study results implicate genetic variation in SLC2A9 in influencing levels of serum urate over a broad range of values in a large Mauritian family cohort...
Association of genetic variation within UBL5 with phenotypes of metabolic syndromeKiymet Bozaoglu
International Diabetes Institute, Caulfield, Victoria, Australia
Hum Biol 78:147-59. 2006..023) in fasted individuals. These data are consistent with our earlier published studies and further support a functional role for the UBL5 gene in influencing physiological traits that underpin the development of metabolic syndrome...
